His primary scientific interests are in Neuroscience, Long-term potentiation, Endocrinology, Internal medicine and NMDA receptor. The various areas that John C. Roder examines in his Neuroscience study include Neurotransmission, Metabotropic glutamate receptor and Cell biology. His Long-term potentiation research is multidisciplinary, relying on both Synaptic plasticity, AMPA receptor, Hippocampal formation and Excitatory postsynaptic potential.
His Endocrinology research focuses on subjects like Prepulse inhibition, which are linked to Mutation and Genetics. Many of his research projects under Internal medicine are closely connected to Cachexia with Cachexia, tying the diverse disciplines of science together. His NMDA receptor study which covers Psychosis that intersects with Clozapine and Schizophrenia.
The scientist’s investigation covers issues in Neuroscience, Cell biology, Molecular biology, Internal medicine and Endocrinology. His Neuroscience research is multidisciplinary, incorporating perspectives in NMDA receptor, Long-term potentiation and Synaptic plasticity. His work carried out in the field of Cell biology brings together such families of science as Neurite, Biochemistry, Myelin, Myelin-associated glycoprotein and Neuronal calcium sensor-1.
He has included themes like Cell culture, Natural killer cell, Complementary DNA and Antibody, Monoclonal antibody in his Molecular biology study. His Internal medicine study frequently draws connections between adjacent fields such as Immunology. In his study, DISC1, Mutation and Glycogen synthase is strongly linked to Mutant, which falls under the umbrella field of Endocrinology.
John C. Roder mainly focuses on Neuroscience, DISC1, Endocrinology, Internal medicine and Schizophrenia. His research in Neuroscience intersects with topics in NMDA receptor and Long-term potentiation. His DISC1 study integrates concerns from other disciplines, such as Genetic model, Mutant, Schizophrenia, Interneuron and Prepulse inhibition.
His Mutant research incorporates themes from Mutation, Glycogen synthase and Immunology. His work in Mutation tackles topics such as Cell biology which are related to areas like Postsynaptic Current. His Endocrinology research incorporates elements of ATP1A3, Na+/K+-ATPase and Valproic Acid.
His primary areas of study are Internal medicine, Endocrinology, DISC1, Neuroscience and Schizophrenia. His research integrates issues of Glycine, ATP1A3 and Serine in his study of Internal medicine. His Endocrinology study combines topics from a wide range of disciplines, such as Protein kinase B, Signal transduction, Calcium signaling, Valproic Acid and Na+/K+-ATPase.
His studies in DISC1 integrate themes in fields like Prepulse inhibition, Antibody, Immunology and Mutant. The concepts of his Neuroscience study are interwoven with issues in Nonsynaptic plasticity, Long-term potentiation and Synaptic scaling. His biological study spans a wide range of topics, including Glutamate receptor and Psychosis.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Derivation of completely cell culture-derived mice from early-passage embryonic stem cells
Andras Nagy;Janet Rossant;Reka Nagy;Wanda Abramow-Newerly.
Proceedings of the National Academy of Sciences of the United States of America (1993)
The Collaborative Cross, a community resource for the genetic analysis of complex traits
Gary A. Churchill;David C. Airey;Hooman Allayee;Joe M. Angel.
Nature Genetics (2004)
A YAC mouse model for Huntington's disease with full-length mutant huntingtin, cytoplasmic toxicity, and selective striatal neurodegeneration.
J.Graeme Hodgson;Nadia Agopyan;Claire-Anne Gutekunst;Blair R Leavitt.
Neuron (1999)
Mice Lacking Metabotropic Glutamate Receptor 5 Show Impaired Learning and Reduced CA1 Long-Term Potentiation (LTP) But Normal CA3 LTP
Y M Lu;Z Jia;C Janus;J T Henderson.
The Journal of Neuroscience (1997)
Myelin-Associated Glycoprotein Is a Myelin Signal that Modulates the Caliber of Myelinated Axons
Xinghua Yin;Thomas O. Crawford;John W. Griffin;Pang Hsien Tu.
The Journal of Neuroscience (1998)
Nuk Controls Pathfinding of Commissural Axons in the Mammalian Central Nervous System
Mark Henkemeyer;Donata Orioli;Jeffrey T Henderson;Tracy M Saxton;Tracy M Saxton.
Cell (1996)
Behavioral phenotypes of Disc1 missense mutations in mice.
Steven J. Clapcote;Tatiana V. Lipina;J. Kirsty Millar;Shaun Mackie.
Neuron (2007)
Enhanced LTP in Mice Deficient in the AMPA Receptor GluR2
Zhengping Jia;Nadia Agopyan;Peter Miu;Zhigang Xiong.
Neuron (1996)
Chemical Induction of mGluR5- and Protein Synthesis–Dependent Long-Term Depression in Hippocampal Area CA1
Kimberly M. Huber;John C. Roder;Mark F. Bear.
Journal of Neurophysiology (2001)
Mutant Mice and Neuroscience: Recommendations Concerning Genetic Background
Alcino J. Silva;Elizabeth M. Simpson;Joseph S. Takahashi;Hans Peter Lipp.
Neuron (1997)
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