D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 65 Citations 13,330 195 World Ranking 1934 National Ranking 43

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Internal medicine

His scientific interests lie mostly in Immunology, Cytokine, Antigen, Mast cell and Molecular biology. John R. Gordon works mostly in the field of Immunology, limiting it down to topics relating to Angiogenesis and, in certain cases, Tissue remodeling and Necrosis, as a part of the same area of interest. His work deals with themes such as Proinflammatory cytokine, Electrophoretic mobility shift assay and Endocrinology, which intersect with Cytokine.

His research in Antigen intersects with topics in CD23, Germinal center and Cell biology. As part of one scientific family, John R. Gordon deals mainly with the area of Mast cell, narrowing it down to issues related to the Degranulation, and often Infiltration, Antiserum and Intradermal injection. John R. Gordon has included themes like NS2-3 protease, Hepatitis C virus, CD40, Virology and Antibody in his Molecular biology study.

His most cited work include:

  • Mast cells as a source of both preformed and immunologically inducible TNF-α/cachectin (886 citations)
  • Mast cells as a source of multifunctional cytokines (645 citations)
  • Mast cells as a source of multifunctional cytokines (645 citations)

What are the main themes of his work throughout his whole career to date?

John R. Gordon mainly investigates Immunology, Molecular biology, Cytokine, Antigen and Antibody. His is doing research in Immunoglobulin E, Chemokine, Mast cell, Inflammation and IL-2 receptor, both of which are found in Immunology. His Mast cell study combines topics from a wide range of disciplines, such as Interleukin 33 and Allergy.

His work carried out in the field of Molecular biology brings together such families of science as Cell culture, CD40, Receptor, Biochemistry and B cell. He combines subjects such as Tumor necrosis factor alpha, Endocrinology and Fibroblast with his study of Cytokine. In Antigen, John R. Gordon works on issues like CD23, which are connected to Interleukin 4 and Cell surface receptor.

He most often published in these fields:

  • Immunology (50.22%)
  • Molecular biology (23.38%)
  • Cytokine (16.02%)

What were the highlights of his more recent work (between 2014-2021)?

  • Immunology (50.22%)
  • Inflammation (9.96%)
  • Cancer research (8.23%)

In recent papers he was focusing on the following fields of study:

His main research concerns Immunology, Inflammation, Cancer research, Dendritic cell and Interleukin 8. His Immunology study frequently links to other fields, such as Retinoic acid. The various areas that he examines in his Cancer research study include Metastasis, Cell growth and In vivo.

As part of the same scientific family, John R. Gordon usually focuses on Dendritic cell, concentrating on T cell and intersecting with Blockade, Pembrolizumab, Effector, Interleukin 10 and Cytokine. He interconnects Chemokine, Receptor, Molecular biology, CXC chemokine receptors and Fibrosis in the investigation of issues within Interleukin 8. His Immune system study integrates concerns from other disciplines, such as Antigen and Cell biology.

Between 2014 and 2021, his most popular works were:

  • Behavioral alterations in rat offspring following maternal immune activation and ELR-CXC chemokine receptor antagonism during pregnancy: implications for neurodevelopmental psychiatric disorders. (41 citations)
  • CXCR1/2 antagonism with CXCL8/Interleukin-8 analogue CXCL8 (3-72) K11R/G31P restricts lung cancer growth by inhibiting tumor cell proliferation and suppressing angiogenesis (39 citations)
  • Therapeutic reversal of food allergen sensitivity by mature retinoic acid–differentiated dendritic cell induction of LAG3+CD49b−Foxp3− regulatory T cells (28 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Internal medicine

John R. Gordon mainly focuses on Immunology, Cancer research, CXC chemokine receptors, Internal medicine and Endocrinology. His research in Immunoglobulin E, Interleukin 8, Tolerance induction, Food allergy and Allergy are components of Immunology. He works mostly in the field of Immunoglobulin E, limiting it down to concerns involving Ovalbumin and, occasionally, Inflammation.

He works in the field of Cancer research, namely Angiogenesis. His study looks at the relationship between Angiogenesis and topics such as Cisplatin, which overlap with Kidney. His research investigates the link between Internal medicine and topics such as Pregnancy that cross with problems in Tumor necrosis factor alpha, Cytokine and Interleukin 6.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Mast cells as a source of both preformed and immunologically inducible TNF-α/cachectin

John R. Gordon;Stephen J. Galli.
Nature (1990)

1228 Citations

Mast cells as a source of multifunctional cytokines

John R Gordon;John R Gordon;Parris R Burd;Stephen J Galli.
Immunology Today (1990)

908 Citations

Interleukin 3-dependent and -independent mast cells stimulated with IgE and antigen express multiple cytokines.

P. R. Burd;H. W. Rogers;J. R. Gordon;C. A. Martin.
Journal of Experimental Medicine (1989)

671 Citations

Release of both preformed and newly synthesized tumor necrosis factor alpha (TNF-alpha)/cachectin by mouse mast cells stimulated via the Fc epsilon RI. A mechanism for the sustained action of mast cell-derived TNF-alpha during IgE-dependent biological responses.

J R Gordon;S J Galli.
Journal of Experimental Medicine (1991)

501 Citations

Recruitment of neutrophils during IgE-dependent cutaneous late phase reactions in the mouse is mast cell-dependent. Partial inhibition of the reaction with antiserum against tumor necrosis factor-alpha.

Barry K. Wershil;Zhen Sheng Wang;John R. Gordon;Stephen J. Galli.
Journal of Clinical Investigation (1991)

415 Citations

Cytokine production by mast cells and basophils

Stephen J. Galli;John R. Gordon;Barry K. Wershil.
Current Opinion in Immunology (1991)

398 Citations

Recombinant 25‐kDa CD23 and interleukin 1α promote the survival of germinal center B cells: evidence for bifurcation in the development of centrocytes rescued from apoptosis

Yong-Jun Liu;Jennifer A. Cairns;Michelle J. Holder;Sandra D. Abbot.
European Journal of Immunology (1991)

315 Citations

The functional significance behind expressing two IL-8 receptor types on PMN.

RoseMarie Stillie;Shukkur Muhammed Farooq;John R. Gordon;Andrew W. Stadnyk.
Journal of Leukocyte Biology (2009)

289 Citations

Promotion of mouse fibroblast collagen gene expression by mast cells stimulated via the Fc epsilon RI. Role for mast cell-derived transforming growth factor beta and tumor necrosis factor alpha.

J R Gordon;S J Galli.
Journal of Experimental Medicine (1994)

272 Citations

Suppression of apoptosis in normal and neoplastic human B lymphocytes by CD40 ligand is independent of Bcl-2 induction

Michelle J. Holder;Hong Wang;Anne E. Milner;Monserat Casamayor.
European Journal of Immunology (1993)

261 Citations

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