The scientist’s investigation covers issues in Spinal cord injury, Spinal cord, Neuroscience, Sodium channel and Neuropathic pain. His Spinal cord injury study frequently links to related topics such as Anatomy. His studies deal with areas such as Anesthesia, Axon, Chronic pain and Neuron as well as Spinal cord.
His Pharmacology research extends to the thematically linked field of Neuroscience. His work on Sodium channel blocker as part of general Sodium channel study is frequently linked to Neuroglia, Immunology, Neuroprotection and Experimental autoimmune encephalomyelitis, bridging the gap between disciplines. His research in Neuropathic pain focuses on subjects like Sensory system, which are connected to Sciatic nerve and Nervous system.
Bryan C. Hains focuses on Spinal cord injury, Spinal cord, Neuroscience, Anesthesia and Central nervous system. His study in Spinal cord injury is interdisciplinary in nature, drawing from both Dendritic spine, Neuropathic pain, Somatosensory system and Anatomy. He interconnects Lesion, Chronic pain, Neuron and Pharmacology in the investigation of issues within Spinal cord.
His work on Nervous system and Sensory system as part of general Neuroscience study is frequently linked to Sodium channel, therefore connecting diverse disciplines of science. His work carried out in the field of Nervous system brings together such families of science as Hippocampal formation and Axon initial segment. His Anesthesia research is multidisciplinary, relying on both Axon and Neuroprotection.
His primary scientific interests are in Neuroscience, Spinal cord injury, Neuropathic pain, Central nervous system and Anesthesia. His work blends Neuroscience and Nociceptive Neurons studies together. The concepts of his Spinal cord injury study are interwoven with issues in Dendritic spine and Dorsum.
His Central nervous system research is multidisciplinary, incorporating perspectives in Hippocampal formation, Axon initial segment and Nervous system. His Anesthesia research includes elements of Laminectomy, Spinal cord and Chronic pain. His Spinal cord research includes themes of Neuroprotection and Sensitization.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Activated Microglia Contribute to the Maintenance of Chronic Pain after Spinal Cord Injury
Bryan C. Hains;Stephen G. Waxman.
The Journal of Neuroscience (2006)
Upregulation of Sodium Channel Nav1.3 and Functional Involvement in Neuronal Hyperexcitability Associated with Central Neuropathic Pain after Spinal Cord Injury
Bryan C. Hains;Joshua P. Klein;Carl Y. Saab;Matthew J. Craner.
The Journal of Neuroscience (2003)
Mechanisms of chronic central neuropathic pain after spinal cord injury.
Claire E. Hulsebosch;Bryan C. Hains;Eric D. Crown;Susan M. Carlton.
Brain Research Reviews (2009)
Altered sodium channel expression in second-order spinal sensory neurons contributes to pain after peripheral nerve injury.
Bryan C. Hains;Carl Y. Saab;Joshua P. Klein;Matthew J. Craner.
The Journal of Neuroscience (2004)
Sodium channels contribute to microglia/macrophage activation and function in EAE and MS.
Matthew J. Craner;Tina G. Damarjian;Shujun Liu;Bryan C. Hains.
Glia (2005)
Modulation of Thalamic Nociceptive Processing after Spinal Cord Injury through Remote Activation of Thalamic Microglia by Cysteine–Cysteine Chemokine Ligand 21
Peng Zhao;Stephen G. Waxman;Bryan C. Hains.
The Journal of Neuroscience (2007)
Extracellular signal-regulated kinase-regulated microglia-neuron signaling by prostaglandin E2 contributes to pain after spinal cord injury.
Peng Zhao;Stephen G. Waxman;Bryan C. Hains.
The Journal of Neuroscience (2007)
Co-localization of sodium channel Nav1.6 and the sodium-calcium exchanger at sites of axonal injury in the spinal cord in EAE.
Matthew J. Craner;Bryan C. Hains;Albert C. Lo;Joel A. Black.
Brain (2004)
Primary cortical motor neurons undergo apoptosis after axotomizing spinal cord injury.
Bryan C. Hains;Joel A. Black;Joel A. Black;Stephen G. Waxman.
The Journal of Comparative Neurology (2003)
Changes in electrophysiological properties and sodium channel Nav1.3 expression in thalamic neurons after spinal cord injury
Bryan C. Hains;Carl Y. Saab;Stephen G. Waxman.
Brain (2005)
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