D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 73 Citations 22,485 211 World Ranking 2479 National Ranking 67

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • DNA

His primary scientific interests are in Neuroscience, Long-term potentiation, Endocrinology, Internal medicine and NMDA receptor. The various areas that John C. Roder examines in his Neuroscience study include Neurotransmission, Metabotropic glutamate receptor and Cell biology. His Long-term potentiation research is multidisciplinary, relying on both Synaptic plasticity, AMPA receptor, Hippocampal formation and Excitatory postsynaptic potential.

His Endocrinology research focuses on subjects like Prepulse inhibition, which are linked to Mutation and Genetics. Many of his research projects under Internal medicine are closely connected to Cachexia with Cachexia, tying the diverse disciplines of science together. His NMDA receptor study which covers Psychosis that intersects with Clozapine and Schizophrenia.

His most cited work include:

  • Derivation of completely cell culture-derived mice from early-passage embryonic stem cells (2176 citations)
  • The Collaborative Cross, a community resource for the genetic analysis of complex traits (852 citations)
  • A YAC mouse model for Huntington's disease with full-length mutant huntingtin, cytoplasmic toxicity, and selective striatal neurodegeneration. (749 citations)

What are the main themes of his work throughout his whole career to date?

The scientist’s investigation covers issues in Neuroscience, Cell biology, Molecular biology, Internal medicine and Endocrinology. His Neuroscience research is multidisciplinary, incorporating perspectives in NMDA receptor, Long-term potentiation and Synaptic plasticity. His work carried out in the field of Cell biology brings together such families of science as Neurite, Biochemistry, Myelin, Myelin-associated glycoprotein and Neuronal calcium sensor-1.

He has included themes like Cell culture, Natural killer cell, Complementary DNA and Antibody, Monoclonal antibody in his Molecular biology study. His Internal medicine study frequently draws connections between adjacent fields such as Immunology. In his study, DISC1, Mutation and Glycogen synthase is strongly linked to Mutant, which falls under the umbrella field of Endocrinology.

He most often published in these fields:

  • Neuroscience (42.19%)
  • Cell biology (26.03%)
  • Molecular biology (27.40%)

What were the highlights of his more recent work (between 2009-2020)?

  • Neuroscience (42.19%)
  • DISC1 (16.99%)
  • Endocrinology (26.58%)

In recent papers he was focusing on the following fields of study:

John C. Roder mainly focuses on Neuroscience, DISC1, Endocrinology, Internal medicine and Schizophrenia. His research in Neuroscience intersects with topics in NMDA receptor and Long-term potentiation. His DISC1 study integrates concerns from other disciplines, such as Genetic model, Mutant, Schizophrenia, Interneuron and Prepulse inhibition.

His Mutant research incorporates themes from Mutation, Glycogen synthase and Immunology. His work in Mutation tackles topics such as Cell biology which are related to areas like Postsynaptic Current. His Endocrinology research incorporates elements of ATP1A3, Na+/K+-ATPase and Valproic Acid.

Between 2009 and 2020, his most popular works were:

  • Assessment of Social Interaction Behaviors (224 citations)
  • A mouse model of Down syndrome trisomic for all human chromosome 21 syntenic regions (156 citations)
  • α5GABAA Receptor Activity Sets the Threshold for Long-Term Potentiation and Constrains Hippocampus-Dependent Memory (134 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • DNA

His primary areas of study are Internal medicine, Endocrinology, DISC1, Neuroscience and Schizophrenia. His research integrates issues of Glycine, ATP1A3 and Serine in his study of Internal medicine. His Endocrinology study combines topics from a wide range of disciplines, such as Protein kinase B, Signal transduction, Calcium signaling, Valproic Acid and Na+/K+-ATPase.

His studies in DISC1 integrate themes in fields like Prepulse inhibition, Antibody, Immunology and Mutant. The concepts of his Neuroscience study are interwoven with issues in Nonsynaptic plasticity, Long-term potentiation and Synaptic scaling. His biological study spans a wide range of topics, including Glutamate receptor and Psychosis.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Derivation of completely cell culture-derived mice from early-passage embryonic stem cells

Andras Nagy;Janet Rossant;Reka Nagy;Wanda Abramow-Newerly.
Proceedings of the National Academy of Sciences of the United States of America (1993)

2754 Citations

The Collaborative Cross, a community resource for the genetic analysis of complex traits

Gary A. Churchill;David C. Airey;Hooman Allayee;Joe M. Angel.
Nature Genetics (2004)

974 Citations

A YAC mouse model for Huntington's disease with full-length mutant huntingtin, cytoplasmic toxicity, and selective striatal neurodegeneration.

J.Graeme Hodgson;Nadia Agopyan;Claire-Anne Gutekunst;Blair R Leavitt.
Neuron (1999)

930 Citations

Mice Lacking Metabotropic Glutamate Receptor 5 Show Impaired Learning and Reduced CA1 Long-Term Potentiation (LTP) But Normal CA3 LTP

Y M Lu;Z Jia;C Janus;J T Henderson.
The Journal of Neuroscience (1997)

659 Citations

Myelin-Associated Glycoprotein Is a Myelin Signal that Modulates the Caliber of Myelinated Axons

Xinghua Yin;Thomas O. Crawford;John W. Griffin;Pang Hsien Tu.
The Journal of Neuroscience (1998)

638 Citations

Nuk Controls Pathfinding of Commissural Axons in the Mammalian Central Nervous System

Mark Henkemeyer;Donata Orioli;Jeffrey T Henderson;Tracy M Saxton;Tracy M Saxton.
Cell (1996)

637 Citations

Behavioral phenotypes of Disc1 missense mutations in mice.

Steven J. Clapcote;Tatiana V. Lipina;J. Kirsty Millar;Shaun Mackie.
Neuron (2007)

571 Citations

Enhanced LTP in Mice Deficient in the AMPA Receptor GluR2

Zhengping Jia;Nadia Agopyan;Peter Miu;Zhigang Xiong.
Neuron (1996)

540 Citations

Chemical Induction of mGluR5- and Protein Synthesis–Dependent Long-Term Depression in Hippocampal Area CA1

Kimberly M. Huber;John C. Roder;Mark F. Bear.
Journal of Neurophysiology (2001)

520 Citations

Mutant Mice and Neuroscience: Recommendations Concerning Genetic Background

Alcino J. Silva;Elizabeth M. Simpson;Joseph S. Takahashi;Hans Peter Lipp.
Neuron (1997)

427 Citations

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