His primary areas of investigation include Genetically modified mouse, Neuroscience, Alzheimer's disease, Internal medicine and Endocrinology. His Presenilin study in the realm of Alzheimer's disease interacts with subjects such as Transgenic Model. His work in the fields of Internal medicine, such as Hippocampus, Lipid peroxidation, Male rats and Adult male, intersects with other areas such as Sexual behavior.
His Hippocampus research is multidisciplinary, relying on both Oxidative stress, Cortex and Anatomy. Endocrinology is frequently linked to Dorsum in his study. His work deals with themes such as Open field and Effects of sleep deprivation on cognitive performance, which intersect with Water maze.
His primary scientific interests are in Internal medicine, Endocrinology, Neuroscience, Alzheimer's disease and Genetically modified mouse. As part of his studies on Internal medicine, he frequently links adjacent subjects like Mitochondrion. His study on Endocrinology is mostly dedicated to connecting different topics, such as Neurodegeneration.
His Neuroscience study frequently intersects with other fields, such as Atrophy. His Alzheimer's disease research includes elements of Water maze, Cognitive disorder and Degenerative disease. Gary W. Arendash combines subjects such as Memoria and Morris water navigation task with his study of Water maze.
Genetically modified mouse, Endocrinology, Internal medicine, Neuroscience and Alzheimer's disease are his primary areas of study. His work carried out in the field of Endocrinology brings together such families of science as Senile plaques, Mitochondrion and Protein kinase A. He interconnects Cingulum and Neuroimaging in the investigation of issues within Internal medicine.
His Neuroscience study incorporates themes from Disease and Neurodegeneration. Alzheimer's disease is closely attributed to Hippocampus in his research. His Hippocampus research is multidisciplinary, incorporating perspectives in Synaptic plasticity, GSK-3, Cotinine, Nicotine and Presenilin.
His primary areas of study are Genetically modified mouse, Alzheimer's disease, Mitochondrion, Internal medicine and Endocrinology. Throughout his Genetically modified mouse studies, he incorporates elements of other sciences such as Amyloid precursor protein, Bone marrow, Analysis of variance, Biochemistry and Effects of sleep deprivation on cognitive performance. His studies deal with areas such as Neuroinflammation and Cerebral amyloid angiopathy, Amyloid as well as Amyloid precursor protein.
The Alzheimer's disease study combines topics in areas such as Cerebral cortex and Cerebral blood flow. His Cerebral cortex research is under the purview of Neuroscience. In Mitochondrion, Gary W. Arendash works on issues like Hippocampus, which are connected to Melatonin, Melatonin receptor, Receptor and Presenilin.
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A beta peptide vaccination prevents memory loss in an animal model of Alzheimer's disease.
Dave Morgan;David M. Diamond;Paul E. Gottschall;Kenneth E. Ugen.
Caffeine protects Alzheimer's mice against cognitive impairment and reduces brain β-amyloid production
G.W. Arendash;W. Schleif;K. Rezai-Zadeh;E.K. Jackson.
Blueberry Supplementation Enhances Signaling and Prevents Behavioral Deficits in an Alzheimer Disease Model
J. A. Joseph;G. Arendash;M Gordon;David M. Diamond.
Nutritional Neuroscience (2003)
Green tea epigallocatechin-3-gallate (EGCG) reduces beta-amyloid mediated cognitive impairment and modulates tau pathology in Alzheimer transgenic mice.
Kavon Rezai-Zadeh;Gary W. Arendash;Huayan Hou;Frank Fernandez.
Brain Research (2008)
Progressive, age-related behavioral impairments in transgenic mice carrying both mutant amyloid precursor protein and presenilin-1 transgenes.
Gary W. Arendash;David L. King;Marcia N. Gordon;Dave Morgan.
Brain Research (2001)
Effects of discrete lesions of the sexually dimorphic nucleus of the preoptic area or other medial preoptic regions on the sexual behavior of male rats.
Gary W. Arendash;Roger A. Gorski.
Brain Research Bulletin (1983)
Evidence That Oxidative Stress Is Associated with the Pathophysiology of Inherited Hydrocephalus in the H-Tx Rat Model
D.J. Socci;K.B. Bjugstad;H.C. Jones;J.V. Pattisapu.
Experimental Neurology (1999)
Environmental enrichment improves cognition in aged Alzheimer's transgenic mice despite stable beta-amyloid deposition.
Gary W Arendash;Marcos F Garcia;David A Costa;Jennifer R Cracchiolo.
Caffeine reverses cognitive impairment and decreases brain amyloid-β levels in aged Alzheimer's disease mice.
Gary W. Arendash;Takashi Mori;Chuanhai Cao;Malgorzata Mamcarz.
Journal of Alzheimer's Disease (2009)
Behavioral characterization of the Tg2576 transgenic model of Alzheimer's disease through 19 months.
David L King;Gary W Arendash.
Physiology & Behavior (2002)
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