What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Cancer
His primary areas of investigation include Pathology, Skeletal muscle, Anatomy, Alzheimer's disease and Cell biology.
His Pathology research is multidisciplinary, relying on both Cerebral cortex and Fibroblast.
Hannu Kalimo interconnects Athletes, Tendon and Strain in the investigation of issues within Skeletal muscle.
His study in Anatomy is interdisciplinary in nature, drawing from both Wound healing, Muscular dystrophy and Connective tissue.
He combines subjects such as Mutation, Genetically modified mouse and Amyloid with his study of Alzheimer's disease.
His work on Myocyte and Signal transduction as part of general Cell biology research is frequently linked to Nestin, bridging the gap between disciplines.
His most cited work include:
- A hexanucleotide repeat expansion in C9ORF72 is the cause of chromosome 9p21-linked ALS-FTD (2859 citations)
- Muscle Injuries: Biology and Treatment (751 citations)
- The lumbar multifidus muscle five years after surgery for a lumbar intervertebral disc herniation (311 citations)
What are the main themes of his work throughout his whole career to date?
Hannu Kalimo mainly investigates Pathology, Internal medicine, Endocrinology, CADASIL and Skeletal muscle.
His Pathology study frequently draws parallels with other fields, such as White matter.
His research in CADASIL intersects with topics in Genetics, Vascular smooth muscle and Cognitive decline.
The various areas that Hannu Kalimo examines in his Skeletal muscle study include Myocyte, Cell biology and Atrophy.
His work in Cell biology addresses subjects such as Myopathy, which are connected to disciplines such as Chaperone.
As part of the same scientific family, Hannu Kalimo usually focuses on Amyloid, concentrating on Alzheimer's disease and intersecting with Genetically modified mouse.
He most often published in these fields:
- Pathology (59.54%)
- Internal medicine (18.21%)
- Endocrinology (15.61%)
What were the highlights of his more recent work (between 2007-2020)?
- Pathology (59.54%)
- Cell biology (14.16%)
- CADASIL (21.10%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Pathology, Cell biology, CADASIL, Myopathy and Autophagy.
Hannu Kalimo focuses mostly in the field of Pathology, narrowing it down to matters related to White matter and, in some cases, Central nervous system disease.
His Cell biology research is multidisciplinary, incorporating elements of Endocrinology, Downregulation and upregulation, Internal medicine and In vitro.
His work carried out in the field of CADASIL brings together such families of science as Vascular smooth muscle, HTRA1, Vascular dementia and Cognitive decline.
His studies in Myopathy integrate themes in fields like Muscle biopsy and Skeletal muscle.
His study explores the link between Autophagy and topics such as Chaperone that cross with problems in Cytoplasm.
Between 2007 and 2020, his most popular works were:
- A hexanucleotide repeat expansion in C9ORF72 is the cause of chromosome 9p21-linked ALS-FTD (2859 citations)
- Notch Signaling Regulates Platelet-Derived Growth Factor Receptor-β Expression in Vascular Smooth Muscle Cells (146 citations)
- Congruence between NOTCH3 mutations and GOM in 131 CADASIL patients. (130 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Cancer
His main research concerns Pathology, Myopathy, Cell biology, CADASIL and Amyloid.
His biological study spans a wide range of topics, including Central nervous system and Spinal cord.
His work is dedicated to discovering how Myopathy, Autophagy are connected with PI3K/AKT/mTOR pathway, Atrophy and Skeletal muscle and other disciplines.
His Cell biology research includes themes of Internal medicine and Downregulation and upregulation.
His research integrates issues of Frontal lobe, HTRA1 and Vascular dementia in his study of CADASIL.
His Amyloid research integrates issues from Caudate nucleus, Putamen, Molecular biology, Alzheimer's disease and Genetically modified mouse.
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