What is she best known for?
The fields of study she is best known for:
Sarah J. Parsons mainly focuses on Cancer research, Proto-oncogene tyrosine-protein kinase Src, Tyrosine kinase, Receptor tyrosine kinase and Growth factor receptor.
The Cancer research study combines topics in areas such as Receptor, Internal medicine, STAT protein and Endocrinology.
Phosphorylation covers Sarah J. Parsons research in Proto-oncogene tyrosine-protein kinase Src.
Her research investigates the connection between Tyrosine kinase and topics such as Kinase that intersect with problems in Exocytosis, DNA-binding protein and Cancer cell.
Her Receptor tyrosine kinase research includes elements of ROR1, Tropomyosin receptor kinase C and Mitogen-activated protein kinase.
Her Growth factor receptor study incorporates themes from Insulin-like growth factor 1 receptor and ERBB3.
Her most cited work include:
- Constitutive activation of Stat3 by the Src and JAK tyrosine kinases participates in growth regulation of human breast carcinoma cells. (669 citations)
- C-Src-mediated phosphorylation of the epidermal growth factor receptor on Tyr845 and Tyr1101 is associated with modulation of receptor function (573 citations)
- Mechanism of biological synergy between cellular Src and epidermal growth factor receptor (425 citations)
What are the main themes of her work throughout her whole career to date?
Cancer research, Proto-oncogene tyrosine-protein kinase Src, Cell biology, Phosphorylation and Tyrosine kinase are her primary areas of study.
Her Cancer research research includes themes of Endocrinology, Carcinogenesis, Internal medicine, LNCaP and Growth factor receptor.
Her studies deal with areas such as ERBB3, Epidermal growth factor receptor and Tyrosine phosphorylation as well as Proto-oncogene tyrosine-protein kinase Src.
While the research belongs to areas of Cell biology, Sarah J. Parsons spends her time largely on the problem of Cytokinesis, intersecting her research to questions surrounding Guanine nucleotide exchange factor.
Her Phosphorylation research incorporates themes from Receptor, Epidermal growth factor, Tyrosine, Molecular biology and Kinase.
Her study in Tyrosine kinase is interdisciplinary in nature, drawing from both Receptor tyrosine kinase, Estrogen receptor and Transfection.
She most often published in these fields:
- Cancer research (43.01%)
- Proto-oncogene tyrosine-protein kinase Src (40.86%)
- Cell biology (36.56%)
What were the highlights of her more recent work (between 2010-2019)?
- Cell biology (36.56%)
- Cancer research (43.01%)
- Prostate cancer (10.75%)
In recent papers she was focusing on the following fields of study:
Sarah J. Parsons focuses on Cell biology, Cancer research, Prostate cancer, Internal medicine and Androgen receptor.
Her study of Phosphorylation is a part of Cell biology.
Her Cancer research study integrates concerns from other disciplines, such as Tyrosine and Protein kinase A.
Her work carried out in the field of Internal medicine brings together such families of science as Endocrinology and Oncology.
Her research in Endocrinology intersects with topics in Tyrosine kinase, Estrogen receptor beta, Receptor tyrosine kinase and Estrogen receptor alpha.
Her Integrin research also works with subjects such as
- Endosome and related Kinase,
- Protein kinase R that intertwine with fields like Proto-oncogene tyrosine-protein kinase Src.
Between 2010 and 2019, her most popular works were:
- microRNA-148a is a prognostic oncomiR that targets MIG6 and BIM to regulate EGFR and apoptosis in glioblastoma (80 citations)
- Androgen receptor degradation by the E3 ligase CHIP modulates mitotic arrest in prostate cancer cells (62 citations)
- Inhibition of Neurotensin Receptor 1 Selectively Sensitizes Prostate Cancer to Ionizing Radiation (37 citations)
In her most recent research, the most cited papers focused on:
Her main research concerns Cell biology, Prostate cancer, Androgen receptor, Cell growth and Neurosphere.
In the field of Cell biology, her study on Focal adhesion, Proto-oncogene tyrosine-protein kinase Src and Protein kinase R overlaps with subjects such as ULK1.
Sarah J. Parsons combines subjects such as Signal transduction, Protein tyrosine phosphatase, Apoptosis, Annexin and Molecular biology with her study of Prostate cancer.
The study incorporates disciplines such as 2-Methoxyestradiol, Gene knockdown, Mdm2 and Small interfering RNA in addition to Androgen receptor.
Sarah J. Parsons interconnects Oncomir, Stem cell and Apoptosis Regulator in the investigation of issues within Cell growth.
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