The scientist’s investigation covers issues in Internal medicine, Endocrinology, Biochemistry, Aldose reductase and Diabetes mellitus. Many of his research projects under Internal medicine are closely connected to Neonatal hypoglycemia with Neonatal hypoglycemia, tying the diverse disciplines of science together. In the field of Endocrinology, his study on Secretion overlaps with subjects such as Cancer cell.
His Biochemistry research integrates issues from Glycation and Molecular biology. His Aldose reductase study integrates concerns from other disciplines, such as Aldo-keto reductase, Sorbitol and Gene expression. His Diabetes mellitus research includes themes of Fructose, Insulin and Lipoprotein.
Kenneth H. Gabbay mostly deals with Internal medicine, Endocrinology, Biochemistry, Aldose reductase and Diabetes mellitus. His Internal medicine study frequently draws connections between related disciplines such as Diabetic neuropathy. His Endocrinology study frequently draws connections to other fields, such as Nerve conduction velocity.
His Biochemistry research is multidisciplinary, incorporating perspectives in Aldose, Molecular biology and Chromatography. His Aldose reductase research is multidisciplinary, relying on both Sorbitol and Stereochemistry. Kenneth H. Gabbay works mostly in the field of Diabetes mellitus, limiting it down to topics relating to Haplotype and, in certain cases, Locus, as a part of the same area of interest.
His main research concerns Biochemistry, Gene, Aldose reductase, Aldehyde Reductase and Molecular biology. His research in Biochemistry intersects with topics in Research use only and Somatostatin. His Gene research focuses on subjects like TCF7L2, which are linked to Expression vector, Methionine and Valine.
His Aldose reductase study necessitates a more in-depth grasp of Endocrinology. His Aldehyde Reductase research incorporates elements of Gene expression and Transcription. He focuses mostly in the field of Molecular biology, narrowing it down to topics relating to Transcription factor and, in certain cases, Regulation of gene expression and Transfection.
His primary areas of investigation include Gene, TCF7L2, Internal medicine, Endocrinology and Cell growth. He has included themes like Proline and Glutamine in his Gene study. He interconnects Kinase, Protein kinase A, MAP3K7IP2, Homology and Intron in the investigation of issues within TCF7L2.
The concepts of his Internal medicine study are interwoven with issues in Intracellular and Cell biology. His Endocrinology research is mostly focused on the topic Aldose reductase. His research integrates issues of Transgene, Osteoclast, Immunology and Osteoblast in his study of Cell growth.
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The glycosylation of hemoglobin: relevance to diabetes mellitus
HF Bunn;KH Gabbay;PM Gallop.
The sorbitol pathway and the complications of diabetes.
Kenneth H. Gabbay.
The New England Journal of Medicine (1973)
The biosynthesis of human hemoglobin A1c. Slow glycosylation of hemoglobin in vivo.
H F Bunn;D N Haney;S Kamin;K H Gabbay.
Journal of Clinical Investigation (1976)
Glycosylated Hemoglobins and Long-Term Blood Glucose Control in Diabetes Mellitus
K H Gabbay;K Hasty;J L Breslow;R C Ellison.
The Journal of Clinical Endocrinology and Metabolism (1977)
Further identification of the nature and linkage of the carbohydrate in hemoglobin A1c.
H.Franklin Bunn;David N. Haney;Kenneth H. Gabbay;Paul M. Gallop.
Biochemical and Biophysical Research Communications (1975)
The aldo-keto reductase superfamily. cDNAs and deduced amino acid sequences of human aldehyde and aldose reductases.
K M Bohren;B Bullock;B Wermuth;K H Gabbay.
Journal of Biological Chemistry (1989)
An unlikely sugar substrate site in the 1.65 Å structure of the human aldose reductase holoenzyme implicated in diabetic complications
David K. Wilson;Kurt M. Bohren;Kenneth H. Gabbay;Florante A. Quiocho.
Sequence of cDNA encoding human insulin-like growth factor I precursor.
M. Jansen;F. M. A. van Schaik;A. T. Ricker;A. T. Ricker;B. Bullock;B. Bullock.
Sorbitol Pathway: Presence in Nerve and Cord with Substrate Accumulation in Diabetes
Kenneth H. Gabbay;Lorenzo O. Merola;Richard A. Field.
A M55V Polymorphism in a Novel SUMO Gene (SUMO-4) Differentially Activates Heat Shock Transcription Factors and Is Associated with Susceptibility to Type I Diabetes Mellitus
Kurt M. Bohren;Varsha Nadkarni;Jian H. Song;Kenneth H. Gabbay.
Journal of Biological Chemistry (2004)
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