D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 65 Citations 14,713 138 World Ranking 1814 National Ranking 879

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Alzheimer's disease
  • Neuron

His primary areas of investigation include Genetically modified mouse, Neuroscience, Alzheimer's disease, Internal medicine and Endocrinology. His Presenilin study in the realm of Alzheimer's disease interacts with subjects such as Transgenic Model. His work in the fields of Internal medicine, such as Hippocampus, Lipid peroxidation, Male rats and Adult male, intersects with other areas such as Sexual behavior.

His Hippocampus research is multidisciplinary, relying on both Oxidative stress, Cortex and Anatomy. Endocrinology is frequently linked to Dorsum in his study. His work deals with themes such as Open field and Effects of sleep deprivation on cognitive performance, which intersect with Water maze.

His most cited work include:

  • Aβ peptide vaccination prevents memory loss in an animal model of Alzheimer's disease (1486 citations)
  • Caffeine protects Alzheimer's mice against cognitive impairment and reduces brain β-amyloid production (335 citations)
  • Blueberry Supplementation Enhances Signaling and Prevents Behavioral Deficits in an Alzheimer Disease Model (314 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Internal medicine, Endocrinology, Neuroscience, Alzheimer's disease and Genetically modified mouse. As part of his studies on Internal medicine, he frequently links adjacent subjects like Mitochondrion. His study on Endocrinology is mostly dedicated to connecting different topics, such as Neurodegeneration.

His Neuroscience study frequently intersects with other fields, such as Atrophy. His Alzheimer's disease research includes elements of Water maze, Cognitive disorder and Degenerative disease. Gary W. Arendash combines subjects such as Memoria and Morris water navigation task with his study of Water maze.

He most often published in these fields:

  • Internal medicine (46.15%)
  • Endocrinology (43.36%)
  • Neuroscience (35.66%)

What were the highlights of his more recent work (between 2010-2020)?

  • Genetically modified mouse (24.48%)
  • Endocrinology (43.36%)
  • Internal medicine (46.15%)

In recent papers he was focusing on the following fields of study:

Genetically modified mouse, Endocrinology, Internal medicine, Neuroscience and Alzheimer's disease are his primary areas of study. His work carried out in the field of Endocrinology brings together such families of science as Senile plaques, Mitochondrion and Protein kinase A. He interconnects Cingulum and Neuroimaging in the investigation of issues within Internal medicine.

His Neuroscience study incorporates themes from Disease and Neurodegeneration. Alzheimer's disease is closely attributed to Hippocampus in his research. His Hippocampus research is multidisciplinary, incorporating perspectives in Synaptic plasticity, GSK-3, Cotinine, Nicotine and Presenilin.

Between 2010 and 2020, his most popular works were:

  • Melatonin treatment restores mitochondrial function in Alzheimer’s mice: a mitochondrial protective role of melatonin membrane receptor signaling (116 citations)
  • Tannic Acid is a Natural β-secretase Inhibitor that Prevents Cognitive Impairment and Mitigates Alzheimer-like Pathology in Transgenic Mice (109 citations)
  • High Blood Caffeine Levels in MCI Linked to Lack of Progression to Dementia (82 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Neuron
  • Alzheimer's disease

His primary areas of study are Genetically modified mouse, Alzheimer's disease, Mitochondrion, Internal medicine and Endocrinology. Throughout his Genetically modified mouse studies, he incorporates elements of other sciences such as Amyloid precursor protein, Bone marrow, Analysis of variance, Biochemistry and Effects of sleep deprivation on cognitive performance. His studies deal with areas such as Neuroinflammation and Cerebral amyloid angiopathy, Amyloid as well as Amyloid precursor protein.

The Alzheimer's disease study combines topics in areas such as Cerebral cortex and Cerebral blood flow. His Cerebral cortex research is under the purview of Neuroscience. In Mitochondrion, Gary W. Arendash works on issues like Hippocampus, which are connected to Melatonin, Melatonin receptor, Receptor and Presenilin.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

A beta peptide vaccination prevents memory loss in an animal model of Alzheimer's disease.

Dave Morgan;David M. Diamond;Paul E. Gottschall;Kenneth E. Ugen.
Nature (2000)

2073 Citations

Caffeine protects Alzheimer's mice against cognitive impairment and reduces brain β-amyloid production

G.W. Arendash;W. Schleif;K. Rezai-Zadeh;E.K. Jackson.
Neuroscience (2006)

542 Citations

Blueberry Supplementation Enhances Signaling and Prevents Behavioral Deficits in an Alzheimer Disease Model

J. A. Joseph;G. Arendash;M Gordon;David M. Diamond.
Nutritional Neuroscience (2003)

488 Citations

Green tea epigallocatechin-3-gallate (EGCG) reduces beta-amyloid mediated cognitive impairment and modulates tau pathology in Alzheimer transgenic mice.

Kavon Rezai-Zadeh;Gary W. Arendash;Huayan Hou;Frank Fernandez.
Brain Research (2008)

461 Citations

Progressive, age-related behavioral impairments in transgenic mice carrying both mutant amyloid precursor protein and presenilin-1 transgenes.

Gary W. Arendash;David L. King;Marcia N. Gordon;Dave Morgan.
Brain Research (2001)

408 Citations

Effects of discrete lesions of the sexually dimorphic nucleus of the preoptic area or other medial preoptic regions on the sexual behavior of male rats.

Gary W. Arendash;Roger A. Gorski.
Brain Research Bulletin (1983)

366 Citations

Evidence That Oxidative Stress Is Associated with the Pathophysiology of Inherited Hydrocephalus in the H-Tx Rat Model

D.J. Socci;K.B. Bjugstad;H.C. Jones;J.V. Pattisapu.
Experimental Neurology (1999)

336 Citations

Environmental enrichment improves cognition in aged Alzheimer's transgenic mice despite stable beta-amyloid deposition.

Gary W Arendash;Marcos F Garcia;David A Costa;Jennifer R Cracchiolo.
Neuroreport (2004)

317 Citations

Caffeine reverses cognitive impairment and decreases brain amyloid-β levels in aged Alzheimer's disease mice.

Gary W. Arendash;Takashi Mori;Chuanhai Cao;Malgorzata Mamcarz.
Journal of Alzheimer's Disease (2009)

317 Citations

Behavioral characterization of the Tg2576 transgenic model of Alzheimer's disease through 19 months.

David L King;Gary W Arendash.
Physiology & Behavior (2002)

315 Citations

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