Graeme J. Stewart focuses on Genetics, Multiple sclerosis, Immunology, Genome-wide association study and Single-nucleotide polymorphism. His is doing research in Allele, CLEC16A and Locus, both of which are found in Genetics. His work in CLEC16A addresses issues such as Genetic predisposition, which are connected to fields such as Lymphocyte differentiation, Genetic architecture and Cellular immunity.
As part of one scientific family, Graeme J. Stewart deals mainly with the area of Multiple sclerosis, narrowing it down to issues related to the Receptor, and often Interleukin-7 receptor-α, Allele frequency, Null allele and Test cross. His biological study deals with issues like Virology, which deal with fields such as Loss of heterozygosity. The concepts of his Genome-wide association study study are interwoven with issues in SNP, Polymorphism, Genotyping, Human leukocyte antigen and Genetic association.
Graeme J. Stewart mostly deals with Immunology, Multiple sclerosis, Genetics, Gene and Haplotype. His biological study spans a wide range of topics, including Acquired immunodeficiency syndrome, Genotype and Virology. His Multiple sclerosis study integrates concerns from other disciplines, such as Gene expression, Pathogenesis, Disease, Antibody and Computational biology.
His is involved in several facets of Genetics study, as is seen by his studies on Single-nucleotide polymorphism, Linkage disequilibrium, Genome-wide association study, Locus and Allele. His Genome-wide association study study frequently draws connections to other fields, such as Genetic association. His research integrates issues of Candidate gene, Molecular biology and Exon in his study of Haplotype.
His scientific interests lie mostly in Multiple sclerosis, Immunology, Gene, Genetics and Genome-wide association study. His Multiple sclerosis study combines topics in areas such as Gene expression, Pathogenesis, Disease, Computational biology and Major histocompatibility complex. His Immunology research includes elements of Phenotype and Genotype.
The study incorporates disciplines such as SNP, Virus, Human leukocyte antigen, Allele and Human genetics in addition to Genome-wide association study. He combines subjects such as Genetic predisposition, Epistasis, Cellular immunity, Lymphocyte differentiation and Genetic architecture with his study of Genetic association. His Hepatitis C virus research is multidisciplinary, relying on both Interferon and Hepatitis C.
The scientist’s investigation covers issues in Immunology, Genome-wide association study, Genetics, Multiple sclerosis and Genetic association. His Immunology study frequently draws connections between related disciplines such as Cell activation. His Genome-wide association study research incorporates themes from Genetic predisposition, Cellular immunity, CLEC16A, Human leukocyte antigen and Allele.
His works in Linkage disequilibrium, Single-nucleotide polymorphism, Lymphocyte differentiation and Genetic architecture are all subjects of inquiry into Genetics. His study in Multiple sclerosis is interdisciplinary in nature, drawing from both Nerve fiber layer, Polymorphism, Visual acuity and Major histocompatibility complex. Graeme J. Stewart has researched Genetic association in several fields, including Epistasis, Quantitative trait locus, Expression quantitative trait loci, Imputation and Intron.
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Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis
Stephen Sawcer;Garrett Hellenthal;Matti Pirinen;Chris C. A. Spencer.
IL28B is associated with response to chronic hepatitis C interferon-alpha and ribavirin therapy
Vijayaprakash Suppiah;Max Moldovan;Golo Ahlenstiel;Thomas Berg.
Nature Genetics (2009)
Analysis of immune-related loci identifies 48 new susceptibility variants for multiple sclerosis
Ashley H. Beecham;Nikolaos A. Patsopoulos;Nikolaos A. Patsopoulos;Dionysia K. Xifara;Mary F. Davis.
Nature Genetics (2013)
Genome-wide association study identifies new multiple sclerosis susceptibility loci on chromosomes 12 and 20
Melanie Bahlo;David R Booth;Simon A Broadley;Matthew A Brown;Matthew A Brown.
Nature Genetics (2009)
HIV-1 infection in an individual homozygous for the CCR5 deletion allele.
Robyn Biti;Rosemary Ffrench;Judy Young;Bruce Bennetts.
Nature Medicine (1997)
A high-density screen for linkage in multiple sclerosis.
Sawcer S;Ban M;Maranian M;Yeo Tw.
American Journal of Human Genetics (2005)
Genome-wide meta-analysis identifies novel multiple sclerosis susceptibility loci
Nikolaos A. Patsopoulos;Federica Esposito;Joachim Reischl;Stephan Lehr.
Annals of Neurology (2011)
Class II HLA interactions modulate genetic risk for multiple sclerosis
L Moutsianas;L Jostins;A H Beecham;A T Dilthey.
Nature Genetics (2015)
MicroRNAs miR-17 and miR-20a Inhibit T Cell Activation Genes and Are Under-Expressed in MS Whole Blood
Mathew B. Cox;Murray J. Cairns;Kaushal S. Gandhi;Adam P. Carroll.
PLOS ONE (2010)
The role of the CD58 locus in multiple sclerosis
Philip L. De Jager;Clare Baecher-Allan;Lisa M. Maier;Ariel T. Arthur.
Proceedings of the National Academy of Sciences of the United States of America (2009)
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