What is he best known for?
The fields of study he is best known for:
Alain Hovnanian mainly focuses on Genetics, Netherton syndrome, LEKTI, KLK5 and KLK7.
Netherton syndrome connects with themes related to Cell biology in his study.
In his research, Filaggrin, Elastase and Skin erythema is intimately related to Proteases, which falls under the overarching field of LEKTI.
His KLK5 research is multidisciplinary, relying on both Cathelicidin, Antimicrobial peptides, Serine Peptidase Inhibitor Kazal-Type 5 and Kallikrein.
He interconnects Gene polymorphism, Immunology and Atopy in the investigation of issues within Serine Peptidase Inhibitor Kazal-Type 5.
His KLK7 research integrates issues from Molecular biology and Stratum corneum.
His most cited work include:
- TLR3 deficiency in patients with herpes simplex encephalitis. (839 citations)
- The classification of inherited epidermolysis bullosa (EB): Report of the Third International Consensus Meeting on Diagnosis and Classification of EB. (688 citations)
- Mutations in SPINK5, encoding a serine protease inhibitor, cause Netherton syndrome. (645 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Genetics, Netherton syndrome, Gene, Dermatology and Molecular biology.
His study in Netherton syndrome is interdisciplinary in nature, drawing from both Serine Peptidase Inhibitor Kazal-Type 5, Stratum corneum, LEKTI, Immunology and KLK5.
His LEKTI research incorporates themes from Proteases and Kallikrein.
Alain Hovnanian combines subjects such as Proinflammatory cytokine, KLK7 and Cell biology with his study of KLK5.
His biological study spans a wide range of topics, including Disease and Pathology.
His Molecular biology study incorporates themes from Epidermolysis bullosa dystrophica and Mutant.
He most often published in these fields:
- Genetics (34.19%)
- Netherton syndrome (33.05%)
- Gene (25.36%)
What were the highlights of his more recent work (between 2018-2021)?
- Netherton syndrome (33.05%)
- Dermatology (22.22%)
- Cell biology (14.53%)
In recent papers he was focusing on the following fields of study:
Alain Hovnanian spends much of his time researching Netherton syndrome, Dermatology, Cell biology, Kallikrein and Hidradenitis suppurativa.
His Netherton syndrome study integrates concerns from other disciplines, such as Protease, KLK5, Stratum corneum and LEKTI.
Many of his studies involve connections with topics such as KLK7 and KLK5.
The various areas that he examines in his LEKTI study include Proteases and Ichthyosis.
His Cell biology research is multidisciplinary, incorporating elements of OLMSTED SYNDROME, Desmoglein 3 and Gene, Mendelian inheritance.
His Kallikrein study which covers Pharmacology that intersects with Serine Peptidase Inhibitor Kazal-Type 5 and Transepidermal water loss.
Between 2018 and 2021, his most popular works were:
- Consensus reclassification of inherited epidermolysis bullosa and other disorders with skin fragility (74 citations)
- Safety and early efficacy outcomes for lentiviral fibroblast gene therapy in recessive dystrophic epidermolysis bullosa. (18 citations)
- Respiratory virus infection triggers acute psoriasis flares across different clinical subtypes and genetic backgrounds (15 citations)
In his most recent research, the most cited papers focused on:
Alain Hovnanian mostly deals with Pathology, Microbiome, Staphylococcus epidermidis, Microbiology and Hidradenitis suppurativa.
His study in the field of Epidermolysis bullosa simplex and Anetoderma also crosses realms of Macular hyperpigmentation.
His Microbiome research encompasses a variety of disciplines, including Actinomyces, Prevotella, Microbiological culture, Mobiluncus and Kocuria.
Along with Staphylococcus epidermidis, other disciplines of study including Staphopain, Protease inhibitor, Proteases, Human skin and Netherton syndrome are integrated into his research.
He interconnects Cohort study, Oncology, Young adult, Mutation and Gene mutation in the investigation of issues within Hidradenitis suppurativa.
His Genetic enhancement research includes elements of Phase i ii, Ex vivo, Transgene, Recombinant DNA and Recessive dystrophic epidermolysis bullosa.
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