D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 41 Citations 10,155 116 World Ranking 3500 National Ranking 166

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Immune system

Graeme J. Stewart focuses on Genetics, Multiple sclerosis, Immunology, Genome-wide association study and Single-nucleotide polymorphism. His is doing research in Allele, CLEC16A and Locus, both of which are found in Genetics. His work in CLEC16A addresses issues such as Genetic predisposition, which are connected to fields such as Lymphocyte differentiation, Genetic architecture and Cellular immunity.

As part of one scientific family, Graeme J. Stewart deals mainly with the area of Multiple sclerosis, narrowing it down to issues related to the Receptor, and often Interleukin-7 receptor-α, Allele frequency, Null allele and Test cross. His biological study deals with issues like Virology, which deal with fields such as Loss of heterozygosity. The concepts of his Genome-wide association study study are interwoven with issues in SNP, Polymorphism, Genotyping, Human leukocyte antigen and Genetic association.

His most cited work include:

  • Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis (1970 citations)
  • IL28B is associated with response to chronic hepatitis C interferon-alpha and ribavirin therapy. (1667 citations)
  • Analysis of immune-related loci identifies 48 new susceptibility variants for multiple sclerosis (906 citations)

What are the main themes of his work throughout his whole career to date?

Graeme J. Stewart mostly deals with Immunology, Multiple sclerosis, Genetics, Gene and Haplotype. His biological study spans a wide range of topics, including Acquired immunodeficiency syndrome, Genotype and Virology. His Multiple sclerosis study integrates concerns from other disciplines, such as Gene expression, Pathogenesis, Disease, Antibody and Computational biology.

His is involved in several facets of Genetics study, as is seen by his studies on Single-nucleotide polymorphism, Linkage disequilibrium, Genome-wide association study, Locus and Allele. His Genome-wide association study study frequently draws connections to other fields, such as Genetic association. His research integrates issues of Candidate gene, Molecular biology and Exon in his study of Haplotype.

He most often published in these fields:

  • Immunology (46.15%)
  • Multiple sclerosis (34.84%)
  • Genetics (27.15%)

What were the highlights of his more recent work (between 2010-2020)?

  • Multiple sclerosis (34.84%)
  • Immunology (46.15%)
  • Gene (16.29%)

In recent papers he was focusing on the following fields of study:

His scientific interests lie mostly in Multiple sclerosis, Immunology, Gene, Genetics and Genome-wide association study. His Multiple sclerosis study combines topics in areas such as Gene expression, Pathogenesis, Disease, Computational biology and Major histocompatibility complex. His Immunology research includes elements of Phenotype and Genotype.

The study incorporates disciplines such as SNP, Virus, Human leukocyte antigen, Allele and Human genetics in addition to Genome-wide association study. He combines subjects such as Genetic predisposition, Epistasis, Cellular immunity, Lymphocyte differentiation and Genetic architecture with his study of Genetic association. His Hepatitis C virus research is multidisciplinary, relying on both Interferon and Hepatitis C.

Between 2010 and 2020, his most popular works were:

  • Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis (1970 citations)
  • Analysis of immune-related loci identifies 48 new susceptibility variants for multiple sclerosis (906 citations)
  • Genome-wide meta-analysis identifies novel multiple sclerosis susceptibility loci (268 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Immune system

The scientist’s investigation covers issues in Immunology, Genome-wide association study, Genetics, Multiple sclerosis and Genetic association. His Immunology study frequently draws connections between related disciplines such as Cell activation. His Genome-wide association study research incorporates themes from Genetic predisposition, Cellular immunity, CLEC16A, Human leukocyte antigen and Allele.

His works in Linkage disequilibrium, Single-nucleotide polymorphism, Lymphocyte differentiation and Genetic architecture are all subjects of inquiry into Genetics. His study in Multiple sclerosis is interdisciplinary in nature, drawing from both Nerve fiber layer, Polymorphism, Visual acuity and Major histocompatibility complex. Graeme J. Stewart has researched Genetic association in several fields, including Epistasis, Quantitative trait locus, Expression quantitative trait loci, Imputation and Intron.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

IL28B is associated with response to chronic hepatitis C interferon-alpha and ribavirin therapy.

Vijayaprakash Suppiah;Max Moldovan;Golo Ahlenstiel;Thomas Berg.
Nature Genetics (2009)

2281 Citations

Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis

Stephen Sawcer;Garrett Hellenthal;Matti Pirinen;Chris C. A. Spencer.
Nature (2011)

2262 Citations

Analysis of immune-related loci identifies 48 new susceptibility variants for multiple sclerosis

Ashley H. Beecham;Nikolaos A. Patsopoulos;Nikolaos A. Patsopoulos;Dionysia K. Xifara;Mary F. Davis.
Nature Genetics (2013)

873 Citations

Genome-wide association study identifies new multiple sclerosis susceptibility loci on chromosomes 12 and 20

Melanie Bahlo;David R Booth;Simon A Broadley;Matthew A Brown;Matthew A Brown.
Nature Genetics (2009)

456 Citations

HIV-1 infection in an individual homozygous for the CCR5 deletion allele.

Robyn Biti;Rosemary Ffrench;Judy Young;Bruce Bennetts.
Nature Medicine (1997)

419 Citations

A high-density screen for linkage in multiple sclerosis.

Stephen Sawcer;Maria Ban;Mel Maranian;Tai Wai Yeo.
American Journal of Human Genetics (2005)

390 Citations

Genome-wide meta-analysis identifies novel multiple sclerosis susceptibility loci

Nikolaos A. Patsopoulos;Federica Esposito;Joachim Reischl;Stephan Lehr.
Annals of Neurology (2011)

313 Citations

MicroRNAs miR-17 and miR-20a Inhibit T Cell Activation Genes and Are Under-Expressed in MS Whole Blood

Mathew B. Cox;Murray J. Cairns;Kaushal S. Gandhi;Adam P. Carroll.
PLOS ONE (2010)

257 Citations

Class II HLA interactions modulate genetic risk for multiple sclerosis

L Moutsianas;L Jostins;A H Beecham;A T Dilthey.
Nature Genetics (2015)

239 Citations

The role of the CD58 locus in multiple sclerosis

Philip L. De Jager;Clare Baecher-Allan;Lisa M. Maier;Ariel T. Arthur.
Proceedings of the National Academy of Sciences of the United States of America (2009)

210 Citations

Best Scientists Citing Graeme J. Stewart

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David A. Hafler

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Yale University

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Hanne F. Harbo

Hanne F. Harbo

Oslo University Hospital

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Jan Hillert

Jan Hillert

Karolinska Institute

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Philip L. De Jager

Philip L. De Jager

Columbia University

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Stephen L. Hauser

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University of California, San Francisco

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Stephen Sawcer

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University of Cambridge

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Lars Alfredsson

Lars Alfredsson

Karolinska Institute

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Ingrid Kockum

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Lisa F. Barcellos

Lisa F. Barcellos

University of California, Berkeley

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University of Oxford

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Autonomous University of Barcelona

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Profile was last updated on December 6th, 2021.
Research.com Ranking is based on data retrieved from the Microsoft Academic Graph (MAG).
The ranking d-index is inferred from publications deemed to belong to the considered discipline.

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