Joel A. Black mostly deals with Sodium channel, Neuroscience, Dorsal root ganglion, Nav1.9 and Endocrinology. Joel A. Black has included themes like Nociception, Sensory neuron, In situ hybridization and Tetrodotoxin in his Sodium channel study. His research integrates issues of Ion channel and Cell biology in his study of Neuroscience.
His Dorsal root ganglion research is multidisciplinary, incorporating elements of Electrophysiology, Patch clamp and Axotomy. His Nav1.9 research incorporates themes from Biophysics, Membrane potential and Resting potential. Many of his studies involve connections with topics such as Internal medicine and Endocrinology.
Joel A. Black mainly focuses on Sodium channel, Neuroscience, Cell biology, Dorsal root ganglion and Endocrinology. The study incorporates disciplines such as In situ hybridization, Nav1.9, Astrocyte, Ion channel and Spinal cord in addition to Sodium channel. The Cell biology study which covers Nerve growth factor that intersects with Neurotrophin.
His Dorsal root ganglion research includes elements of Patch clamp, Axotomy, Sensory neuron and Nociception. His biological study spans a wide range of topics, including Anesthesia, Internal medicine and NAV1. His Internal medicine study combines topics in areas such as Sodium-calcium exchanger and Downregulation and upregulation.
Joel A. Black mainly investigates Sodium channel, Neuroscience, Cell biology, NAV1 and Multiple sclerosis. The various areas that he examines in his Sodium channel study include Mutation, Neuropathic pain, Microglia and Dorsal root ganglion. His Dorsal root ganglion research is multidisciplinary, incorporating perspectives in Axotomy and PEPD.
His Neuroscience research is multidisciplinary, relying on both Sodium channel blocker, Sodium-calcium exchanger, Neurite and Tetrodotoxin. His research in Cell biology intersects with topics in Glycogen and Compound muscle action potential. Joel A. Black combines subjects such as Endocrinology, Sensory system, Internal medicine, Chronic pain and Spinal Cord Dorsal Horn with his study of NAV1.
His primary areas of study are Neuroscience, Sodium channel, Paroxysmal extreme pain disorder, Sodium channel blocker and Neuropathic pain. His study in Neuroscience is interdisciplinary in nature, drawing from both Tetrodotoxin, Phagocytosis, Cell biology, NAV1 and Mutation. The concepts of his Cell biology study are interwoven with issues in Compound muscle action potential and Sciatic nerve.
His work carried out in the field of Mutation brings together such families of science as Pain therapy and Ion channel. In his works, he undertakes multidisciplinary study on Sodium channel and Peripheral neuropathy. His Sodium channel blocker research incorporates elements of Nav1.9, Nociceptor, Sensory Receptor Cells, Sensory neuron and Peripheral nervous system.
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Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotomy.
Stephen G. Waxman;Jeffery D. Kocsis;Joel A. Black.
Journal of Neurophysiology (1994)
NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy
S. D. Dib-Hajj;L. Tyrrell;J. A. Black;S. G. Waxman.
Proceedings of the National Academy of Sciences of the United States of America (1998)
Sodium Channels in Normal and Pathological Pain
Sulayman D. Dib-Hajj;Theodore R. Cummins;Joel A. Black;Stephen G. Waxman.
Annual Review of Neuroscience (2010)
Molecular changes in neurons in multiple sclerosis: altered axonal expression of Nav1.2 and Nav1.6 sodium channels and Na+/Ca2+ exchanger.
Matthew J. Craner;Jia Newcombe;Joel A. Black;Caroline Hartle.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Spinal sensory neurons express multiple sodium channel α-subunit mRNAs
J.A Black;S Dib-Hajj;S Dib-Hajj;K McNabola;K McNabola;S Jeste;S Jeste.
Molecular Brain Research (1996)
The Na(V)1.7 sodium channel: from molecule to man.
Sulayman D. Dib-Hajj;Yang Yang;Yang Yang;Joel A. Black;Joel A. Black;Stephen G. Waxman;Stephen G. Waxman.
Nature Reviews Neuroscience (2013)
A novel persistent tetrodotoxin-resistant sodium current in SNS-null and wild-type small primary sensory neurons.
Theodore R. Cummins;Sulayman D. Dib-Hajj;Joel A. Black;Armen N. Akopian.
The Journal of Neuroscience (1999)
Sodium channel α-subunit mRNAs I, II, III, NaG, Na6 and hNE (PN1): Different expression patterns in developing rat nervous system
P.A. Felts;S. Yokoyama;S. Yokoyama;S. Yokoyama;S. Dib-Hajj;S. Dib-Hajj;J.A. Black;J.A. Black.
Molecular Brain Research (1997)
Sodium channels and pain
S. G. Waxman;S. Dib-Hajj;T. R. Cummins;J. A. Black.
Proceedings of the National Academy of Sciences of the United States of America (1999)
Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain.
Joel A Black;Shujun Liu;Masaki Tanaka;Theodore R Cummins.
Pain (2004)
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