D-Index & Metrics Best Publications

Kazunori Imaizumi

Hiroshima University
Japan

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name Citations Publications World Ranking National Ranking

Biology and Biochemistry D-index 50 Citations 12,688 156 World Ranking 12947 National Ranking 959

Overview

What is he best known for?

The fields of study Kazunori Imaizumi is best known for:

Gene
Enzyme
Apoptosis

Cell biology is closely attributed to Kinase in his research. His Kinase study frequently links to other fields, such as Cell biology. His research on Endoplasmic reticulum frequently links to adjacent areas such as Activating transcription factor. Activating transcription factor and Unfolded protein response are commonly linked in his work. His ATF6 research extends to the thematically linked field of Unfolded protein response. His Endoplasmic reticulum research extends to ATF6, which is thematically connected. Borrowing concepts from Mutant, he weaves in ideas under Gene. He performs integrative study on Mutant and Gene. His research on Biochemistry often connects related topics like Messenger RNA.

His most cited work include:

Autophagy Is Activated for Cell Survival after Endoplasmic ReticulumStress (1581 citations)
Involvement of caspase-4 in endoplasmic reticulum stress-induced apoptosis and Aβ-induced cell death (771 citations)
Activation of Caspase-12, an Endoplastic Reticulum (ER) Resident Caspase, through Tumor Necrosis Factor Receptor-associated Factor 2-dependent Mechanism in Response to the ER Stress (759 citations)

What are the main themes of his work throughout his whole career to date

While working on this project, Kazunori Imaizumi studies both Cell biology and Neuroscience. Kazunori Imaizumi connects Neuroscience with Cell biology in his research. His work often combines Gene and Gene expression studies. As part of his studies on Endoplasmic reticulum, he frequently links adjacent subjects like Genetics. His studies link Unfolded protein response with Genetics. Kazunori Imaizumi performs integrative Unfolded protein response and Endoplasmic reticulum research in his work. Kazunori Imaizumi performs integrative study on Biochemistry and Molecular biology in his works. In his work, he performs multidisciplinary research in Molecular biology and Gene. In his work, he performs multidisciplinary research in Internal medicine and Endocrinology.

Kazunori Imaizumi most often published in these fields:

Cell biology (84.85%)
Gene (72.73%)
Endoplasmic reticulum (58.59%)

What were the highlights of his more recent work (between 2017-2021)?

Cell biology (87.50%)
Biochemistry (87.50%)
Gene (75.00%)

In recent works Kazunori Imaizumi was focusing on the following fields of study:

His work on Homeostasis expands to the thematically related Cell biology. His Homeostasis study frequently draws connections to other fields, such as Cell biology. Biochemistry is closely attributed to Protein degradation in his study. His Protein degradation study frequently draws parallels with other fields, such as Biochemistry. He conducts interdisciplinary study in the fields of Gene and Golgi apparatus through his works. By researching both Golgi apparatus and Endoplasmic reticulum, he produces research that crosses academic boundaries. Kazunori Imaizumi connects Endoplasmic reticulum with Calcium signaling in his study. Kazunori Imaizumi integrates many fields, such as Calcium signaling and Signal transduction, in his works. His Genetics research extends to the thematically linked field of Signal transduction.

Between 2017 and 2021, his most popular works were:

Unfolded Protein Response-Dependent Communication and Contact among Endoplasmic Reticulum, Mitochondria, and Plasma Membrane (35 citations)
Neuronal activity-dependent local activation of dendritic unfolded protein response promotes expression of brain-derived neurotrophic factor in cell soma (29 citations)
The Role of Tissue-Specific Ubiquitin Ligases, RNF183, RNF186, RNF182 and RNF152, in Disease and Biological Function (22 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Autophagy Is Activated for Cell Survival after Endoplasmic Reticulum Stress

Maiko Ogata;Shin Ichiro Hino;Atsushi Saito;Keisuke Morikawa.
Molecular and Cellular Biology (2006)

1959 Citations

Activation of caspase-12, an endoplastic reticulum (ER) resident caspase, through tumor necrosis factor receptor-associated factor 2-dependent mechanism in response to the ER stress.

Takunari Yoneda;Kazunori Imaizumi;Kayoko Oono;Daishi Yui.
Journal of Biological Chemistry (2001)

1159 Citations

Involvement of caspase-4 in endoplasmic reticulum stress-induced apoptosis and Aβ-induced cell death

Junichi Hitomi;Junichi Hitomi;Taiichi Katayama;Taiichi Katayama;Yutaka Eguchi;Yutaka Eguchi;Takashi Kudo.
Journal of Cell Biology (2004)

922 Citations

Induction of neuronal death by ER stress in Alzheimer's disease.

Taiichi Katayama;Kazunori Imaizumi;Takayuki Manabe;Junichi Hitomi.
Journal of Chemical Neuroanatomy (2004)

393 Citations

Angiotensin II Type 1a Receptor-deficient Mice with Hypotension and Hyperreninemia

Takeshi Sugaya;Shin-ichiro Nishimatsu;Keiji Tanimoto;Eriko Takimoto.
Journal of Biological Chemistry (1995)

386 Citations

Obesity-induced endoplasmic reticulum stress causes chronic inflammation in adipose tissue

Noritaka Kawasaki;Rie Asada;Atsushi Saito;Soshi Kanemoto.
Scientific Reports (2012)

352 Citations

A molecular chaperone inducer protects neurons from ER stress

T Kudo;S Kanemoto;S Kanemoto;H Hara;N Morimoto.
Cell Death & Differentiation (2008)

327 Citations

Signalling mediated by the endoplasmic reticulum stress transducer OASIS is involved in bone formation.

Tomohiko Murakami;Atsushi Saito;Shin Ichiro Hino;Shinichi Kondo.
Nature Cell Biology (2009)

316 Citations

Apoptosis induced by endoplasmic reticulum stress depends on activation of caspase-3 via caspase-12.

Junichi Hitomi;Taiichi Katayama;Manabu Taniguchi;Akiko Honda.
Neuroscience Letters (2004)

311 Citations

OASIS, a CREB/ATF-family member, modulates UPR signalling in astrocytes

Shinichi Kondo;Tomohiko Murakami;Tomohiko Murakami;Kouko Tatsumi;Maiko Ogata;Maiko Ogata.
Nature Cell Biology (2005)

299 Citations

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Frequent Co-Authors

External Links

Personal Website for Kazunori Imaizumi