World's Best Scientists 2026 revealed!

D-Index & Metrics

Neuroscience

D-Index
42
Citations
6056
World Ranking
7704
National Ranking
586

Overview

Peter C. Kind is affiliated with the University of Edinburgh in the United Kingdom and has made contributions primarily in the fields of Biochemistry, Genetics and Molecular Biology, and Neuroscience. Their research outputs include a significant number of publications related to Genetics, Molecular Biology, Cellular and Molecular Neuroscience, Cognitive Neuroscience, and Cell Biology.

The main research topics covered in their work include Genetics and Neurodevelopmental Disorders, Autism Spectrum Disorder Research, Neuroscience and Neuropharmacology Research, Cellular transport and secretion, Neurogenesis and neuroplasticity mechanisms, Congenital heart defects research, and Neuroinflammation and Neurodegeneration Mechanisms.

Peter C. Kind has collaborated frequently with several researchers, including Owen Dando, Sam A. Booker, Sumantra Chattarji, David J. A. Wyllie, and Giles E. Hardingham.

The scientist has published regularly in a variety of venues, with notable frequent contributions to bioRxiv (Cold Spring Harbor Laboratory), Molecular Autism, Cell Reports, Brain Communications, and Journal of Instrumentation.

Recent published papers include:

  • "Reactive astrocytes acquire neuroprotective as well as deleterious signatures in response to Tau and Aß pathology", 2022, Nature Communications
  • "Selective vulnerability of inhibitory networks in multiple sclerosis", 2021, Acta Neuropathologica
  • "Input-Output Relationship of CA1 Pyramidal Neurons Reveals Intact Homeostatic Mechanisms in a Mouse Model of Fragile X Syndrome", 2020, Cell Reports
  • "Medial septal GABAergic neurons reduce seizure duration upon optogenetic closed-loop stimulation", 2021, Brain
  • "Excess ribosomal protein production unbalances translation in a model of Fragile X Syndrome", 2022, Nature Communications

Best Publications

  • Reactive astrocytes acquire neuroprotective as well as deleterious signatures in response to Tau and Aß pathology

    Unknown

  • Critical Period Plasticity Is Disrupted in the Barrel Cortex of Fmr1 Knockout Mice

    Emily G. Harlow;Sally M. Till;Theron A. Russell;Lasani S. Wijetunge

  • Reversal of Disease-Related Pathologies in the Fragile X Mouse Model by Selective Activation of GABAB Receptors with Arbaclofen

    Christina Henderson;Lasani Wijetunge;Mika Nakamoto Kinoshita;Matthew Shumway

  • A family of activity-dependent neuronal cell-surface chondroitin sulfate proteoglycans in cat visual cortex.

    Cynthia Lander;Peter Kind;Michael Maleski;Susan Hockfield

  • Neural activity: sculptor of 'barrels' in the neocortex

    Reha S Erzurumlu;Peter C Kind

  • PLC-β1, activated via mGluRs, mediates activity-dependent differentiation in cerebral cortex

    Anthony J. Hannan;Colin Blakemore;Alla Katsnelson;Tania Vitalis

  • GRIN2A-related disorders : genotype and functional consequence predict phenotype

    Vincent Strehlow;Henrike O Heyne;Henrike O Heyne;Henrike O Heyne;Danique R M Vlaskamp;Katie F M Marwick

  • The Subtype of GluN2 C-terminal Domain Determines the Response to Excitotoxic Insults

    Marc-André Martel;Tomás J Ryan;Tomás J Ryan;Karen F.S. Bell;Jill H Fowler

  • The role of activity in development of the visual system.

    Frank Sengpiel;Peter C. Kind

  • C9ORF72 repeat expansion causes vulnerability of motor neurons to Ca2+-permeable AMPA receptor-mediated excitotoxicity

    Bhuvaneish T. Selvaraj;Matthew R. Livesey;Chen Zhao;Jenna M. Gregory

  • Interocular suppression in the visual cortex of strabismic cats

    Frank Sengpiel;Cohn Blakemore;Peter C. Kind;Richard Harrad

  • Correlated binocular activity guides recovery from monocular deprivation

    Peter C. Kind;Donald E. Mitchell;Bashir Ahmed;Colin Blakemore

  • Conserved hippocampal cellular pathophysiology but distinct behavioural deficits in a new rat model of FXS

    Sally M. Till;Antonis Asiminas;Adam D. Jackson;Danai Katsanevaki

  • Altered maturation of the primary somatosensory cortex in a mouse model of fragile X syndrome

    Sally M. Till;Lasani S. Wijetunge;Viktoria G. Seidel;Emily Harlow

  • Cell-Type-Specific Translation Profiling Reveals a Novel Strategy for Treating Fragile X Syndrome

    Sophie R. Thomson;Sang S. Seo;Stephanie A. Barnes;Susana R. Louros

  • mGluR5 Regulates Glutamate-Dependent Development of the Mouse Somatosensory Cortex

    Lasani S. Wijetunge;Sally M. Till;Thomas H. Gillingwater;Cali A. Ingham

  • SynGAP isoforms exert opposing effects on synaptic strength

    A C McMahon;Mark Barnett;Timothy O'Leary;P N Stoney

  • Characterisation of CDKL5 Transcript Isoforms in Human and Mouse.

    Ralph D. Hector;Owen Dando;Nicoletta Landsberger;Charlotte Kilstrup-Nielsen

  • Activity-dependent regulation of synapse and dendritic spine morphology in developing barrel cortex requires phospholipase C-beta1 signalling.

    Tara L. Spires;Zoltán Molnár;Peter C. Kind;Patricia M. Cordery

  • Convergence of Hippocampal Pathophysiology in Syngap+/- and Fmr1-/y Mice.

    Stephanie A. Barnes;Lasani S. Wijetunge;Lasani S. Wijetunge;Adam D. Jackson;Danai Katsanevaki

  • Fgf receptor 3 activation promotes selective growth and expansion of occipitotemporal cortex

    Rachel E Thomson;Peter C Kind;Nicholas A Graham;Michelle L Etherson

  • The Role of Activity in Development of the Visual System Review

    Frank Sengpiel;Peter C. Kind

Frequent Co-Authors

David J. A. Wyllie
David J. A. Wyllie University of Edinburgh
Giles E. Hardingham
Giles E. Hardingham University of Edinburgh
Sumantra Chattarji
Sumantra Chattarji National Centre for Biological Sciences
Seth G. N. Grant
Seth G. N. Grant University of Edinburgh
Frank Sengpiel
Frank Sengpiel Cardiff University
Colin Blakemore
Colin Blakemore City University of Hong Kong
Emma R. Wood
Emma R. Wood University of Edinburgh
Donald E. Mitchell
Donald E. Mitchell Dalhousie University
John T.R. Isaac
John T.R. Isaac Johnson & Johnson (United States)

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