What is he best known for?
The fields of study he is best known for:
Genetics, LRRK2, Mitochondrion, Cell biology and Parkinson's disease are his primary areas of study.
His LRRK2 research is multidisciplinary, incorporating elements of Leucine-rich repeat and Kinase activity.
His Mitochondrion research integrates issues from Oxidative stress, PINK1 and Molecular biology.
He has researched Cell biology in several fields, including Parkin, Neurodegeneration and Substantia nigra.
His study of Alpha-synuclein is a part of Parkinson's disease.
His Disease research includes elements of Neurology, Neuroscience and Toxicity.
His most cited work include:
- α-Synuclein Locus Triplication Causes Parkinson's Disease (3413 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
- PINK1 is selectively stabilized on impaired mitochondria to activate Parkin. (1824 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Genetics, LRRK2, Cell biology, Disease and Parkinson's disease.
His study in Genetics is interdisciplinary in nature, drawing from both Parkin and Parkinsonism.
His research integrates issues of Leucine-rich repeat, Kinase, Kinase activity and GTPase in his study of LRRK2.
His biological study deals with issues like PINK1, which deal with fields such as mitochondrial fusion.
His Disease research is multidisciplinary, relying on both Neuroscience and Bioinformatics.
His work is connected to Alpha-synuclein and Synuclein, as a part of Parkinson's disease.
He most often published in these fields:
- Genetics (31.94%)
- LRRK2 (30.56%)
- Cell biology (26.39%)
What were the highlights of his more recent work (between 2018-2021)?
- LRRK2 (30.56%)
- Disease (20.49%)
- Cell biology (26.39%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include LRRK2, Disease, Cell biology, Parkinson's disease and Genetics.
His research in LRRK2 intersects with topics in GTPase, Rab and Kinase, Kinase activity.
His Disease research includes themes of Genome-wide association study, Computational biology, Gene and Oncology.
His work in the fields of Phosphorylation overlaps with other areas such as Voltage-dependent anion channel.
His Parkinson's disease study also includes fields such as
- Function and related Parkin, PINK1, Isogenic human disease models and Gene knockout,
- Protein kinase A which intersects with area such as Astrogliosis.
In his articles, Mark R. Cookson combines various disciplines, including Genetics and DNAJC5.
Between 2018 and 2021, his most popular works were:
- Identification of novel risk loci, causal insights, and heritable risk for Parkinson's disease: a meta-analysis of genome-wide association studies (353 citations)
- LRRK2 links genetic and sporadic Parkinson's disease (54 citations)
- The role of monogenic genes in idiopathic Parkinson's disease. (47 citations)
In his most recent research, the most cited papers focused on:
Mark R. Cookson mostly deals with LRRK2, Disease, Cell biology, Genetics and Parkinson's disease.
His work deals with themes such as Oxidative phosphorylation, Kinase activity, Neuroinflammation, SOD2 and Neuroscience, which intersect with LRRK2.
Mark R. Cookson has included themes like GTPase, Rab and Gene in his Disease study.
His Cell biology study incorporates themes from Vesicle and Lysosome.
Mark R. Cookson combines Genetics and DNAJC5 in his research.
His Parkinson's disease study combines topics from a wide range of disciplines, such as Phagocytosis, Phagocytic dysfunction, Molecular medicine, Degeneration and Apoptosis.
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