2013 - APA Award for Distinguished Scientific Contributions to Psychology, American Psychological Association
1996 - Fellow of the American Association for the Advancement of Science (AAAS)
1987 - Fellow of Alfred P. Sloan Foundation
1987 - Fellow of the MacArthur Foundation
His primary areas of investigation include Endocrinology, Internal medicine, Glucocorticoid, Hippocampus and Neuroscience. His Receptor research extends to the thematically linked field of Endocrinology. As part of the same scientific family, Robert M. Sapolsky usually focuses on Glucocorticoid, concentrating on Hydrocortisone and intersecting with Social status and Baboon.
The various areas that Robert M. Sapolsky examines in his Hippocampus study include Kainic acid, NMDA receptor, Atrophy, Neuron death and Depression. The concepts of his Neuroscience study are interwoven with issues in Synaptic plasticity and Calcium. The various areas that Robert M. Sapolsky examines in his Hippocampal formation study include Glutamate receptor, Neurotoxicity, Neuron and In vivo.
Robert M. Sapolsky focuses on Internal medicine, Endocrinology, Glucocorticoid, Neuroscience and Hippocampal formation. Kainic acid, Glutamate receptor, Central nervous system, Hormone and Steroid hormone are among the areas of Internal medicine where the researcher is concentrating his efforts. His Endocrinology research incorporates themes from Receptor and Neurotoxicity.
Robert M. Sapolsky has researched Glucocorticoid in several fields, including Hydrocortisone, Basal, Dexamethasone and Amygdala. The Neuroscience study combines topics in areas such as Neuron death and Programmed cell death. His Neuroprotection study also includes fields such as
Robert M. Sapolsky mostly deals with Neuroscience, Immunology, Internal medicine, Endocrinology and Developmental psychology. His Neuroscience research focuses on subjects like Neuron death, which are linked to Glutamate receptor, Kainic acid, Neurotoxicity and Excitotoxicity. His Immunology research includes elements of Stroke, PI3K/AKT/mTOR pathway and Cell biology.
Robert M. Sapolsky merges Endocrinology with Cutaneous irritation in his study. His Inflammation study integrates concerns from other disciplines, such as Tumor necrosis factor alpha and Glucocorticoid. His Glucocorticoid research incorporates elements of Myeloid and Caveolin 1.
The scientist’s investigation covers issues in Neuroscience, Toxoplasma gondii, Amygdala, Hippocampus and Immunology. In the field of Neuroscience, his study on Neurogenesis and Hippocampal formation overlaps with subjects such as Truism, Inverted u and Abstract thoughts. His study in Hippocampus is interdisciplinary in nature, drawing from both Suprachiasmatic nucleus and Chronobiology.
Many of his research projects under Immunology are closely connected to Sexual attraction with Sexual attraction, tying the diverse disciplines of science together. His work in Dentate gyrus addresses subjects such as Glucocorticoid receptor, which are connected to disciplines such as Corticosterone. Robert M. Sapolsky is investigating Endocrinology and Internal medicine as part of his examination of Chronic stress.
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How do glucocorticoids influence stress responses? Integrating permissive, suppressive, stimulatory, and preparative actions.
Robert M. Sapolsky;L. Michael Romero;Allan U. Munck.
Endocrine Reviews (2000)
The Neuroendocrinology of Stress and Aging: The Glucocorticoid Cascade Hypothesis*
Robert M. Sapolsky;Lewis C. Krey;Bruce S. McEwen.
Endocrine Reviews (1986)
Glucocorticoids and hippocampal atrophy in neuropsychiatric disorders.
Robert M. Sapolsky.
Archives of General Psychiatry (2000)
Stress and cognitive function
Bruce S McEwen;Robert M Sapolsky.
Current Opinion in Neurobiology (1995)
The role of the hippocampus in feedback regulation of the hypothalamic-pituitary-adrenocortical axis.
Lauren Jacobson;Robert Sapolsky.
Endocrine Reviews (1991)
The influence of social hierarchy on primate health
Robert M. Sapolsky.
Science (2005)
Why Stress Is Bad for Your Brain
Robert M. Sapolsky.
Science (1996)
Interleukin-1 stimulates the secretion of hypothalamic corticotropin-releasing factor.
Robert Sapolsky;Catherine Rivier;Gayle Yamamoto;Paul Plotsky.
Science (1987)
Prolonged glucocorticoid exposure reduces hippocampal neuron number: implications for aging
RM Sapolsky;LC Krey;BS McEwen.
The Journal of Neuroscience (1985)
Hippocampal damage associated with prolonged glucocorticoid exposure in primates
RM Sapolsky;H Uno;CS Rebert;CE Finch.
The Journal of Neuroscience (1990)
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