Laminin, Cell biology, Molecular biology, Extracellular matrix and Hemidesmosome are his primary areas of study. His Laminin research incorporates elements of Epithelial cell migration, Basement membrane, Migration Assay, Cell adhesion molecule and Gene isoform. His Cell biology study combines topics in areas such as Integrin and Transcription factor.
His research in Molecular biology intersects with topics in Cell culture, Transfection, Heparan sulfate, Papillomaviridae and Frameshift mutation. His work deals with themes such as Epidermolysis bullosa, Epidermolysis bullosa simplex, Muscular dystrophy, Nonsense mutation and Desmin, which intersect with Hemidesmosome. His research investigates the connection between Epidermolysis bullosa and topics such as Junctional epidermolysis bullosa that intersect with issues in Neural tube.
The scientist’s investigation covers issues in Molecular biology, Laminin, Cell biology, Pathology and Junctional epidermolysis bullosa. The concepts of his Molecular biology study are interwoven with issues in Cell culture, Mutation, Transfection, Complementary DNA and Gene. Guerrino Meneguzzi works mostly in the field of Laminin, limiting it down to topics relating to Epidermolysis bullosa and, in certain cases, Junctional epidermolysis bullosa, as a part of the same area of interest.
His studies deal with areas such as Integrin, Transcription factor, Cell adhesion and Keratinocyte as well as Cell biology. His studies in Pathology integrate themes in fields like Phenotype, Prenatal diagnosis, Muscular dystrophy and Plectin. Guerrino Meneguzzi has included themes like Dermatology and Protein subunit in his Junctional epidermolysis bullosa study.
His main research concerns Cell biology, Cancer research, Pathology, Extracellular matrix and Missense mutation. His Cell biology research integrates issues from Transcription factor and Keratinocyte. His Pathology study combines topics in areas such as Phenotype, Muscular dystrophy and Plectin.
His Muscular dystrophy research incorporates elements of Epidermolysis bullosa and Polyhydramnios. His Extracellular matrix study integrates concerns from other disciplines, such as Tumor microenvironment and Cell adhesion. His studies examine the connections between Allele and genetics, as well as such issues in Complementary DNA, with regards to RNA splicing and Molecular biology.
Guerrino Meneguzzi spends much of his time researching Cell biology, Cancer research, Extracellular matrix, Transcription factor and Carcinogenesis. The study incorporates disciplines such as Integrin, Cell growth, Keratinocyte and Small interfering RNA in addition to Cell biology. His study in Cancer research is interdisciplinary in nature, drawing from both Fibroblast, Regulation of gene expression, microRNA and Downregulation and upregulation.
His Extracellular matrix research includes elements of Tumor microenvironment, Cancer cell and Crosstalk. His Tumor microenvironment research is multidisciplinary, incorporating perspectives in Squamous carcinoma, Signal transduction, Contractility and Cytokine. His research integrates issues of Wound healing, Immunology, Gene silencing and Keratinocyte migration in his study of Transcription factor.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
A simplified laminin nomenclature
Monique Aumailley;Leena Bruckner-Tuderman;William G. Carter;Rainer Deutzmann.
Matrix Biology (2005)
A new nomenclature for the laminins.
Robert E. Burgeson;Matthias Chiquet;Rainer Deutzmann;Peter Ekblom.
Matrix Biology (1994)
Integrin β4 mutations associated with junctional epidermolysis bullosa with pyloric atresia
Frédérique Vidal;Daniel Aberdam;Corinne Miquel;Angela M. Christiano.
Nature Genetics (1995)
Herlitz's junctional epidermolysis bullosa is linked to mutations in the gene (LAMC2) for the gamma 2 subunit of nicein/kalinin (LAMININ-5)
Aberdam D;Galliano Mf;Vailly J;Pulkkinen L.
Nature Genetics (1994)
ROCK and JAK1 Signaling Cooperate to Control Actomyosin Contractility in Tumor Cells and Stroma
Victoria Sanz-Moreno;Cedric Gaggioli;Cedric Gaggioli;Maggie Yeo;Jean Albrengues;Jean Albrengues.
Cancer Cell (2011)
Different heparan sulfate proteoglycans serve as cellular receptors for human papillomaviruses.
Saeed Shafti-Keramat;Alessandra Handisurya;Ernst Kriehuber;Guerrino Meneguzzi.
Journal of Virology (2003)
Defective Expression of Plectin/HD1 in Epidermolysis Bullosa Simplex with Muscular Dystrophy
Yannick Gache;Stéphane Chavanas;Jean Philippe Lacour;Gerhard Wiche.
Journal of Clinical Investigation (1996)
Epigenetic switch drives the conversion of fibroblasts into proinvasive cancer-associated fibroblasts
Jean Albrengues;Thomas Bertero;Eloise Grasset;Stephanie Bonan.
Nature Communications (2015)
A homozygous mutation in the integrin alpha6 gene in junctional epidermolysis bullosa with pyloric atresia
Laura Ruzzi;Laurent Gagnoux-Palacios;Mari Pinola;Serena Belli.
Journal of Clinical Investigation (1997)
Immunization against human papillomavirus type 16 tumor cells with recombinant vaccinia viruses expressing E6 and E7.
Guerrino Meneguzzi;Christa Cerni;Marie Paule Kieny;Richard Lathe;Richard Lathe.
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