Lawrence B. Schwartz focuses on Tryptase, Mast cell, Immunology, Histamine and Molecular biology. Lawrence B. Schwartz combines subjects such as Systemic mastocytosis, Biochemistry, Pathology, Chymase and Antibody with his study of Tryptase. His research integrates issues of Endocrinology, Cell growth, Interleukin 5, Internal medicine and Basophil in his study of Mast cell.
His Immunology study is mostly concerned with Anaphylaxis, Allergy, Immunoglobulin E, Asthma and Allergen. His work deals with themes such as Allergic response, Lysosome, Bioavailability, Bronchoconstriction and Antihistamine, which intersect with Histamine. His Molecular biology study combines topics in areas such as Cell, Fibrinogenolysis, Plasmin, Proteases and Degranulation.
His primary areas of study are Immunology, Tryptase, Mast cell, Molecular biology and Histamine. His study in Allergy, Immunoglobulin E, Systemic mastocytosis, Asthma and Basophil falls under the purview of Immunology. His Tryptase research is multidisciplinary, relying on both Biochemistry, Anaphylaxis, Internal medicine, Chymase and Antibody.
His Mast cell research is multidisciplinary, incorporating perspectives in Stem cell factor, Cytokine, Pathology, Cell biology and Degranulation. The Molecular biology study combines topics in areas such as Complementary DNA and Cell culture. His Histamine research is multidisciplinary, incorporating elements of Bronchoalveolar lavage, Antigen and In vivo.
Lawrence B. Schwartz mostly deals with Immunology, Tryptase, Mast cell, Systemic mastocytosis and Anaphylaxis. His research in Tryptase intersects with topics in Humanized mouse, Immune system, Immunoglobulin E, Cell type and Allosteric regulation. His studies deal with areas such as Cancer research, TPSD1, Severe asthma and Mediator release as well as Mast cell.
His biological study spans a wide range of topics, including Mast cell activation syndrome, Mast cell activation and Dermatology. His studies deal with areas such as Prostaglandin D2, Perioperative, TPSB2 and Internal medicine as well as Anaphylaxis. His biological study spans a wide range of topics, including Allergen, Histamine and Somatic cell.
His scientific interests lie mostly in Immunology, Tryptase, Internal medicine, Systemic mastocytosis and Hereditary angioedema. Asthma, TPSAB1, Humanized mouse, Mast cell and Antigen presentation are subfields of Immunology in which his conducts study. His Mast cell research includes elements of Inflammation, Antibody and Biomarker.
His Tryptase research is multidisciplinary, relying on both Cell Degranulation, Degranulation, Mast cell homeostasis, Anaphylaxis and Allosteric regulation. The Observational study research he does as part of his general Internal medicine study is frequently linked to other disciplines of science, such as Emergency department, therefore creating a link between diverse domains of science. His Hereditary angioedema study integrates concerns from other disciplines, such as Angioedema, C1-inhibitor, Clinical trial, Adverse effect and Randomized controlled trial.
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Evidence That Severe Asthma Can Be Divided Pathologically into Two Inflammatory Subtypes with Distinct Physiologic and Clinical Characteristics
Sally E. Wenzel;Lawrence B. Schwartz;Esther L. Langmack;Janet L. Halliday.
American Journal of Respiratory and Critical Care Medicine (1999)
Two types of human mast cells that have distinct neutral protease compositions.
A A Irani;N M Schechter;S S Craig;G DeBlois.
Proceedings of the National Academy of Sciences of the United States of America (1986)
Diagnostic criteria and classification of mastocytosis: a consensus proposal
Peter Valent;Hans P. Horny;Luis Escribano;B. Jack Longley.
Leukemia Research (2001)
Tryptase Levels as an Indicator of Mast-Cell Activation in Systemic Anaphylaxis and Mastocytosis
Lawrence B. Schwartz;Dean D. Metcalfe;Jeffrey S. Miller;Harry Earl.
The New England Journal of Medicine (1987)
Quantitation of histamine, tryptase, and chymase in dispersed human T and TC mast cells.
L. B. Schwartz;A.-M. A. Irani;K. Roller;M. C. Castells.
Journal of Immunology (1987)
Time course of appearance and disappearance of human mast cell tryptase in the circulation after anaphylaxis.
L B Schwartz;J W Yunginger;J Miller;R Bokhari.
Journal of Clinical Investigation (1989)
Treatment of patients with the hypereosinophilic syndrome with mepolizumab
Marc E. Rothenberg;Amy D. Klion;Florence E. Roufosse;Jean Emmanuel Kahn.
The New England Journal of Medicine (2008)
Human mast cells stimulate vascular tube formation. Tryptase is a novel, potent angiogenic factor.
R J Blair;H Meng;M J Marchese;S Ren.
Journal of Clinical Investigation (1997)
Activation of Pulmonary Mast Cells by Bronchoalveolar Allergen Challenge: In Vivo Release of Histamine and Tryptase in Atopic Subjects with and without Asthma
Sally E. Wenzel;Alpha A. Fowler;Lawrence B. Schwartz.
The American review of respiratory disease (1988)
Detection of MCT and MCTC types of human mast cells by immunohistochemistry using new monoclonal anti-tryptase and anti-chymase antibodies.
Anne-Marie A. Irani;Timothy R. Bradford;Christopher L. Kepley;Norman M. Schechter.
Journal of Histochemistry and Cytochemistry (1989)
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