Member of the Association of American Physicians
His primary areas of investigation include Immunology, Cell biology, Molecular biology, Basement membrane and Laminin. His studies link Human skin with Immunology. His biological study spans a wide range of topics, including Wound healing, Secretion and Keratinocyte.
His Molecular biology research incorporates themes from Focal adhesion, Collagen, type I, alpha 1, Function, Dystrophic epidermolysis bullosa and Platelet-derived growth factor. His research integrates issues of Dermatology and Dermis in his study of Bullous pemphigoid. His Epidermolysis bullosa acquisita research is multidisciplinary, incorporating perspectives in Immunoglobulin G, Anchoring fibrils and Pathology.
His primary scientific interests are in Cell biology, Molecular biology, Pathology, Immunology and Dermatology. The study incorporates disciplines such as Wound healing, Cell migration and Keratinocyte in addition to Cell biology. The various areas that David T. Woodley examines in his Molecular biology study include Cell culture, Recombinant DNA, Collagen, type I, alpha 1, Laminin and Fibronectin.
His study looks at the intersection of Pathology and topics like Epidermolysis bullosa acquisita with Anchoring fibrils, Basement membrane, Autoimmunity and Epitope. David T. Woodley combines subjects such as Cancer research and Human skin with his study of Immunology. His Dermatology study incorporates themes from Type VII collagen and Disease.
David T. Woodley mainly focuses on Dermatology, Pathology, Wound healing, Nonsense mutation and Cell biology. His Dermatology research is multidisciplinary, relying on both Bullous pemphigoid and Pemphigoid. His studies deal with areas such as Skin wound and Transplantation as well as Pathology.
His research in Wound healing focuses on subjects like Human skin, which are connected to Anatomy and Intradermal injection. His Cell biology study integrates concerns from other disciplines, such as Heat shock protein and Hypoxia-inducible factors. David T. Woodley combines subjects such as Fibronectin and Gene, Transfection with his study of Molecular biology.
His primary areas of study are Dermatology, Wound healing, Epidermolysis bullosa, Molecular biology and Anchoring fibrils. The study incorporates disciplines such as AKT2 and Motility in addition to Wound healing. David T. Woodley focuses mostly in the field of Motility, narrowing it down to topics relating to Receptor and, in certain cases, Immunology and Hypoxia.
His studies in Epidermolysis bullosa integrate themes in fields like Gastroenterology and Internal medicine. His biological study spans a wide range of topics, including Cell culture and Transfection. His Anchoring fibrils research integrates issues from Epidermolysis bullosa dystrophica, Dermis and Human skin.
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The classification of inherited epidermolysis bullosa (EB): Report of the Third International Consensus Meeting on Diagnosis and Classification of EB.
Jo David Fine;Robin A J Eady;Eugene A. Bauer;Johann W. Bauer.
Journal of The American Academy of Dermatology (2008)
Inherited epidermolysis bullosa: Updated recommendations on diagnosis and classification
Jo David Fine;Leena Bruckner-Tuderman;Robin A.J. Eady;Eugene A. Bauer.
Journal of The American Academy of Dermatology (2014)
The first international consensus on mucous membrane pemphigoid: definition, diagnostic criteria, pathogenic factors, medical treatment, and prognostic indicators.
Lawrence S. Chan;A. Razzaque Ahmed;Grant J. Anhalt;Wolfgang Bernauer.
Archives of Dermatology (2002)
Identification of the Skin Basement-Membrane Autoantigen in Epidermolysis Bullosa Acquisita
D T Woodley;R A Briggaman;E J O'Keefe;A O Inman.
The New England Journal of Medicine (1984)
Epidermolysis bullosa acquisita antigen is the globular carboxyl terminus of type VII procollagen.
D T Woodley;R E Burgeson;G Lunstrum;L Bruckner-Tuderman.
Journal of Clinical Investigation (1988)
Epitope spreading : Lessons from autoimmune skin diseases
Lawrence S. Chan;Carol J. Vanderlugt;Takashi Hashimoto;Takeji Nishikawa.
Journal of Investigative Dermatology (1998)
Bone Marrow Transplantation for Recessive Dystrophic Epidermolysis Bullosa
John E Wagner;Akemi Ishida-Yamamoto;John A McGrath;Maria Hordinsky.
The New England Journal of Medicine (2010)
TANGO1 facilitates cargo loading at endoplasmic reticulum exit sites.
Kota Saito;Mei Chen;Fred Bard;Shenghong Chen.
Extracellular heat shock protein-90α: linking hypoxia to skin cell motility and wound healing.
Wei Li;Wei Li;Yong Li;Yong Li;Yong Li;Shengxi Guan;Jianhua Fan;Jianhua Fan.
The EMBO Journal (2007)
The Role of Oxygen in Wound Healing: A Review of the Literature
Paola G. Rodriguez;Frances N. Felix;David T. Woodley;Elisabeth K. Shim.
Dermatologic Surgery (2008)
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