Cell biology is closely attributed to Kinase in his research. His Kinase study frequently links to other fields, such as Cell biology. His research on Endoplasmic reticulum frequently links to adjacent areas such as Activating transcription factor. Activating transcription factor and Unfolded protein response are commonly linked in his work. His ATF6 research extends to the thematically linked field of Unfolded protein response. His Endoplasmic reticulum research extends to ATF6, which is thematically connected. Borrowing concepts from Mutant, he weaves in ideas under Gene. He performs integrative study on Mutant and Gene. His research on Biochemistry often connects related topics like Messenger RNA.
While working on this project, Kazunori Imaizumi studies both Cell biology and Neuroscience. Kazunori Imaizumi connects Neuroscience with Cell biology in his research. His work often combines Gene and Gene expression studies. As part of his studies on Endoplasmic reticulum, he frequently links adjacent subjects like Genetics. His studies link Unfolded protein response with Genetics. Kazunori Imaizumi performs integrative Unfolded protein response and Endoplasmic reticulum research in his work. Kazunori Imaizumi performs integrative study on Biochemistry and Molecular biology in his works. In his work, he performs multidisciplinary research in Molecular biology and Gene. In his work, he performs multidisciplinary research in Internal medicine and Endocrinology.
His work on Homeostasis expands to the thematically related Cell biology. His Homeostasis study frequently draws connections to other fields, such as Cell biology. Biochemistry is closely attributed to Protein degradation in his study. His Protein degradation study frequently draws parallels with other fields, such as Biochemistry. He conducts interdisciplinary study in the fields of Gene and Golgi apparatus through his works. By researching both Golgi apparatus and Endoplasmic reticulum, he produces research that crosses academic boundaries. Kazunori Imaizumi connects Endoplasmic reticulum with Calcium signaling in his study. Kazunori Imaizumi integrates many fields, such as Calcium signaling and Signal transduction, in his works. His Genetics research extends to the thematically linked field of Signal transduction.
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Autophagy Is Activated for Cell Survival after Endoplasmic Reticulum Stress
Maiko Ogata;Shin Ichiro Hino;Atsushi Saito;Keisuke Morikawa.
Molecular and Cellular Biology (2006)
Activation of caspase-12, an endoplastic reticulum (ER) resident caspase, through tumor necrosis factor receptor-associated factor 2-dependent mechanism in response to the ER stress.
Takunari Yoneda;Kazunori Imaizumi;Kayoko Oono;Daishi Yui.
Journal of Biological Chemistry (2001)
Involvement of caspase-4 in endoplasmic reticulum stress-induced apoptosis and Aβ-induced cell death
Junichi Hitomi;Junichi Hitomi;Taiichi Katayama;Taiichi Katayama;Yutaka Eguchi;Yutaka Eguchi;Takashi Kudo.
Journal of Cell Biology (2004)
Induction of neuronal death by ER stress in Alzheimer's disease.
Taiichi Katayama;Kazunori Imaizumi;Takayuki Manabe;Junichi Hitomi.
Journal of Chemical Neuroanatomy (2004)
Angiotensin II Type 1a Receptor-deficient Mice with Hypotension and Hyperreninemia
Takeshi Sugaya;Shin-ichiro Nishimatsu;Keiji Tanimoto;Eriko Takimoto.
Journal of Biological Chemistry (1995)
Obesity-induced endoplasmic reticulum stress causes chronic inflammation in adipose tissue
Noritaka Kawasaki;Rie Asada;Atsushi Saito;Soshi Kanemoto.
Scientific Reports (2012)
A molecular chaperone inducer protects neurons from ER stress
T Kudo;S Kanemoto;S Kanemoto;H Hara;N Morimoto.
Cell Death & Differentiation (2008)
Signalling mediated by the endoplasmic reticulum stress transducer OASIS is involved in bone formation.
Tomohiko Murakami;Atsushi Saito;Shin Ichiro Hino;Shinichi Kondo.
Nature Cell Biology (2009)
Apoptosis induced by endoplasmic reticulum stress depends on activation of caspase-3 via caspase-12.
Junichi Hitomi;Taiichi Katayama;Manabu Taniguchi;Akiko Honda.
Neuroscience Letters (2004)
OASIS, a CREB/ATF-family member, modulates UPR signalling in astrocytes
Shinichi Kondo;Tomohiko Murakami;Tomohiko Murakami;Kouko Tatsumi;Maiko Ogata;Maiko Ogata.
Nature Cell Biology (2005)
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