What is he best known for?
The fields of study he is best known for:
Cell biology, Internal medicine, Endocrinology, Biochemistry and Coated vesicle are his primary areas of study.
His work carried out in the field of Cell biology brings together such families of science as Neurite and Tissue culture.
His studies in Endocrinology integrate themes in fields like Molecular biology, Protein disulfide-isomerase and Proteasome.
In general Biochemistry study, his work on Gene, Gene duplication and Calmodulin often relates to the realm of Isoleucine, thereby connecting several areas of interest.
Richard E. Fine has researched Coated vesicle in several fields, including Integral membrane protein, Adenosine triphosphate, Protein kinase A and Kinase activity.
While the research belongs to areas of Alzheimer's disease, Richard E. Fine spends his time largely on the problem of Amyloid, intersecting his research to questions surrounding Neurotrophin.
His most cited work include:
- Receptor-mediated transcytosis of transferrin across the blood-brain barrier. (428 citations)
- Binding of beta-amyloid to the p75 neurotrophin receptor induces apoptosis. A possible mechanism for Alzheimer's disease. (289 citations)
- Non-age Related Differences in Thrombin Responses by Platelets from Male Patients with Advanced Alzheimer′s Disease (177 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Biochemistry, Cell biology, Molecular biology, Receptor and Endocrinology.
The various areas that he examines in his Biochemistry study include Biophysics and Axoplasmic transport.
Richard E. Fine interconnects Transferrin and Internalization in the investigation of issues within Cell biology.
His studies deal with areas such as Complementary DNA, Apoptosis and Gene isoform as well as Molecular biology.
The Receptor study combines topics in areas such as Vasopressin and Neuroscience.
His Endocrinology research is multidisciplinary, incorporating perspectives in Alzheimer's disease, Amyloid precursor protein, Internal medicine and Platelet activation.
He most often published in these fields:
- Biochemistry (46.43%)
- Cell biology (36.43%)
- Molecular biology (31.43%)
What were the highlights of his more recent work (between 2008-2019)?
- Neuroscience (14.29%)
- Receptor (16.43%)
- Neuron (5.71%)
In recent papers he was focusing on the following fields of study:
Richard E. Fine mainly investigates Neuroscience, Receptor, Neuron, Cell biology and Axon.
Richard E. Fine combines subjects such as Cell, Enteric nervous system, Signal transduction, Synapse and Estrogen with his study of Receptor.
His research in Neuron intersects with topics in Dementia, Disease, Neurite and Dopamine.
His research integrates issues of Presenilin and Multicellular organism in his study of Cell biology.
His studies in Axon integrate themes in fields like Tight junction and Dendrite.
His work carried out in the field of Dendrite brings together such families of science as Axoplasmic transport, Kinesin, Microtubule and Organelle.
Between 2008 and 2019, his most popular works were:
- RETROMER DISRUPTION PROMOTES AMYLOIDOGENIC APP PROCESSING (74 citations)
- Intraperitoneal injection of the pancreatic peptide amylin potently reduces behavioral impairment and brain amyloid pathology in murine models of Alzheimer's disease. (61 citations)
- Pyroglutamate-Aβ 3 and 11 colocalize in amyloid plaques in Alzheimer's disease cerebral cortex with pyroglutamate-Aβ 11 forming the central core. (30 citations)
In his most recent research, the most cited papers focused on:
His primary areas of investigation include Neuroscience, Synapse, Receptor, Amyloid beta and Polyclonal antibodies.
His study in Nicotine, Olfactory bulb, Amphetamine, Nucleus accumbens and Dopaminergic is done as part of Neuroscience.
His work deals with themes such as Enteric nervous system, Neurite, Neuron and Synaptic pruning, which intersect with Synapse.
He interconnects Growth cone and Gene in the investigation of issues within Receptor.
His Amyloid beta research incorporates elements of BACE1-AS, Antibody, Colocalization and Antigen.
Richard E. Fine has included themes like Cerebral cortex and Senile plaques in his Polyclonal antibodies study.
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