What is he best known for?
The fields of study he is best known for:
- Neuron
- Neuroscience
- Genetics
Kimberly M. Huber mainly focuses on Neuroscience, Metabotropic glutamate receptor, Synaptic plasticity, Long-term depression and Fragile X syndrome.
As part of his studies on Neuroscience, he often connects relevant subjects like AMPA receptor.
In his papers, Kimberly M. Huber integrates diverse fields, such as Metabotropic glutamate receptor and Translation.
His Synaptic plasticity study combines topics from a wide range of disciplines, such as NMDA receptor and Long-term potentiation.
His Long-term depression study incorporates themes from Metabotropic glutamate receptor 5, Long-Term Synaptic Depression, Metabotropic glutamate receptor 7 and MAPK/ERK pathway.
The concepts of his Fragile X syndrome study are interwoven with issues in Neural Inhibition, Autism and FMR1.
His most cited work include:
- The mGluR theory of fragile X mental retardation (1286 citations)
- Altered synaptic plasticity in a mouse model of fragile X mental retardation (1065 citations)
- Altered synaptic plasticity in a mouse model of fragile X mental retardation (1065 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Neuroscience, Metabotropic glutamate receptor, Fragile X syndrome, FMR1 and Synaptic plasticity.
His research integrates issues of Metabotropic glutamate receptor 5, Long-term depression and Postsynaptic potential in his study of Neuroscience.
Kimberly M. Huber works mostly in the field of Metabotropic glutamate receptor, limiting it down to topics relating to Postsynaptic density and, in certain cases, Scaffold protein.
His Fragile X syndrome research is multidisciplinary, relying on both Dendritic spine, Hippocampus and Autism.
His Mavoglurant study, which is part of a larger body of work in FMR1, is frequently linked to Ribosome and Translation, bridging the gap between disciplines.
His work on Synaptic tagging is typically connected to Regulator as part of general Synaptic plasticity study, connecting several disciplines of science.
He most often published in these fields:
- Neuroscience (79.01%)
- Metabotropic glutamate receptor (38.27%)
- Fragile X syndrome (32.10%)
What were the highlights of his more recent work (between 2014-2021)?
- Neuroscience (79.01%)
- Cell biology (23.46%)
- FMR1 (29.63%)
In recent papers he was focusing on the following fields of study:
The scientist’s investigation covers issues in Neuroscience, Cell biology, FMR1, Fragile X syndrome and Synapse.
His studies in Neuroscience integrate themes in fields like Metabotropic glutamate receptor 5, Metabotropic glutamate receptor, Signal transduction and Neurotransmission.
Kimberly M. Huber has researched Metabotropic glutamate receptor in several fields, including Postsynaptic density, Immunology and Long-Term Synaptic Depression.
His research investigates the connection between Cell biology and topics such as Synaptic plasticity that intersect with problems in Endocytic cycle and Long-term potentiation.
His FMR1 research incorporates elements of Cortex and Knockout mouse.
The study incorporates disciplines such as Dendritic spine and Hippocampus in addition to Fragile X syndrome.
Between 2014 and 2021, his most popular works were:
- Dysregulation of Mammalian Target of Rapamycin Signaling in Mouse Models of Autism (96 citations)
- MEF2C regulates cortical inhibitory and excitatory synapses and behaviors relevant to neurodevelopmental disorders. (88 citations)
- Increased expression of the PI3K enhancer PIKE mediates deficits in synaptic plasticity and behavior in Fragile X syndrome (77 citations)
In his most recent research, the most cited papers focused on:
- Neuron
- Neuroscience
- Genetics
His primary areas of investigation include Neuroscience, Signal transduction, Fragile X syndrome, Metabotropic glutamate receptor 5 and Metabotropic glutamate receptor.
Kimberly M. Huber combines topics linked to Neurodevelopmental disorder with his work on Neuroscience.
In his work, Sirolimus and PI3K/AKT/mTOR pathway is strongly intertwined with Synaptic plasticity, which is a subfield of Signal transduction.
His work in Fragile X syndrome is not limited to one particular discipline; it also encompasses FMR1.
Kimberly M. Huber interconnects Scaffold protein, Postsynaptic density and Metabotropic receptor in the investigation of issues within Metabotropic glutamate receptor 5.
His Metabotropic glutamate receptor study frequently draws parallels with other fields, such as Neuroplasticity.
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