What is she best known for?
The fields of study she is best known for:
Ulrike Mayer mainly investigates Laminin, Cell biology, Biochemistry, Basement membrane assembly and Basement membrane.
Her study on Nidogen-2 is often connected to Cortical dysplasia as part of broader study in Laminin.
Her Nidogen-2 study incorporates themes from Protein structure and Molecular biology.
Her Cell biology study combines topics from a wide range of disciplines, such as Phenotype, Genetics, Mutant and Cell division.
Her studies deal with areas such as EGF-like domain and Laminin 111 as well as Basement membrane assembly.
Her Basement membrane research integrates issues from Morphogenesis, Meninges and Dysplasia.
Her most cited work include:
- A simplified laminin nomenclature (681 citations)
- Collagen IV is essential for basement membrane stability but dispensable for initiation of its assembly during early development (565 citations)
- The Arabidopsis KNOLLE Protein Is a Cytokinesis-specific Syntaxin (429 citations)
What are the main themes of her work throughout her whole career to date?
Her primary scientific interests are in Cell biology, Laminin, Biochemistry, Molecular biology and Integrin.
Her biological study spans a wide range of topics, including Autophagy, ATG16L1, Transcriptome and Phenotype.
Her research integrates issues of Perlecan and Basement membrane in her study of Laminin.
The Recombinant DNA, Binding site, Proteases and Site-directed mutagenesis research Ulrike Mayer does as part of her general Biochemistry study is frequently linked to other disciplines of science, such as Osteonectin, therefore creating a link between diverse domains of science.
Her Molecular biology research is multidisciplinary, relying on both Embryonic stem cell, Messenger RNA, Gene, Gene isoform and Mutagenesis.
Her Integrin study incorporates themes from Muscular dystrophy, ITGA7, Skeletal muscle and Regeneration.
She most often published in these fields:
- Cell biology (40.59%)
- Laminin (33.66%)
- Biochemistry (25.74%)
What were the highlights of her more recent work (between 2016-2021)?
- Autophagy (12.87%)
- Cell biology (40.59%)
- ATG16L1 (7.92%)
In recent papers she was focusing on the following fields of study:
Autophagy, Cell biology, ATG16L1, Paneth cell and Transcriptome are her primary areas of study.
Her work deals with themes such as Crohn's disease, Computational biology, Systems biology and Multi omics, which intersect with Autophagy.
Ulrike Mayer combines subjects such as Sequestosome 1 and Endocytosis with her study of Cell biology.
Her work carried out in the field of ATG16L1 brings together such families of science as ATG5, Phagosome, Tissue homeostasis, Viral replication and UVRAG.
Her Transcriptome study combines topics from a wide range of disciplines, such as Phenotype and microRNA.
Her Phenotype study integrates concerns from other disciplines, such as Calcitriol receptor and Transcription factor.
Between 2016 and 2021, her most popular works were:
- The WD40 domain of ATG16L1 is required for its non‐canonical role in lipidation of LC3 at single membranes (92 citations)
- The ATG5-binding and coiled coil domains of ATG16L1 maintain autophagy and tissue homeostasis in mice independently of the WD domain required for LC3 associated phagocytosis (37 citations)
- LC3-Associated Phagocytosis Is Required for Dendritic Cell Inflammatory Cytokine Response to Gut Commensal Yeast Saccharomyces cerevisiae. (14 citations)
In her most recent research, the most cited papers focused on:
Ulrike Mayer mainly focuses on Cell biology, ATG16L1, Autophagy, Phagocytosis and Paneth cell.
The Cell biology study combines topics in areas such as Microvesicles, Gut flora and In vitro.
Her research in ATG16L1 intersects with topics in Phagosome, Tissue homeostasis, UVRAG, Sequestosome 1 and Autophagosome.
Her studies in Autophagy integrate themes in fields like Cell and Vacuole.
Her Phagocytosis research incorporates themes from Proinflammatory cytokine, Immune system and Dysbiosis.
Her Paneth cell research overlaps with other disciplines such as Protein degradation, Secretion, Transcriptome, Downregulation and upregulation and Organoid.
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