D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 84 Citations 22,243 176 World Ranking 786 National Ranking 434
Medicine D-index 84 Citations 22,379 182 World Ranking 10148 National Ranking 5346

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Neuron
  • Central nervous system

Joel A. Black mostly deals with Sodium channel, Neuroscience, Dorsal root ganglion, Nav1.9 and Endocrinology. Joel A. Black has included themes like Nociception, Sensory neuron, In situ hybridization and Tetrodotoxin in his Sodium channel study. His research integrates issues of Ion channel and Cell biology in his study of Neuroscience.

His Dorsal root ganglion research is multidisciplinary, incorporating elements of Electrophysiology, Patch clamp and Axotomy. His Nav1.9 research incorporates themes from Biophysics, Membrane potential and Resting potential. Many of his studies involve connections with topics such as Internal medicine and Endocrinology.

His most cited work include:

  • Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotomy. (488 citations)
  • Sodium Channels in Normal and Pathological Pain (435 citations)
  • A novel persistent tetrodotoxin-resistant sodium current in SNS-null and wild-type small primary sensory neurons. (394 citations)

What are the main themes of his work throughout his whole career to date?

Joel A. Black mainly focuses on Sodium channel, Neuroscience, Cell biology, Dorsal root ganglion and Endocrinology. The study incorporates disciplines such as In situ hybridization, Nav1.9, Astrocyte, Ion channel and Spinal cord in addition to Sodium channel. The Cell biology study which covers Nerve growth factor that intersects with Neurotrophin.

His Dorsal root ganglion research includes elements of Patch clamp, Axotomy, Sensory neuron and Nociception. His biological study spans a wide range of topics, including Anesthesia, Internal medicine and NAV1. His Internal medicine study combines topics in areas such as Sodium-calcium exchanger and Downregulation and upregulation.

He most often published in these fields:

  • Sodium channel (96.90%)
  • Neuroscience (77.88%)
  • Cell biology (54.42%)

What were the highlights of his more recent work (between 2011-2019)?

  • Sodium channel (96.90%)
  • Neuroscience (77.88%)
  • Cell biology (54.42%)

In recent papers he was focusing on the following fields of study:

Joel A. Black mainly investigates Sodium channel, Neuroscience, Cell biology, NAV1 and Multiple sclerosis. The various areas that he examines in his Sodium channel study include Mutation, Neuropathic pain, Microglia and Dorsal root ganglion. His Dorsal root ganglion research is multidisciplinary, incorporating perspectives in Axotomy and PEPD.

His Neuroscience research is multidisciplinary, relying on both Sodium channel blocker, Sodium-calcium exchanger, Neurite and Tetrodotoxin. His research in Cell biology intersects with topics in Glycogen and Compound muscle action potential. Joel A. Black combines subjects such as Endocrinology, Sensory system, Internal medicine, Chronic pain and Spinal Cord Dorsal Horn with his study of NAV1.

Between 2011 and 2019, his most popular works were:

  • The Na(V)1.7 sodium channel: from molecule to man. (356 citations)
  • The Na(V)1.7 sodium channel: from molecule to man. (356 citations)
  • A painful neuropathy-associated Nav1.7 mutant leads to time-dependent degeneration of small-diameter axons associated with intracellular Ca2+ dysregulation and decrease in ATP levels. (189 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Neuron
  • Internal medicine

His primary areas of study are Neuroscience, Sodium channel, Paroxysmal extreme pain disorder, Sodium channel blocker and Neuropathic pain. His study in Neuroscience is interdisciplinary in nature, drawing from both Tetrodotoxin, Phagocytosis, Cell biology, NAV1 and Mutation. The concepts of his Cell biology study are interwoven with issues in Compound muscle action potential and Sciatic nerve.

His work carried out in the field of Mutation brings together such families of science as Pain therapy and Ion channel. In his works, he undertakes multidisciplinary study on Sodium channel and Peripheral neuropathy. His Sodium channel blocker research incorporates elements of Nav1.9, Nociceptor, Sensory Receptor Cells, Sensory neuron and Peripheral nervous system.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotomy.

Stephen G. Waxman;Jeffery D. Kocsis;Joel A. Black.
Journal of Neurophysiology (1994)

767 Citations

NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy

S. D. Dib-Hajj;L. Tyrrell;J. A. Black;S. G. Waxman.
Proceedings of the National Academy of Sciences of the United States of America (1998)

757 Citations

Sodium Channels in Normal and Pathological Pain

Sulayman D. Dib-Hajj;Theodore R. Cummins;Joel A. Black;Stephen G. Waxman.
Annual Review of Neuroscience (2010)

605 Citations

Molecular changes in neurons in multiple sclerosis: altered axonal expression of Nav1.2 and Nav1.6 sodium channels and Na+/Ca2+ exchanger.

Matthew J. Craner;Jia Newcombe;Joel A. Black;Caroline Hartle.
Proceedings of the National Academy of Sciences of the United States of America (2004)

584 Citations

Spinal sensory neurons express multiple sodium channel α-subunit mRNAs

J.A Black;S Dib-Hajj;S Dib-Hajj;K McNabola;K McNabola;S Jeste;S Jeste.
Molecular Brain Research (1996)

523 Citations

The Na(V)1.7 sodium channel: from molecule to man.

Sulayman D. Dib-Hajj;Yang Yang;Yang Yang;Joel A. Black;Joel A. Black;Stephen G. Waxman;Stephen G. Waxman.
Nature Reviews Neuroscience (2013)

498 Citations

A novel persistent tetrodotoxin-resistant sodium current in SNS-null and wild-type small primary sensory neurons.

Theodore R. Cummins;Sulayman D. Dib-Hajj;Joel A. Black;Armen N. Akopian.
The Journal of Neuroscience (1999)

483 Citations

Sodium channel α-subunit mRNAs I, II, III, NaG, Na6 and hNE (PN1): Different expression patterns in developing rat nervous system

P.A. Felts;S. Yokoyama;S. Yokoyama;S. Yokoyama;S. Dib-Hajj;S. Dib-Hajj;J.A. Black;J.A. Black.
Molecular Brain Research (1997)

467 Citations

Sodium channels and pain

S. G. Waxman;S. Dib-Hajj;T. R. Cummins;J. A. Black.
Proceedings of the National Academy of Sciences of the United States of America (1999)

443 Citations

Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain.

Joel A Black;Shujun Liu;Masaki Tanaka;Theodore R Cummins.
Pain (2004)

435 Citations

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