His primary areas of study are Cancer research, Cell biology, Cell cycle, Cancer and Retinoblastoma. Michael A. Dyer combines subjects such as Mutation, Panobinostat, Carcinogenesis and Autopsy with his study of Cancer research. His Cell biology research includes elements of Cell type, Retinal, Retina, Gliosis and Macrophage.
His studies in Cancer integrate themes in fields like Immunology, Genomics, Disease, Neuroblastoma and Rhabdomyosarcoma. Michael A. Dyer interconnects Molecular genetics, Developmental biology, Neuroscience and Solid tumour in the investigation of issues within Immunology. His Retinoblastoma research is multidisciplinary, relying on both Retinoblastoma protein, MDMX, Mdm2, Tumor suppressor gene and Regulation of gene expression.
His primary areas of investigation include Cancer research, Retinoblastoma, Cell biology, Genetics and Retina. His work deals with themes such as Carcinogenesis, Cancer, Immunology, Neuroblastoma and Epigenetics, which intersect with Cancer research. His study in Cancer is interdisciplinary in nature, drawing from both Rhabdomyosarcoma and Genomics.
The various areas that he examines in his Retinoblastoma study include Molecular biology, Clinical trial, MDMX and Programmed cell death. His Cell biology research integrates issues from Cell cycle, Retinal, Cellular differentiation and Induced pluripotent stem cell. His Neuroscience research incorporates themes from Developmental biology and Disease.
Michael A. Dyer mainly investigates Cancer research, Epigenetics, Neuroblastoma, Cell biology and Computational biology. The concepts of his Cancer research study are interwoven with issues in Cancer, Cytotoxic T cell and In vivo. Michael A. Dyer works mostly in the field of Cancer, limiting it down to concerns involving Rhabdomyosarcoma and, occasionally, Internal medicine and Oncology.
His Epigenetics study incorporates themes from Histone and Pediatric cancer. He has researched Neuroblastoma in several fields, including ATRX, Antibody-dependent cell-mediated cytotoxicity, Cytotoxicity, Antibody and Immunotherapy. His Cell biology research also works with subjects such as
His primary scientific interests are in Epigenetics, Rhabdomyosarcoma, DNA methylation, Reprogramming and Induced pluripotent stem cell. His Epigenetics research incorporates elements of Pediatric cancer and Computational biology. His DNA methylation study combines topics in areas such as KLF4, Ectopic expression, Cell type and Cell biology.
His biological study spans a wide range of topics, including Carcinogenesis, Retinoblastoma protein, Cellular differentiation and Histone. Specifically, his work in Genetics is concerned with the study of Mutation. His Cancer study frequently draws connections to other fields, such as Cell.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Somatic histone H3 alterations in pediatric diffuse intrinsic pontine gliomas and non-brainstem glioblastomas
Gang Wu;Alberto Broniscer;Troy A McEachron;Charles Lu.
Nature Genetics (2012)
Neuroblastoma: developmental biology, cancer genomics and immunotherapy
Nai-Kong V. Cheung;Michael A. Dyer;Michael A. Dyer;Michael A. Dyer.
Nature Reviews Cancer (2013)
Inactivation of the p53 pathway in retinoblastoma
Nikia A. Laurie;Stacy L. Donovan;Chie Schin Shih;Jiakun Zhang.
Control of Müller glial cell proliferation and activation following retinal injury.
Michael A. Dyer;Constance L. Cepko.
Nature Neuroscience (2000)
Recurrent Somatic Structural Variations Contribute to Tumorigenesis in Pediatric Osteosarcoma
Xiang Chen;Armita Bahrami;Alberto Pappo;John Easton.
Cell Reports (2014)
A novel retinoblastoma therapy from genomic and epigenetic analyses
Jinghui Zhang;Claudia A. Benavente;Justina McEvoy;Jacqueline Flores-Otero.
Prox1 function controls progenitor cell proliferation and horizontal cell genesis in the mammalian retina
Michael A. Dyer;Frederick J. Livesey;Frederick J. Livesey;Constance L. Cepko;Guillermo Oliver.
Nature Genetics (2003)
Genomic Landscape of Ewing Sarcoma Defines an Aggressive Subtype with Co-Association of STAG2 and TP53 Mutations
Franck Tirode;Didier Surdez;Xiaotu Ma;Matthew Parker.
Cancer Discovery (2014)
Association of Age at Diagnosis and Genetic Mutations in Patients With Neuroblastoma
Nai-Kong V. Cheung;Jinghui Zhang;Charles Lu;Matthew Parker.
p27Kip1 and p57Kip2 Regulate Proliferation in Distinct Retinal Progenitor Cell Populations
Michael A. Dyer;Constance L. Cepko.
The Journal of Neuroscience (2001)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: