By researching both Genetics and Anatomy, Masashi Akiyama produces research that crosses academic boundaries. Anatomy and Genetics are two areas of study in which Masashi Akiyama engages in interdisciplinary work. His Internal medicine study frequently links to related topics such as Receptor. Many of his studies on Receptor apply to Internal medicine as well. While working on this project, Masashi Akiyama studies both Cell biology and Biochemistry. Masashi Akiyama combines Biochemistry and Cell biology in his research. Masashi Akiyama integrates many fields, such as Gene and Mutation, in his works. Masashi Akiyama integrates Mutation and Gene in his studies. His Autoantibody research extends to Immunology, which is thematically connected.
Genetics and Ichthyosis are commonly linked in his work. His Ichthyosis study frequently draws connections between adjacent fields such as Genetics. Masashi Akiyama incorporates Dermatology and Pathology in his research. Masashi Akiyama combines Pathology and Dermatology in his studies. Masashi Akiyama integrates Gene with Mutation in his study. He integrates several fields in his works, including Mutation and Gene. In his research, he undertakes multidisciplinary study on Internal medicine and Endocrinology. Masashi Akiyama performs multidisciplinary study in Endocrinology and Internal medicine in his work. He combines Immunology and Antibody in his research.
His research combines Atopic dermatitis and Dermatology. He combines Immunology and Cytokine in his research. In most of his Internal medicine studies, his work intersects topics such as Receptor. Masashi Akiyama integrates Genetics with Mutation in his research. His work blends Mutation and Gene studies together. Gene and Genetics are two areas of study in which he engages in interdisciplinary research. He undertakes multidisciplinary investigations into Pathology and Disease in his work. His work often combines Disease and Pathology studies. In his articles, he combines various disciplines, including Antibody and Autoantibody.
His Psoriasis study is within the categories of Generalized pustular psoriasis and Pustular psoriasis. In most of his Generalized pustular psoriasis studies, his work intersects topics such as Immunology. His Immunology study frequently links to related topics such as Interleukin. He carries out multidisciplinary research, doing studies in Interleukin and Chemokine. He performs integrative study on Chemokine and Proinflammatory cytokine. Proinflammatory cytokine and Inflammation are two areas of study in which Masashi Akiyama engages in interdisciplinary work. He merges Inflammation with Neutrophil extracellular traps in his study. His study connects Psoriasis and Pustular psoriasis. Dermatology is closely attributed to Keratitis in his work.
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Revised nomenclature and classification of inherited ichthyoses: Results of the First Ichthyosis Consensus Conference in Sorèze 2009
Vinzenz Oji;Gianluca Tadini;Masashi Akiyama;Claudine Blanchet Bardon.
Journal of The American Academy of Dermatology (2010)
Restricted feeding entrains liver clock without participation of the suprachiasmatic nucleus
Reiko Hara;Keiko Wan;Hisanori Wakamatsu;Reiko Aida.
Genes to Cells (2001)
Mutations in lipid transporter ABCA12 in harlequin ichthyosis and functional recovery by corrective gene transfer
Masashi Akiyama;Yoriko Sugiyama-Nakagiri;Kaori Sakai;James R. McMillan.
Journal of Clinical Investigation (2005)
Restricted-feeding-induced anticipatory activity rhythm is associated with a phase-shift of the expression of mPer1 and mPer2 mRNA in the cerebral cortex and hippocampus but not in the suprachiasmatic nucleus of mice.
Hisanori Wakamatsu;Yuko Yoshinobu;Reiko Aida;Takahiro Moriya.
European Journal of Neuroscience (2001)
Inhibition of Light- or Glutamate-Induced mPer1 Expression Represses the Phase Shifts into the Mouse Circadian Locomotor and Suprachiasmatic Firing Rhythms
Masashi Akiyama;Yasuko Kouzu;Satomi Takahashi;Hisanori Wakamatsu.
The Journal of Neuroscience (1999)
Adrenergic regulation of clock gene expression in mouse liver
Hideyuki Terazono;Tatsushi Mutoh;Shun Yamaguchi;Masaki Kobayashi.
Proceedings of the National Academy of Sciences of the United States of America (2003)
Loss of kindlin-1, a human homolog of the Caenorhabditis elegans actin-extracellular-matrix linker protein UNC-112, causes Kindler syndrome
Dawn H. Siegel;Gabrielle H.S. Ashton;Homero G. Penagos;James V. Lee.
American Journal of Human Genetics (2003)
Humanization of autoantigen
Wataru Nishie;Daisuke Sawamura;Maki Goto;Kei Ito.
Nature Medicine (2007)
Unique mutations in the filaggrin gene in Japanese patients with ichthyosis vulgaris and atopic dermatitis.
Toshifumi Nomura;Aileen Sandilands;Masashi Akiyama;Haihui Liao.
The Journal of Allergy and Clinical Immunology (2007)
Nonphotic entrainment by 5-HT1A/7 receptor agonists accompanied by reduced Per1 and Per2 mRNA levels in the suprachiasmatic nuclei.
Kazumasa Horikawa;Shin Ichi Yokota;Kazuyuki Fuji;Masashi Akiyama.
The Journal of Neuroscience (2000)
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