Her primary areas of investigation include Molecular biology, Epidermolysis bullosa, Cell biology, Pathology and Basement membrane. Her Molecular biology study integrates concerns from other disciplines, such as Point mutation, Biochemistry, Ectodomain and Keratinocyte. Her Epidermolysis bullosa research includes elements of Mutation, Missense mutation, Junctional epidermolysis bullosa and Disease.
Her Disease research incorporates elements of Dermatology, Bullous disease, Epidermolysis bullosa simplex and Kindler syndrome. Her biological study spans a wide range of topics, including Autoantibody and Immunology. Her studies in Basement membrane integrate themes in fields like Laminin, Extracellular matrix, Epidermis and Dermoepidermal junction.
Leena Bruckner-Tuderman mostly deals with Pathology, Epidermolysis bullosa, Dermatology, Molecular biology and Cell biology. Her Pathology research includes themes of Autoantibody and Bullous pemphigoid. Her research integrates issues of Mutation, Junctional epidermolysis bullosa and Immunology in her study of Epidermolysis bullosa.
Her Dermatology study frequently intersects with other fields, such as Disease. Her Molecular biology study incorporates themes from Ectodomain and Keratinocyte. The various areas that Leena Bruckner-Tuderman examines in her Cell biology study include Wound healing, Integrin and Cell adhesion.
Leena Bruckner-Tuderman spends much of her time researching Epidermolysis bullosa, Dermatology, Pathology, Cell biology and Cancer research. Her Epidermolysis bullosa study improves the overall literature in Genetics. Her Dermatology research is multidisciplinary, incorporating elements of Retrospective cohort study and Hypotrichosis.
Connective tissue and Genetic heterogeneity is closely connected to Molecular genetics in her research, which is encompassed under the umbrella topic of Pathology. She interconnects Wound healing and Ectodomain in the investigation of issues within Cell biology. Her work focuses on many connections between ATAC-seq and other disciplines, such as Cell, that overlap with her field of interest in Molecular biology.
Her main research concerns Pathology, Epidermolysis bullosa dystrophica, Extracellular matrix, Epidermolysis bullosa and Fibrosis. Her Pathology research incorporates themes from Cell and Regeneration. Extracellular matrix is a subfield of Cell biology that she studies.
Her research in Cell biology is mostly focused on Basement membrane. Her study in Epidermolysis bullosa is interdisciplinary in nature, drawing from both Therapeutic approach, Disease, Molecular pathology, Phenotype and Immunology. Her research in Disease tackles topics such as Mucous membrane pemphigoid which are related to areas like Dermatology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The classification of inherited epidermolysis bullosa (EB): Report of the Third International Consensus Meeting on Diagnosis and Classification of EB.
Jo David Fine;Robin A J Eady;Eugene A. Bauer;Johann W. Bauer.
Journal of The American Academy of Dermatology (2008)
A simplified laminin nomenclature
Monique Aumailley;Leena Bruckner-Tuderman;William G. Carter;Rainer Deutzmann.
Matrix Biology (2005)
Inherited epidermolysis bullosa: Updated recommendations on diagnosis and classification
Jo David Fine;Leena Bruckner-Tuderman;Robin A.J. Eady;Eugene A. Bauer.
Journal of The American Academy of Dermatology (2014)
Revised classification system for inherited epidermolysis bullosa: Report of the second international consensus meeting on diagnosis and classification of epidermolysis bullosa
Jo David Fine;Robin A J Eady;Eugene A. Bauer;Robert A. Briggaman.
Journal of The American Academy of Dermatology (2000)
Epidermolysis bullosa acquisita antigen is the globular carboxyl terminus of type VII procollagen.
D T Woodley;R E Burgeson;G Lunstrum;L Bruckner-Tuderman.
Journal of Clinical Investigation (1988)
Mutations in the Gene Encoding the RER Protein FKBP65 Cause Autosomal-Recessive Osteogenesis Imperfecta
Yasemin Alanay;Hrispima Avaygan;Natalia Camacho;G. Eda Utine.
American Journal of Human Genetics (2010)
The Shed Ectodomain of Collagen XVII/BP180 Is Targeted by Autoantibodies in Different Blistering Skin Diseases
Hauke Schumann;Jens Baetge;Kaisa Tasanen;Kaisa Tasanen;Fenella Wojnarowska.
American Journal of Pathology (2000)
Transmembrane collagen XVII, an epithelial adhesion protein, is shed from the cell surface by ADAMs.
Claus‐Werner Franzke;Kaisa Tasanen;Heike Schäcke;Zhongjun Zhou.
The EMBO Journal (2002)
Antibodies to tissue transglutaminase as serologic markers in patients with dermatitis herpetiformis.
Walburga Dieterich;Eberhardt Laag;Leena Bruckner-Tuderman;Timo Reunala.
Journal of Investigative Dermatology (1999)
Integrin α3 mutations with kidney, lung, and skin disease
Cristina Has;Giuseppina Spartà;Dimitra Kiritsi;Lisa Weibel.
The New England Journal of Medicine (2012)
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