What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Enzyme
Martin Schaller spends much of his time researching Microbiology, Candida albicans, Cell biology, Innate immune system and Immune system.
Martin Schaller specializes in Microbiology, namely Bartonella Infection.
His study of Corpus albicans is a part of Candida albicans.
His studies in Cell biology integrate themes in fields like Proteases, Molecular biology, Cell adhesion and Cell membrane.
His Innate immune system research integrates issues from Signal transduction, TLR4, Immunity and Antimicrobial peptides.
His Immune system study necessitates a more in-depth grasp of Immunology.
His most cited work include:
- Radiation-induced epidermal growth factor receptor nuclear import is linked to activation of DNA-dependent protein kinase (444 citations)
- T-helper-1-cell cytokines drive cancer into senescence (418 citations)
- Granulocytes govern the transcriptional response, morphology and proliferation of Candida albicans in human blood. (378 citations)
What are the main themes of his work throughout his whole career to date?
Microbiology, Dermatology, Candida albicans, Cell biology and Immunology are his primary areas of study.
His research investigates the connection between Microbiology and topics such as Innate immune system that intersect with issues in Immunity.
His Dermatology research incorporates themes from Doxycycline and Pathology.
He is involved in the study of Candida albicans that focuses on Corpus albicans in particular.
His work is dedicated to discovering how Cell biology, Platelet are connected with Thrombus and other disciplines.
All of his Immunology and Interleukin and Inflammation investigations are sub-components of the entire Immunology study.
He most often published in these fields:
- Microbiology (24.50%)
- Dermatology (20.47%)
- Candida albicans (15.77%)
What were the highlights of his more recent work (between 2017-2021)?
- Cancer research (10.07%)
- Dermatology (20.47%)
- Rosacea (8.72%)
In recent papers he was focusing on the following fields of study:
The scientist’s investigation covers issues in Cancer research, Dermatology, Rosacea, Immune system and Melanoma.
His Cancer research study combines topics from a wide range of disciplines, such as Immune checkpoint, Immunotherapy, Cell growth, Tumor microenvironment and Senescence.
The study incorporates disciplines such as Melanoma patient, Targeted therapy and Binimetinib in addition to Dermatology.
The concepts of his Rosacea study are interwoven with issues in Erythema, Prior treatment and Ivermectin.
He has researched Immune system in several fields, including Ebola virus, Ebolavirus, Glycoprotein and Virology.
His Melanoma research is multidisciplinary, incorporating perspectives in Cell culture, Interquartile range and Mitogen-activated protein kinase.
Between 2017 and 2021, his most popular works were:
- Candida albicans-Induced Epithelial Damage Mediates Translocation through Intestinal Barriers. (55 citations)
- Paneth cell α-defensins HD-5 and HD-6 display differential degradation into active antimicrobial fragments. (29 citations)
- Platelet-RBC interaction mediated by FasL/FasR induces procoagulant activity important for thrombosis (29 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Enzyme
Martin Schaller focuses on Rosacea, Cancer research, Dermatology, Ivermectin and Antimicrobial.
His research investigates the connection between Rosacea and topics such as Erythema that intersect with problems in Combination therapy and Concomitant.
Martin Schaller combines subjects such as Platelet activation, Inferior vena cava, Thrombus and Immunotherapy with his study of Cancer research.
He interconnects Facial flushing and Flushing in the investigation of issues within Dermatology.
In Antimicrobial, Martin Schaller works on issues like Peptide, which are connected to Microbiology and Corpus albicans.
In the field of Microbiology, his study on Candida albicans overlaps with subjects such as Candidalysin.
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