John Easton spends much of his time researching Cancer research, Mutation, Genetics, Cancer and ATRX. John Easton has included themes like Somatic cell, Leukemia, Immunology, Neuroblastoma RAS viral oncogene homolog and PI3K/AKT/mTOR pathway in his Cancer research study. The Mutation study combines topics in areas such as Gene rearrangement, Epigenetics and Transplantation.
His study in Genomics, Gene, Genome, Transcription factor and Exome sequencing is carried out as part of his Genetics studies. His research integrates issues of Retinoblastoma, Exome and Syk in his study of Cancer. His ATRX research integrates issues from Fusion gene and Glioma.
His primary areas of study are Cancer research, Genetics, Cancer, Gene and Mutation. His Cancer research research includes elements of Leukemia, PI3K/AKT/mTOR pathway, Somatic evolution in cancer and ATRX. His Leukemia study combines topics in areas such as Myeloid and Myeloid leukemia.
His work investigates the relationship between Cancer and topics such as Oncology that intersect with problems in Cohort and Germline mutation. His biological study deals with issues like Molecular biology, which deal with fields such as Gene isoform and Alternative splicing. His research is interdisciplinary, bridging the disciplines of Somatic cell and Mutation.
John Easton mainly investigates Cancer research, Cancer, Computational biology, Internal medicine and Cohort. His study in Cancer research is interdisciplinary in nature, drawing from both Mutation, Gene, Leukemia and Immunotherapy. His Mutation research is multidisciplinary, incorporating elements of Gene duplication, Transcriptome and Clone.
John Easton works in the field of Cancer, focusing on Pediatric cancer in particular. John Easton interconnects RNA-Seq, Genome, DNA sequencing and Indel in the investigation of issues within Computational biology. His research investigates the connection with Internal medicine and areas like Oncology which intersect with concerns in Cancer treatment.
John Easton mostly deals with Mutation, Cancer research, Internal medicine, Childhood cancer and Oncology. His research in Mutation intersects with topics in Clone and Genomics. In his works, John Easton undertakes multidisciplinary study on Cancer research and Increased drug tolerance.
His Internal medicine research is multidisciplinary, relying on both Neoplasm, Germline mutation and DNA repair. His studies in Childhood cancer integrate themes in fields like Biomarker, Cohort study, Thyroid cancer and Telomere. John Easton combines subjects such as Single-nucleotide polymorphism and Cohort with his study of Oncology.
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The genetic basis of early T-cell precursor acute lymphoblastic leukaemia
Jinghui Zhang;Li Ding;Linda Holmfeldt;Gang Wu.
Targetable Kinase-Activating Lesions in Ph-like Acute Lymphoblastic Leukemia
K. G. Roberts;Y. Li;D. Payne-Turner;R. C. Harvey.
The New England Journal of Medicine (2014)
Germline Mutations in Predisposition Genes in Pediatric Cancer
Jinghui Zhang;Michael F. Walsh;Gang Wu;Michael N. Edmonson.
The New England Journal of Medicine (2015)
The genomic landscape of diffuse intrinsic pontine glioma and pediatric non-brainstem high-grade glioma
Gang Wu;Alexander K Diaz;Alexander K Diaz;Barbara S Paugh;Sherri L Rankin.
Nature Genetics (2014)
Novel mutations target distinct subgroups of medulloblastoma
Giles Robinson;Matthew Parker;Tanya A. Kranenburg;Charles Lu;Charles Lu.
Whole-genome sequencing identifies genetic alterations in pediatric low-grade gliomas
Jinghui Zhang;Gang Wu;Claudia P. Miller;Ruth G. Tatevossian.
Nature Genetics (2013)
The genomic landscape of hypodiploid acute lymphoblastic leukemia
Linda Holmfeldt;Lei Wei;Ernesto Diaz-Flores;Michael Walsh.
Nature Genetics (2013)
mTOR and cancer therapy.
J B Easton;P J Houghton.
The genomic landscape of pediatric and young adult T-lineage acute lymphoblastic leukemia
Yu Liu;John Easton;Ying Shao;Jamie Maciaszek.
Nature Genetics (2017)
C11orf95 – RELA fusions drive oncogenic NF-κB signalling in ependymoma
Matthew Parker;Kumarasamypet M. Mohankumar;Chandanamali Punchihewa;Ricardo Weinlich.
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