D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 30 Citations 5,601 43 World Ranking 7105 National Ranking 2987

Overview

What is he best known for?

The fields of study he is best known for:

  • Neuron
  • Central nervous system
  • Axon

The scientist’s investigation covers issues in Spinal cord injury, Spinal cord, Neuroscience, Sodium channel and Neuropathic pain. His Spinal cord injury study frequently links to related topics such as Anatomy. His studies deal with areas such as Anesthesia, Axon, Chronic pain and Neuron as well as Spinal cord.

His Pharmacology research extends to the thematically linked field of Neuroscience. His work on Sodium channel blocker as part of general Sodium channel study is frequently linked to Neuroglia, Immunology, Neuroprotection and Experimental autoimmune encephalomyelitis, bridging the gap between disciplines. His research in Neuropathic pain focuses on subjects like Sensory system, which are connected to Sciatic nerve and Nervous system.

His most cited work include:

  • Activated Microglia Contribute to the Maintenance of Chronic Pain after Spinal Cord Injury (482 citations)
  • Upregulation of Sodium Channel Nav1.3 and Functional Involvement in Neuronal Hyperexcitability Associated with Central Neuropathic Pain after Spinal Cord Injury (315 citations)
  • Altered sodium channel expression in second-order spinal sensory neurons contributes to pain after peripheral nerve injury. (223 citations)

What are the main themes of his work throughout his whole career to date?

Bryan C. Hains focuses on Spinal cord injury, Spinal cord, Neuroscience, Anesthesia and Central nervous system. His study in Spinal cord injury is interdisciplinary in nature, drawing from both Dendritic spine, Neuropathic pain, Somatosensory system and Anatomy. He interconnects Lesion, Chronic pain, Neuron and Pharmacology in the investigation of issues within Spinal cord.

His work on Nervous system and Sensory system as part of general Neuroscience study is frequently linked to Sodium channel, therefore connecting diverse disciplines of science. His work carried out in the field of Nervous system brings together such families of science as Hippocampal formation and Axon initial segment. His Anesthesia research is multidisciplinary, relying on both Axon and Neuroprotection.

He most often published in these fields:

  • Spinal cord injury (88.24%)
  • Spinal cord (76.47%)
  • Neuroscience (60.78%)

What were the highlights of his more recent work (between 2008-2011)?

  • Neuroscience (60.78%)
  • Spinal cord injury (88.24%)
  • Neuropathic pain (27.45%)

In recent papers he was focusing on the following fields of study:

His primary scientific interests are in Neuroscience, Spinal cord injury, Neuropathic pain, Central nervous system and Anesthesia. His work blends Neuroscience and Nociceptive Neurons studies together. The concepts of his Spinal cord injury study are interwoven with issues in Dendritic spine and Dorsum.

His Central nervous system research is multidisciplinary, incorporating perspectives in Hippocampal formation, Axon initial segment and Nervous system. His Anesthesia research includes elements of Laminectomy, Spinal cord and Chronic pain. His Spinal cord research includes themes of Neuroprotection and Sensitization.

Between 2008 and 2011, his most popular works were:

  • Mechanisms of chronic central neuropathic pain after spinal cord injury. (216 citations)
  • Role of hippocampal sodium channel Nav1.6 in kindling epileptogenesis (116 citations)
  • Role of hippocampal sodium channel Nav1.6 in kindling epileptogenesis (116 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Activated Microglia Contribute to the Maintenance of Chronic Pain after Spinal Cord Injury

Bryan C. Hains;Stephen G. Waxman.
The Journal of Neuroscience (2006)

778 Citations

Upregulation of Sodium Channel Nav1.3 and Functional Involvement in Neuronal Hyperexcitability Associated with Central Neuropathic Pain after Spinal Cord Injury

Bryan C. Hains;Joshua P. Klein;Carl Y. Saab;Matthew J. Craner.
The Journal of Neuroscience (2003)

431 Citations

Mechanisms of chronic central neuropathic pain after spinal cord injury.

Claire E. Hulsebosch;Bryan C. Hains;Eric D. Crown;Susan M. Carlton.
Brain Research Reviews (2009)

366 Citations

Altered sodium channel expression in second-order spinal sensory neurons contributes to pain after peripheral nerve injury.

Bryan C. Hains;Carl Y. Saab;Joshua P. Klein;Matthew J. Craner.
The Journal of Neuroscience (2004)

326 Citations

Sodium channels contribute to microglia/macrophage activation and function in EAE and MS.

Matthew J. Craner;Tina G. Damarjian;Shujun Liu;Bryan C. Hains.
Glia (2005)

284 Citations

Modulation of Thalamic Nociceptive Processing after Spinal Cord Injury through Remote Activation of Thalamic Microglia by Cysteine–Cysteine Chemokine Ligand 21

Peng Zhao;Stephen G. Waxman;Bryan C. Hains.
The Journal of Neuroscience (2007)

261 Citations

Extracellular signal-regulated kinase-regulated microglia-neuron signaling by prostaglandin E2 contributes to pain after spinal cord injury.

Peng Zhao;Stephen G. Waxman;Bryan C. Hains.
The Journal of Neuroscience (2007)

259 Citations

Co-localization of sodium channel Nav1.6 and the sodium-calcium exchanger at sites of axonal injury in the spinal cord in EAE.

Matthew J. Craner;Bryan C. Hains;Albert C. Lo;Joel A. Black.
Brain (2004)

251 Citations

Primary cortical motor neurons undergo apoptosis after axotomizing spinal cord injury.

Bryan C. Hains;Joel A. Black;Joel A. Black;Stephen G. Waxman.
The Journal of Comparative Neurology (2003)

242 Citations

Changes in electrophysiological properties and sodium channel Nav1.3 expression in thalamic neurons after spinal cord injury

Bryan C. Hains;Carl Y. Saab;Stephen G. Waxman.
Brain (2005)

232 Citations

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