What is he best known for?
The fields of study he is best known for:
- Gene
- Neuron
- Phosphorylation
Richard L. Huganir spends much of his time researching Cell biology, AMPA receptor, Neuroscience, Synaptic plasticity and Long-term depression.
He has researched Cell biology in several fields, including NMDA receptor, Receptor and Biochemistry.
His work deals with themes such as Ca2+/calmodulin-dependent protein kinase and Protein kinase A, which intersect with AMPA receptor.
His Neuroscience research incorporates elements of Long-Term Synaptic Depression and Metabotropic glutamate receptor.
His studies in Synaptic plasticity integrate themes in fields like Long-term potentiation and Neurotransmission.
His biological study spans a wide range of topics, including Kainate receptor and Endocrinology.
His most cited work include:
- MAPK cascade signalling and synaptic plasticity (1164 citations)
- Regulation of distinct AMPA receptor phosphorylation sites during bidirectional synaptic plasticity. (944 citations)
- Homer: a protein that selectively binds metabotropic glutamate receptors. (937 citations)
What are the main themes of his work throughout his whole career to date?
His primary scientific interests are in Cell biology, AMPA receptor, Neuroscience, Synaptic plasticity and Biochemistry.
In his work, Signal transduction is strongly intertwined with Receptor, which is a subfield of Cell biology.
Richard L. Huganir usually deals with AMPA receptor and limits it to topics linked to Phosphorylation and Protein subunit and Tyrosine.
His Neuroscience study incorporates themes from Long-term potentiation, Metaplasticity and Synaptic scaling.
His study looks at the relationship between Synaptic plasticity and fields such as Neurotransmission, as well as how they intersect with chemical problems.
His research integrates issues of Ion channel linked receptors, Metabotropic glutamate receptor and Kainate receptor in his study of Long-term depression.
He most often published in these fields:
- Cell biology (50.00%)
- AMPA receptor (47.59%)
- Neuroscience (42.98%)
What were the highlights of his more recent work (between 2013-2021)?
- Neuroscience (42.98%)
- AMPA receptor (47.59%)
- Cell biology (50.00%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Neuroscience, AMPA receptor, Cell biology, Synaptic plasticity and Long-term potentiation.
His Neuroscience research integrates issues from Glutamate receptor and Neurotransmission.
His AMPA receptor research is multidisciplinary, relying on both In vivo, SYNGAP1 and Phosphorylation.
The study incorporates disciplines such as Dendritic spine, Synapse and Postsynaptic potential, Postsynaptic density in addition to Cell biology.
Synaptic plasticity connects with themes related to Long-term depression in his study.
As a part of the same scientific family, Richard L. Huganir mostly works in the field of Long-term potentiation, focusing on Arc and, on occasion, Memory consolidation.
Between 2013 and 2021, his most popular works were:
- The C9orf72 repeat expansion disrupts nucleocytoplasmic transport (559 citations)
- Homer1a drives homeostatic scaling-down of excitatory synapses during sleep (244 citations)
- Phase Transition in Postsynaptic Densities Underlies Formation of Synaptic Complexes and Synaptic Plasticity. (205 citations)
In his most recent research, the most cited papers focused on:
Richard L. Huganir focuses on Neuroscience, AMPA receptor, Synaptic plasticity, Long-term potentiation and Cell biology.
His Neuroscience research includes elements of Glutamatergic and Neurotransmission.
The AMPA receptor study combines topics in areas such as LTP induction and Phosphorylation.
His research in the fields of Metaplasticity and Synaptic scaling overlaps with other disciplines such as Schizophrenia.
His Long-term potentiation research incorporates themes from Hippocampus, Nerve injury, Postsynaptic potential and Long-Term Synaptic Depression.
He has included themes like Postsynaptic density, Internalization, Protein turnover, Environmental enrichment and Synaptosome in his Cell biology study.
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