Robert D. Hawkins spends much of his time researching Long-term potentiation, Neuroscience, Hippocampus, Hippocampal formation and Cell biology. His research in Long-term potentiation intersects with topics in Synaptic plasticity and Nitric oxide. His works in Aplysia and Facilitation are all subjects of inquiry into Neuroscience.
The concepts of his Aplysia study are interwoven with issues in Withdrawal reflex and Reflex. His work deals with themes such as NMDA receptor, Receptor, Schaffer collateral and Homosynaptic plasticity, which intersect with Hippocampal formation. His work on Protein kinase A as part of his general Cell biology study is frequently connected to Mossy fiber, thereby bridging the divide between different branches of science.
The scientist’s investigation covers issues in Neuroscience, Aplysia, Long-term potentiation, Postsynaptic potential and Synaptic plasticity. As a part of the same scientific family, Robert D. Hawkins mostly works in the field of Neuroscience, focusing on Metaplasticity and, on occasion, Memory consolidation. Robert D. Hawkins combines subjects such as Stimulation, Withdrawal reflex, Reflex and Sensitization with his study of Aplysia.
His Long-term potentiation research integrates issues from Protein kinase A, Hippocampal formation, Nitric oxide, Cell biology and Hippocampus. He focuses mostly in the field of Hippocampal formation, narrowing it down to matters related to NMDA receptor and, in some cases, Biophysics. He has researched Postsynaptic potential in several fields, including Synapse, Retrograde signaling and Excitatory postsynaptic potential.
Robert D. Hawkins mainly investigates Neuroscience, Synaptic plasticity, Aplysia, Postsynaptic potential and Nonsynaptic plasticity. His study ties his expertise on Long-term potentiation together with the subject of Neuroscience. The Long-term potentiation study combines topics in areas such as Glutamate receptor and Long-term depression.
His Aplysia research includes elements of Neurotrophin, HCN channel and Sensory neuron. His Sensory neuron study combines topics in areas such as Voltage clamp, Reflex and Serotonin. His work in Neuron covers topics such as Autocrine signalling which are related to areas like Retrograde signaling and Synapse.
His primary areas of investigation include Neuroscience, Postsynaptic potential, Synaptic plasticity, Nonsynaptic plasticity and Neural facilitation. The various areas that Robert D. Hawkins examines in his Neuroscience study include Long-term potentiation and Metaplasticity. His Long-term potentiation study integrates concerns from other disciplines, such as Glutamate receptor, Associative learning and Long-term depression.
Robert D. Hawkins combines subjects such as Ca2+/calmodulin-dependent protein kinase and Intermediate-term memory with his study of Metaplasticity. His Neural facilitation study which covers Post-tetanic potentiation that intersects with Synaptic augmentation and Synaptic fatigue. His Aplysia study combines topics in areas such as NMDA receptor and Hyperpolarization.
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Tests of the roles of two diffusible substances in long-term potentiation: evidence for nitric oxide as a possible early retrograde messenger.
Thomas J. O'dell;Robert D. Hawkins;Eric R. Kandel;Ottavio Arancio.
Proceedings of the National Academy of Sciences of the United States of America (1991)
A cellular mechanism of classical conditioning in Aplysia: activity-dependent amplification of presynaptic facilitation.
R. D. Hawkins;T. W. Abrams;T. J. Carew;E. R. Kandel.
Age-related defects in spatial memory are correlated with defects in the late phase of hippocampal long-term potentiation in vitro and are attenuated by drugs that enhance the cAMP signaling pathway.
Mary Elizabeth Bach;Mark Barad;Hyeon Son;Min Zhuo.
Proceedings of the National Academy of Sciences of the United States of America (1999)
Nitric oxide and carbon monoxide produce activity-dependent long-term synaptic enhancement in hippocampus
Min Zhuo;Scott A. Small;Eric R. Kandel;Robert D. Hawkins.
Abolition of Long-Term Stability of New Hippocampal Place Cell Maps by NMDA Receptor Blockade
Clifford Kentros;Eric Hargreaves;Robert D. Hawkins;Eric R. Kandel.
Nitric Oxide Signaling Contributes to Late-Phase LTP and CREB Phosphorylation in the Hippocampus
Yun-Fei Lu;Eric R. Kandel;Eric R. Kandel;Robert D. Hawkins.
The Journal of Neuroscience (1999)
Learning to Modulate Transmitter Release: Themes and Variations in Synaptic Plasticity
Robert D. Hawkins;Eric R. Kandel;Steven A. Siegelbaum.
Annual Review of Neuroscience (1993)
Is there a cell-biological alphabet for simple forms of learning?
Robert D. Hawkins;Eric R. Kandel.
Psychological Review (1984)
Differential classical conditioning of a defensive withdrawal reflex in Aplysia californica
TJ Carew;RD Hawkins;ER Kandel.
Role of guanylyl cyclase and cGMP-dependent protein kinase in long-term potentiation
Min Zhuo;Yinghe Hu;Carsten Schultz;Eric R. Kandel;Eric R. Kandel;Eric R. Kandel.
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