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Biology and Biochemistry

D-Index
69
Citations
18989
World Ranking
7316
National Ranking
3351

Overview

Julie K. Andersen is affiliated with the Buck Institute for Research on Aging in the United States. Their research contributions cover several domains primarily within biochemistry, genetics, molecular biology, and medicine, with a particular focus on aging and related molecular mechanisms.

The scientist's recent published papers include:

  • Taurine deficiency as a driver of aging, 2023, Science
  • mTORC2: The other mTOR in autophagy regulation, 2021, Aging Cell
  • Alpha-Synuclein Preformed Fibrils Induce Cellular Senescence in Parkinson's Disease Models, 2021, Cells
  • The intestinal immune system and gut barrier function in obesity and ageing, 2022, FEBS Journal
  • The mitochondrial permeability transition pore activates the mitochondrial unfolded protein response and promotes aging, 2021, eLife

Their frequent co-authors include:

  • Gordon J. Lithgow
  • Manish Chamoli
  • Anand Rane
  • Anna Foulger
  • Shankar J. Chinta

The scientist publishes regularly in several venues, with multiple papers appearing in:

  • bioRxiv (Cold Spring Harbor Laboratory)
  • GeroScience
  • Aging Cell
  • PubMed
  • Journal of Translational Medicine

The main fields of study represented in Julie K. Andersen's work are:

  • Biochemistry, Genetics and Molecular Biology
  • Medicine

Within these fields, they focus on subfields including:

  • Molecular Biology
  • Physiology
  • Aging
  • Neurology
  • Nutrition and Dietetics

Key research topics covered in their publications include:

  • Genetics, Aging, and Longevity in Model Organisms
  • Alzheimer's disease research and treatments
  • Mitochondrial Function and Pathology
  • Telomeres, Telomerase, and Senescence
  • Pomegranate: compositions and health benefits
  • Neuroinflammation and Neurodegeneration Mechanisms
  • Autophagy in Disease and Therapy

Best Publications

  • Oxidative stress in neurodegeneration: cause or consequence?

    Julie K Andersen

  • Mice Deficient in Cellular Glutathione Peroxidase Show Increased Vulnerability to Malonate, 3-Nitropropionic Acid, and 1-Methyl-4-Phenyl-1,2,5,6-Tetrahydropyridine

    Peter Klivenyi;Ole A. Andreassen;Robert J. Ferrante;Alpaslan Dedeoglu

  • Genetic or Pharmacological Iron Chelation Prevents MPTP-Induced Neurotoxicity In Vivo: A Novel Therapy for Parkinson's Disease

    Deepinder Kaur;Ferda Yantiri;Subramanian Rajagopalan;Jyothi Kumar

  • Glutathione, iron and Parkinson’s disease

    Srinivas Bharath;Michael Hsu;Deepinder Kaur;Subramanian Rajagopalan

  • Cellular senescence: a link between cancer and age-related degenerative disease?

    Judith Campisi;Julie K. Andersen;Pankaj Kapahi;Simon Melov

  • Mitochondrial alpha-synuclein accumulation impairs complex I function in dopaminergic neurons and results in increased mitophagy in vivo

    Shankar J. Chinta;Jyothi K. Mallajosyula;Anand Rane;Julie K. Andersen

  • Cellular Senescence Is Induced by the Environmental Neurotoxin Paraquat and Contributes to Neuropathology Linked to Parkinson's Disease

    Shankar J. Chinta;Georgia Woods;Marco Demaria;Anand Rane

  • Caspase-9 activation results in downstream caspase-8 activation and bid cleavage in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced Parkinson's disease.

    Veena Viswanath;Yongqin Wu;Rapee Boonplueang;Rapee Boonplueang;Sylvia Chen

  • Redox imbalance in Parkinson's disease

    Shankar J. Chinta;Julie K. Andersen

  • Cell in focusDopaminergic neurons

    Shankar J. Chinta;Julie K. Andersen

  • Role of oxidative stress in paraquat‐induced dopaminergic cell degeneration

    Alison L. McCormack;Jennifer G. Atienza;Louisa C. Johnston;Julie K. Andersen

  • Glutathione Depletion in PC12 Results in Selective Inhibition of Mitochondrial Complex I Activity IMPLICATIONS FOR PARKINSON′S DISEASE

    Nandita Jha;Octavian Jurma;Giovanna Lalli;Yi Liu

  • Cognitive efficiency on a match to sample task decreases at the onset of puberty in children.

    Robert F McGivern;Julie Andersen;Desiree Byrd;Kandis L Mutter

  • MAO-B Elevation in Mouse Brain Astrocytes Results in Parkinson's Pathology

    Jyothi K. Mallajosyula;Deepinder Kaur;Shankar J. Chinta;Subramanian Rajagopalan

  • Cellular senescence and the aging brain.

    Shankar J Chinta;Georgia Woods;Anand Rane;Marco Demaria

  • The herbicide paraquat induces dopaminergic nigral apoptosis through sustained activation of the JNK pathway.

    Jun Peng;Xiao Ou Mao;Fang Feng Stevenson;Michael Hsu

  • Time to Talk SENS: Critiquing the Immutability of Human Aging

    Aubrey D. N. J. De Grey;Bruce N. Ames;Julie K. Andersen;Andrzej Bartke

  • Oxidative and nitrative protein modifications in Parkinson's disease.

    Steven R. Danielson;Julie K. Andersen

  • Environmental stress, ageing and glial cell senescence: a novel mechanistic link to Parkinson's disease?

    Shankar J Chinta;Christopher A Lieu;Marco DeMaria;Remi-Martin Laberge

  • Superoxide Dismutase/Catalase Mimetics Are Neuroprotective against Selective Paraquat-mediated Dopaminergic Neuron Death in the Substantial Nigra IMPLICATIONS FOR PARKINSON DISEASE

    Jun Peng;Fang Feng Stevenson;Susan R. Doctrow;Julie K. Andersen

Frequent Co-Authors

Gordon J. Lithgow
Gordon J. Lithgow Buck Institute for Research on Aging
Xandra O. Breakefield
Xandra O. Breakefield Harvard University
David G. Nicholls
David G. Nicholls Buck Institute for Research on Aging
Ole Isacson
Ole Isacson Harvard University
Judith Campisi
Judith Campisi Buck Institute for Research on Aging
M. Flint Beal
M. Flint Beal Cornell University
Simon Melov
Simon Melov Buck Institute for Research on Aging
Bradford W. Gibson
Bradford W. Gibson Buck Institute for Research on Aging
Pankaj Kapahi
Pankaj Kapahi Buck Institute for Research on Aging
Marco Demaria
Marco Demaria University Medical Center Groningen

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