His main research concerns Long-term potentiation, Neuroscience, Cell biology, Synaptic plasticity and Alzheimer's disease. His Long-term potentiation research integrates issues from Postsynaptic potential, Neurotransmitter, Protein kinase A, Neurotransmission and Hippocampal formation. His Neuroscience research is multidisciplinary, incorporating elements of Epigenetics in learning and memory and Histone deacetylase 5.
His work deals with themes such as Synapse, Biochemistry, Nitric oxide and Transgene, which intersect with Cell biology. His Synaptic plasticity research includes elements of Neuroplasticity and Amyloid beta. His study looks at the relationship between Alzheimer's disease and fields such as Mitochondrion, as well as how they intersect with chemical problems.
Ottavio Arancio mainly investigates Neuroscience, Long-term potentiation, Synaptic plasticity, Cell biology and Alzheimer's disease. His Neuroscience study integrates concerns from other disciplines, such as Disease, Neurodegeneration, Amyloid and Neurotransmission. His Neurotransmission research incorporates themes from NMDA receptor, Anatomy, Excitatory postsynaptic potential and Neurotransmitter.
His Long-term potentiation research is multidisciplinary, incorporating elements of Hippocampal formation, Endocrinology, Hippocampus and Protein kinase A. His Cell biology study combines topics from a wide range of disciplines, such as AMPA receptor and Receptor, Biochemistry. His Alzheimer's disease research focuses on Transgene and how it relates to Protein phosphatase 2.
Ottavio Arancio mainly focuses on Synaptic plasticity, Cell biology, Neuroscience, Long-term potentiation and Neuroinflammation. His Synaptic plasticity research is multidisciplinary, relying on both Protein phosphatase 2, Electrophysiology, Transgene, Neurotransmission and Methyltransferase. His research integrates issues of Endocrinology, Genetically modified mouse and Internal medicine in his study of Protein phosphatase 2.
His studies deal with areas such as Postsynaptic density, Mitophagy, Single-Chain Variable Fragment Antibody and Neural facilitation as well as Cell biology. Ottavio Arancio frequently studies issues relating to Tau protein and Neuroscience. His study in Long-term potentiation is interdisciplinary in nature, drawing from both Neurotransmitter, Neuroplasticity, Neurodegeneration and Memory impairment.
Ottavio Arancio focuses on Synaptic plasticity, Long-term potentiation, Neuroscience, Cell biology and Drug discovery. His Synaptic plasticity study incorporates themes from Biochemistry of Alzheimer's disease and Electrophysiology. Ottavio Arancio interconnects Postsynaptic potential, Neurotransmitter, Neurotransmission, Memory impairment and Neuroplasticity in the investigation of issues within Long-term potentiation.
He integrates many fields in his works, including Neuroscience and Behavioral methods. His research in Cell biology intersects with topics in Mitophagy, BECN1 and Synaptic tagging. His Drug discovery study combines topics in areas such as Neurotoxicity, Sildenafil, cGMP-specific phosphodiesterase type 5, Signal transduction and Pharmacology.
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Requirement of Hippocampal Neurogenesis for the Behavioral Effects of Antidepressants
Luca Santarelli;Michael Saxe;Cornelius Gross;Alexandre Surget.
ABAD Directly Links Aß to Mitochondrial Toxicity in Alzheimer's Disease
Joyce W. Lustbader;Maurizio Cirilli;Chang Lin;Hong Wei Xu;Hong Wei Xu.
Tests of the roles of two diffusible substances in long-term potentiation: evidence for nitric oxide as a possible early retrograde messenger.
Thomas J. O'dell;Robert D. Hawkins;Eric R. Kandel;Ottavio Arancio.
Proceedings of the National Academy of Sciences of the United States of America (1991)
Cyclophilin D deficiency attenuates mitochondrial and neuronal perturbation and ameliorates learning and memory in Alzheimer's disease.
Heng Du;Lan Guo;Fang Fang;Doris Chen.
Nature Medicine (2008)
Loss of mTOR-Dependent Macroautophagy Causes Autistic-like Synaptic Pruning Deficits
Guomei Tang;Kathryn Gudsnuk;Sheng-Han Kuo;Marisa L. Cotrina;Marisa L. Cotrina.
Picomolar amyloid-beta positively modulates synaptic plasticity and memory in hippocampus.
Daniela Puzzo;Lucia Privitera;Elena Leznik;Mauro Fà.
The Journal of Neuroscience (2008)
Amyloid β-peptide inhibition of the PKA/CREB pathway and long-term potentiation: Reversibility by drugs that enhance cAMP signaling
Ottavio V. Vitolo;Antonino Sant'Angelo;Vincenzo Costanzo;Fortunato Battaglia.
Proceedings of the National Academy of Sciences of the United States of America (2002)
Learning and Memory and Synaptic Plasticity Are Impaired in a Mouse Model of Rett Syndrome
Paolo Moretti;Jonathan M. Levenson;Fortunato Battaglia;Richard Atkinson.
The Journal of Neuroscience (2006)
Persistent improvement in synaptic and cognitive functions in an Alzheimer mouse model after rolipram treatment
Bing Gong;Ottavio V. Vitolo;Fabrizio Trinchese;Shumin Liu.
Journal of Clinical Investigation (2004)
NITRIC OXIDE ACTS DIRECTLY IN THE PRESYNAPTIC NEURON TO PRODUCE LONG-TERM POTENTIATION IN CULTURED HIPPOCAMPAL NEURONS
Ottavio Arancio;Michael Kiebler;C.Justin Lee;Varda Lev-Ram.
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