2012 - Fellow of the American Association for the Advancement of Science (AAAS)
2012 - Fellow, National Academy of Inventors
Howard J. Federoff spends much of his time researching Neuroscience, Cell biology, Programmed cell death, Substantia nigra and Internal medicine. Howard J. Federoff has included themes like Neurotrophic factors and Neurite in his Neuroscience study. The concepts of his Cell biology study are interwoven with issues in Microglia, Excitotoxicity, Molecular biology, Apoptosis-inducing factor and Proinflammatory cytokine.
His work deals with themes such as Gene expression, Transgene, Ischemia and Neuroprotection, which intersect with Programmed cell death. His studies deal with areas such as Glial cell line-derived neurotrophic factor and Putamen as well as Substantia nigra. He studied Internal medicine and Endocrinology that intersect with Nerve growth factor.
Howard J. Federoff focuses on Virology, Molecular biology, Disease, Herpes simplex virus and Neuroscience. His Virology study combines topics in areas such as Genetic enhancement and Immune system. His research integrates issues of Gene expression and Transgene in his study of Molecular biology.
As part of the same scientific family, Howard J. Federoff usually focuses on Disease, concentrating on Immunology and intersecting with Cell biology, Genetically modified mouse and Amyloid. His Cell biology study integrates concerns from other disciplines, such as Programmed cell death and Nerve growth factor. His Herpes simplex virus research is multidisciplinary, incorporating perspectives in Cell culture, Oncolytic virus and In vivo.
His primary areas of investigation include Disease, Bioinformatics, Internal medicine, Parkinson's disease and Memory impairment. His Disease research incorporates themes from Microvesicles, Immunology, Biomarker, Pharmacology and Cohort. His biological study spans a wide range of topics, including Genetic enhancement and Neurodegeneration.
He interconnects Endocrinology and Oncology in the investigation of issues within Internal medicine. Many of his research projects under Parkinson's disease are closely connected to Genome-wide association study with Genome-wide association study, tying the diverse disciplines of science together. His Substantia nigra study integrates concerns from other disciplines, such as Neuroinflammation, Microglia and Alpha-synuclein.
His main research concerns Disease, Parkinson's disease, Bioinformatics, Internal medicine and Substantia nigra. His Disease research is mostly focused on the topic Alzheimer's disease. His study on Parkinson's disease also encompasses disciplines like
In his research, Apolipoprotein E is intimately related to Oncology, which falls under the overarching field of Internal medicine. His Substantia nigra study introduces a deeper knowledge of Dopamine. His Microglia study frequently draws connections between related disciplines such as Neuroscience.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Mediation of poly(ADP-ribose) polymerase-1-dependent cell death by apoptosis-inducing factor.
Seong Woon Yu;Hongmin Wang;Marc F. Poitras;Carmen Coombs.
Science (2002)
Plasma phospholipids identify antecedent memory impairment in older adults
Mark Mapstone;Amrita K Cheema;Massimo S Fiandaca;Xiaogang Zhong.
Nature Medicine (2014)
PGC-1α, A Potential Therapeutic Target for Early Intervention in Parkinson’s Disease
Bin Zheng;Zhixiang Liao;Joseph J. Locascio;Kristen A. Lesniak.
Science Translational Medicine (2010)
Identification of preclinical Alzheimer's disease by a profile of pathogenic proteins in neurally derived blood exosomes: A case-control study
Massimo S. Fiandaca;Dimitrios Kapogiannis;Mark Mapstone;Adam Boxer.
Alzheimers & Dementia (2015)
Paraquat elicited neurobehavioral syndrome caused by dopaminergic neuron loss.
A.I Brooks;C.A Chadwick;H.A Gelbard;D.A Cory-Slechta.
Brain Research (1999)
Regulation of neuronal traits by a novel transcriptional complex.
Nurit Ballas;Elena Battaglioli;Fouad Atouf;Maria E. Andres.
Neuron (2001)
Synuclein activates microglia in a model of Parkinson’s Disease
Xiaomin Su;Kathleen A. Maguire-Zeiss;Rita Giuliano;Landa Prifti.
Neurobiology of Aging (2008)
Expression of bcl-2 From a Defective Herpes Simplex Virus–1 Vector Limits Neuronal Death in Focal Cerebral Ischemia
Matthew D. Linnik;Peter Zahos;Michael D. Geschwind;Howard J. Federoff.
Stroke (1995)
Regulated release and polarized localization of brain-derived neurotrophic factor in hippocampal neurons.
Laurie J. Goodman;Janet Valverde;Filip Lim;Michael D. Geschwind.
Molecular and Cellular Neuroscience (1996)
Inhibitors of leucine-rich repeat kinase-2 protect against models of Parkinson's disease
Byoung Dae Lee;Joo Ho Shin;Jackalina Vankampen;Leonard Petrucelli.
Nature Medicine (2010)
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