D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 63 Citations 15,627 178 World Ranking 6553 National Ranking 3056

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Enzyme
  • Genetics

Julie K. Andersen mainly focuses on Substantia nigra, Oxidative stress, Parkinson's disease, Dopaminergic and Neurodegeneration. Julie K. Andersen interconnects Neurotoxicity and MPTP in the investigation of issues within Substantia nigra. Her Oxidative stress study integrates concerns from other disciplines, such as Mitochondrion and Cell biology.

As part of her studies on Parkinson's disease, Julie K. Andersen often connects relevant subjects like Neuroscience. Her Dopaminergic study is concerned with the field of Dopamine as a whole. Julie K. Andersen combines subjects such as Glutathione and Protein degradation with her study of Endocrinology.

Her most cited work include:

  • Oxidative stress in neurodegeneration: cause or consequence? (1373 citations)
  • Mice Deficient in Cellular Glutathione Peroxidase Show Increased Vulnerability to Malonate, 3-Nitropropionic Acid, and 1-Methyl-4-Phenyl-1,2,5,6-Tetrahydropyridine (1050 citations)
  • Genetic or Pharmacological Iron Chelation Prevents MPTP-Induced Neurotoxicity In Vivo: A Novel Therapy for Parkinson's Disease (512 citations)

What are the main themes of her work throughout her whole career to date?

Her scientific interests lie mostly in Oxidative stress, Parkinson's disease, Cell biology, Substantia nigra and Dopaminergic. Her Oxidative stress study combines topics in areas such as Alzheimer's disease and Molecular biology. Her Parkinson's disease study incorporates themes from Paraquat, Neurodegeneration and Pharmacology.

Her research in Cell biology intersects with topics in Calcium, Apoptosis, Programmed cell death and Caenorhabditis elegans. The concepts of her Substantia nigra study are interwoven with issues in Ferritin, Mitochondrion, MPTP and Neuroprotection. Her Dopaminergic study deals with Glutathione intersecting with Cysteine, Antioxidant and Protein degradation.

She most often published in these fields:

  • Oxidative stress (37.79%)
  • Parkinson's disease (34.88%)
  • Cell biology (31.98%)

What were the highlights of her more recent work (between 2013-2021)?

  • Cell biology (31.98%)
  • Parkinson's disease (34.88%)
  • Pharmacology (16.86%)

In recent papers she was focusing on the following fields of study:

Julie K. Andersen mainly investigates Cell biology, Parkinson's disease, Pharmacology, Neurodegeneration and Neuroscience. Her studies in Cell biology integrate themes in fields like Autophagy, Regulator, Neurotoxin and Caenorhabditis elegans. She studies Parkinson's disease, namely Substantia nigra.

Her Substantia nigra study is associated with Dopaminergic. Julie K. Andersen has included themes like Glycogen synthase and MPTP in her Pharmacology study. Her Neuroscience research is multidisciplinary, incorporating elements of Parkin and Disease.

Between 2013 and 2021, her most popular works were:

  • Cellular Senescence Is Induced by the Environmental Neurotoxin Paraquat and Contributes to Neuropathology Linked to Parkinson's Disease (146 citations)
  • Cellular senescence and the aging brain. (135 citations)
  • Mitochondrial Quality Control via the PGC1α-TFEB Signaling Pathway Is Compromised by Parkin Q311X Mutation But Independently Restored by Rapamycin (66 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Genetics

The scientist’s investigation covers issues in Cell biology, Neuroprotection, Substantia nigra, Pharmacology and Neurodegeneration. Her Cell biology study combines topics from a wide range of disciplines, such as Regulator, Biochemistry and Caenorhabditis elegans. Her study looks at the relationship between Neuroprotection and topics such as MPTP, which overlap with Agonist, Dopaminergic Cell, Downregulation and upregulation and Pars compacta.

Her Substantia nigra study is concerned with the larger field of Dopaminergic. As a part of the same scientific study, Julie K. Andersen usually deals with the Dopaminergic, concentrating on Parkinson's disease and frequently concerns with Lithium, Genetically modified mouse and Neuroscience. Her Pharmacology research is multidisciplinary, incorporating perspectives in Oxidative stress and Neurotoxicity.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Oxidative stress in neurodegeneration: cause or consequence?

Julie K Andersen.
Nature Medicine (2004)

2177 Citations

Mice Deficient in Cellular Glutathione Peroxidase Show Increased Vulnerability to Malonate, 3-Nitropropionic Acid, and 1-Methyl-4-Phenyl-1,2,5,6-Tetrahydropyridine

Peter Klivenyi;Ole A. Andreassen;Robert J. Ferrante;Alpaslan Dedeoglu.
The Journal of Neuroscience (2000)

1080 Citations

Genetic or Pharmacological Iron Chelation Prevents MPTP-Induced Neurotoxicity In Vivo: A Novel Therapy for Parkinson's Disease

Deepinder Kaur;Ferda Yantiri;Subramanian Rajagopalan;Jyothi Kumar.
Neuron (2003)

727 Citations

Glutathione, iron and Parkinson’s disease

Srinivas Bharath;Michael Hsu;Deepinder Kaur;Subramanian Rajagopalan.
Biochemical Pharmacology (2002)

518 Citations

Cellular senescence: a link between cancer and age-related degenerative disease?

Judith Campisi;Julie K. Andersen;Pankaj Kapahi;Simon Melov.
Seminars in Cancer Biology (2011)

422 Citations

Mitochondrial alpha-synuclein accumulation impairs complex I function in dopaminergic neurons and results in increased mitophagy in vivo

Shankar J. Chinta;Jyothi K. Mallajosyula;Anand Rane;Julie K. Andersen.
Neuroscience Letters (2010)

402 Citations

Caspase-9 activation results in downstream caspase-8 activation and bid cleavage in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced Parkinson's disease.

Veena Viswanath;Yongqin Wu;Rapee Boonplueang;Rapee Boonplueang;Sylvia Chen.
The Journal of Neuroscience (2001)

365 Citations

Redox imbalance in Parkinson's disease

Shankar J. Chinta;Julie K. Andersen.
Biochimica et Biophysica Acta (2008)

331 Citations

Glutathione Depletion in PC12 Results in Selective Inhibition of Mitochondrial Complex I Activity IMPLICATIONS FOR PARKINSON′S DISEASE

Nandita Jha;Octavian Jurma;Giovanna Lalli;Yi Liu.
Journal of Biological Chemistry (2000)

312 Citations

Cognitive efficiency on a match to sample task decreases at the onset of puberty in children.

Robert F McGivern;Julie Andersen;Desiree Byrd;Kandis L Mutter.
Brain and Cognition (2002)

304 Citations

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