What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Enzyme
Sodium channel, Neuroscience, Dorsal root ganglion, Paroxysmal extreme pain disorder and Nav1.9 are his primary areas of study.
Sulayman D. Dib-Hajj studies Sodium channel, namely NAV1.
His Neuroscience research integrates issues from Biophysics, Nociceptor and Ion channel.
His Dorsal root ganglion research incorporates elements of Anesthesia, Nociception, Molecular biology, Bursting and Tetrodotoxin.
The study incorporates disciplines such as Sodium channel blocker, Erythromelalgia, Chronic pain and Neuropathic pain in addition to Paroxysmal extreme pain disorder.
His Nav1.9 research incorporates themes from Membrane potential and Anatomy.
His most cited work include:
- NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy (477 citations)
- Sodium Channels in Normal and Pathological Pain (435 citations)
- A novel persistent tetrodotoxin-resistant sodium current in SNS-null and wild-type small primary sensory neurons. (394 citations)
What are the main themes of his work throughout his whole career to date?
Sulayman D. Dib-Hajj mainly focuses on Sodium channel, Neuroscience, Dorsal root ganglion, Erythromelalgia and NAV1.
His studies deal with areas such as Mutation, Nav1.9, Patch clamp and Cell biology as well as Sodium channel.
His Mutation study combines topics from a wide range of disciplines, such as Peripheral neuropathy and Mutant.
The study of Neuroscience is intertwined with the study of Nociceptor in a number of ways.
Sulayman D. Dib-Hajj usually deals with Dorsal root ganglion and limits it to topics linked to Neuropathic pain and Carbamazepine.
His Erythromelalgia research is multidisciplinary, incorporating elements of Sodium channel blocker and Channelopathy, Internal medicine, Sympathetic ganglion.
He most often published in these fields:
- Sodium channel (118.71%)
- Neuroscience (58.90%)
- Dorsal root ganglion (58.59%)
What were the highlights of his more recent work (between 2015-2021)?
- Sodium channel (118.71%)
- Neuroscience (58.90%)
- Dorsal root ganglion (58.59%)
In recent papers he was focusing on the following fields of study:
His primary scientific interests are in Sodium channel, Neuroscience, Dorsal root ganglion, Neuropathic pain and Chronic pain.
His Sodium channel study focuses on NAV1 in particular.
Sulayman D. Dib-Hajj regularly ties together related areas like Nav1.9 in his Neuroscience studies.
His Dorsal root ganglion research is multidisciplinary, incorporating perspectives in Peripheral neuropathy, Mutation, Channelopathy, Trigeminal ganglion and Pharmacology.
Sulayman D. Dib-Hajj combines subjects such as Electrophysiology and Mutant with his study of Mutation.
His study in Chronic pain is interdisciplinary in nature, drawing from both Anesthesia, Erythromelalgia, Pharmacotherapy and Bioinformatics.
Between 2015 and 2021, his most popular works were:
- The Role of Voltage-Gated Sodium Channels in Pain Signaling (112 citations)
- Subtype-Selective Small Molecule Inhibitors Reveal a Fundamental Role for Nav1.7 in Nociceptor Electrogenesis, Axonal Conduction and Presynaptic Release (89 citations)
- Pharmacological characterisation of the highly NaV1.7 selective spider venom peptide Pn3a. (81 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Enzyme
His main research concerns Neuroscience, Sodium channel, Chronic pain, Dorsal root ganglion and Erythromelalgia.
His Neuroscience research incorporates elements of Mutation and Nav1.9.
His Sodium channel study incorporates themes from Nociceptor, Patch clamp and Membrane potential.
He interconnects Peripheral nervous system, Anesthesia, Pharmacotherapy and Pharmacogenomics in the investigation of issues within Chronic pain.
His Dorsal root ganglion study integrates concerns from other disciplines, such as Neuropathic pain, Denervation, Mutant, Pathology and Intracellular.
Sulayman D. Dib-Hajj has included themes like Paroxysmal extreme pain disorder, Genetic model and Bioinformatics in his Erythromelalgia study.
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