Member of the Association of American Physicians
His main research concerns Pancreas, Pancreatic cancer, Cancer research, Internal medicine and Endocrinology. His work deals with themes such as Pancreatitis, Northern blot and Pathology, which intersect with Pancreas. His Pancreatic cancer research incorporates themes from Cancer cell, Fibroblast growth factor and Adenocarcinoma.
His Cancer research research is multidisciplinary, incorporating perspectives in Epidermal growth factor, Fibroblast growth factor receptor 4, microRNA, Growth factor and Cyclin D. His Internal medicine study incorporates themes from Messenger RNA and Amylase. His work on Transforming growth factor beta and Central nervous system as part of general Endocrinology study is frequently linked to Substantia nigra, bridging the gap between disciplines.
Murray Korc mainly investigates Pancreatic cancer, Cancer research, Internal medicine, Endocrinology and Pancreas. His work carried out in the field of Pancreatic cancer brings together such families of science as Cancer cell, Metastasis and Pathology. His research integrates issues of Growth factor, Epidermal growth factor, Autocrine signalling and Cell growth in his study of Cancer research.
His Internal medicine research integrates issues from Cell culture, Oncology and Amylase. Murray Korc has researched Endocrinology in several fields, including Receptor, Molecular biology and Northern blot. His studies in Pancreas integrate themes in fields like Pancreatitis and Fibroblast growth factor.
Pancreatic cancer, Cancer research, Cancer, microRNA and Cancer cell are his primary areas of study. His Pancreatic cancer study is concerned with Internal medicine in general. His Internal medicine research is multidisciplinary, incorporating elements of Genetics, Endocrinology and Oncology.
The Cancer research study combines topics in areas such as Carcinogenesis, KRAS, Immunology and Cell growth. His Cancer study combines topics in areas such as Cell culture, Transfection, Lung and Bioinformatics. The concepts of his microRNA study are interwoven with issues in Lung cancer and In situ hybridization.
Murray Korc mainly focuses on Cancer research, Pancreatic cancer, Pancreas, Cell growth and Cancer cell. His studies link microRNA with Cancer research. His Pancreatic cancer study frequently draws connections between adjacent fields such as Epidermal growth factor.
His studies deal with areas such as KRAS, Pancreatitis and Pathology as well as Pancreas. His Cell growth study also includes fields such as
Angiogenesis, which have a strong connection to Pancreatic tumor, Tumor progression, Cell biology and Fibroblast growth factor,
Vascular endothelial growth factor A which is related to area like Fibroblast growth factor receptor and Neovascularization. His Cancer cell study also includes
Tumor microenvironment and related Growth factor, Erlotinib and Transforming growth factor,
Molecular biology most often made with reference to Stromal cell.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Estrogen binding, receptor mRNA, and biologic response in osteoblast-like osteosarcoma cells
Barry S. Komm;Christopher M. Terpening;David J. Benz;Kimberlie A. Graeme.
Overexpression of the epidermal growth factor receptor in human pancreatic cancer is associated with concomitant increases in the levels of epidermal growth factor and transforming growth factor alpha.
M Korc;B Chandrasekar;Y Yamanaka;H Friess.
Journal of Clinical Investigation (1992)
Enhanced expression of transforming growth factor β isoforms in pancreatic cancer correlates with decreased survival
Helmut Friess;Yoichiro Yamanaka;Markus Büchler;Matthias Ebert.
In vivo induction of massive proliferation, directed migration, and differentiation of neural cells in the adult mammalian brain
James Fallon;Steve Reid;Richard Kinyamu;Isaac Opole.
Proceedings of the National Academy of Sciences of the United States of America (2000)
Action of secretagogues on a new preparation of functionally intact, isolated pancreatic acini.
John A. Williams;Murray Korc;Robert L. Dormer.
American Journal of Physiology (1978)
Coexpression of epidermal growth factor receptor and ligands in human pancreatic cancer is associated with enhanced tumor aggressiveness.
Yamanaka Y;Friess H;Kobrin Ms;Buchler M.
Anticancer Research (1993)
Id-1 and Id-2 are overexpressed in pancreatic cancer and in dysplastic lesions in chronic pancreatitis.
Haruhisa Maruyama;Jörg Kleeff;Stefan Wildi;Helmut Friess.
American Journal of Pathology (1999)
MicroRNA-31 functions as an oncogenic microRNA in mouse and human lung cancer cells by repressing specific tumor suppressors
Xi Liu;Lorenzo F. Sempere;Haoxu Ouyang;Vincent A. Memoli.
Journal of Clinical Investigation (2010)
Overexpression of Acidic and Basic Fibroblast Growth Factors in Human Pancreatic Cancer Correlates with Advanced Tumor Stage
Yoichiro Yamanaka;Helmut Friess;Markus Buchler;Hans G. Beger.
Cancer Research (1993)
Vickie E. Baracos;Lisa Martin;Murray Korc;Denis C. Guttridge.
Nature Reviews Disease Primers (2018)
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