D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Molecular Biology D-index 58 Citations 11,300 167 World Ranking 1408 National Ranking 43

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Genetics

Laminin, Cell biology, Molecular biology, Extracellular matrix and Hemidesmosome are his primary areas of study. His Laminin research incorporates elements of Epithelial cell migration, Basement membrane, Migration Assay, Cell adhesion molecule and Gene isoform. His Cell biology study combines topics in areas such as Integrin and Transcription factor.

His research in Molecular biology intersects with topics in Cell culture, Transfection, Heparan sulfate, Papillomaviridae and Frameshift mutation. His work deals with themes such as Epidermolysis bullosa, Epidermolysis bullosa simplex, Muscular dystrophy, Nonsense mutation and Desmin, which intersect with Hemidesmosome. His research investigates the connection between Epidermolysis bullosa and topics such as Junctional epidermolysis bullosa that intersect with issues in Neural tube.

His most cited work include:

  • A simplified laminin nomenclature (681 citations)
  • A new nomenclature for the laminins. (668 citations)
  • Integrin β4 mutations associated with junctional epidermolysis bullosa with pyloric atresia (334 citations)

What are the main themes of his work throughout his whole career to date?

The scientist’s investigation covers issues in Molecular biology, Laminin, Cell biology, Pathology and Junctional epidermolysis bullosa. The concepts of his Molecular biology study are interwoven with issues in Cell culture, Mutation, Transfection, Complementary DNA and Gene. Guerrino Meneguzzi works mostly in the field of Laminin, limiting it down to topics relating to Epidermolysis bullosa and, in certain cases, Junctional epidermolysis bullosa, as a part of the same area of interest.

His studies deal with areas such as Integrin, Transcription factor, Cell adhesion and Keratinocyte as well as Cell biology. His studies in Pathology integrate themes in fields like Phenotype, Prenatal diagnosis, Muscular dystrophy and Plectin. Guerrino Meneguzzi has included themes like Dermatology and Protein subunit in his Junctional epidermolysis bullosa study.

He most often published in these fields:

  • Molecular biology (33.16%)
  • Laminin (24.49%)
  • Cell biology (27.55%)

What were the highlights of his more recent work (between 2007-2020)?

  • Cell biology (27.55%)
  • Cancer research (10.71%)
  • Pathology (23.47%)

In recent papers he was focusing on the following fields of study:

His main research concerns Cell biology, Cancer research, Pathology, Extracellular matrix and Missense mutation. His Cell biology research integrates issues from Transcription factor and Keratinocyte. His Pathology study combines topics in areas such as Phenotype, Muscular dystrophy and Plectin.

His Muscular dystrophy research incorporates elements of Epidermolysis bullosa and Polyhydramnios. His Extracellular matrix study integrates concerns from other disciplines, such as Tumor microenvironment and Cell adhesion. His studies examine the connections between Allele and genetics, as well as such issues in Complementary DNA, with regards to RNA splicing and Molecular biology.

Between 2007 and 2020, his most popular works were:

  • ROCK and JAK1 Signaling Cooperate to Control Actomyosin Contractility in Tumor Cells and Stroma (245 citations)
  • ROCK and JAK1 Signaling Cooperate to Control Actomyosin Contractility in Tumor Cells and Stroma (245 citations)
  • Epigenetic switch drives the conversion of fibroblasts into proinvasive cancer-associated fibroblasts (148 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Genetics

Guerrino Meneguzzi spends much of his time researching Cell biology, Cancer research, Extracellular matrix, Transcription factor and Carcinogenesis. The study incorporates disciplines such as Integrin, Cell growth, Keratinocyte and Small interfering RNA in addition to Cell biology. His study in Cancer research is interdisciplinary in nature, drawing from both Fibroblast, Regulation of gene expression, microRNA and Downregulation and upregulation.

His Extracellular matrix research includes elements of Tumor microenvironment, Cancer cell and Crosstalk. His Tumor microenvironment research is multidisciplinary, incorporating perspectives in Squamous carcinoma, Signal transduction, Contractility and Cytokine. His research integrates issues of Wound healing, Immunology, Gene silencing and Keratinocyte migration in his study of Transcription factor.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

A simplified laminin nomenclature

Monique Aumailley;Leena Bruckner-Tuderman;William G. Carter;Rainer Deutzmann.
Matrix Biology (2005)

998 Citations

A new nomenclature for the laminins.

Robert E. Burgeson;Matthias Chiquet;Rainer Deutzmann;Peter Ekblom.
Matrix Biology (1994)

915 Citations

Integrin β4 mutations associated with junctional epidermolysis bullosa with pyloric atresia

Frédérique Vidal;Daniel Aberdam;Corinne Miquel;Angela M. Christiano.
Nature Genetics (1995)

436 Citations

Herlitz's junctional epidermolysis bullosa is linked to mutations in the gene (LAMC2) for the gamma 2 subunit of nicein/kalinin (LAMININ-5)

Aberdam D;Galliano Mf;Vailly J;Pulkkinen L.
Nature Genetics (1994)

395 Citations

ROCK and JAK1 Signaling Cooperate to Control Actomyosin Contractility in Tumor Cells and Stroma

Victoria Sanz-Moreno;Cedric Gaggioli;Cedric Gaggioli;Maggie Yeo;Jean Albrengues;Jean Albrengues.
Cancer Cell (2011)

381 Citations

Different heparan sulfate proteoglycans serve as cellular receptors for human papillomaviruses.

Saeed Shafti-Keramat;Alessandra Handisurya;Ernst Kriehuber;Guerrino Meneguzzi.
Journal of Virology (2003)

342 Citations

Defective Expression of Plectin/HD1 in Epidermolysis Bullosa Simplex with Muscular Dystrophy

Yannick Gache;Stéphane Chavanas;Jean Philippe Lacour;Gerhard Wiche.
Journal of Clinical Investigation (1996)

306 Citations

Epigenetic switch drives the conversion of fibroblasts into proinvasive cancer-associated fibroblasts

Jean Albrengues;Thomas Bertero;Eloise Grasset;Stephanie Bonan.
Nature Communications (2015)

246 Citations

A homozygous mutation in the integrin alpha6 gene in junctional epidermolysis bullosa with pyloric atresia

Laura Ruzzi;Laurent Gagnoux-Palacios;Mari Pinola;Serena Belli.
Journal of Clinical Investigation (1997)

240 Citations

Immunization against human papillomavirus type 16 tumor cells with recombinant vaccinia viruses expressing E6 and E7.

Guerrino Meneguzzi;Christa Cerni;Marie Paule Kieny;Richard Lathe;Richard Lathe.
Virology (1991)

219 Citations

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