His main research concerns Alzheimer's disease, Neuroscience, Dementia, Hippocampal formation and VPS35. Internal medicine and Disease are the areas that his Alzheimer's disease study falls under. He incorporates Neuroscience and In situ hybridization in his research.
Scott A. Small combines subjects such as Cohort study, Gerontology, Cognitive test, Apolipoprotein E and Cohort with his study of Dementia. Scott A. Small interconnects Prepulse inhibition, Hippocampus, Prefrontal cortex and Cognitive decline in the investigation of issues within Hippocampal formation. His VPS35 study is related to the wider topic of Retromer.
His primary scientific interests are in Neuroscience, Disease, Hippocampal formation, Hippocampus and Cell biology. His research integrates issues of Alzheimer's disease, Cerebral blood volume and Neurodegeneration in his study of Neuroscience. His Alzheimer's disease research incorporates themes from Endocrinology, Apolipoprotein E, Dementia and Degenerative disease.
His Hippocampal formation research incorporates elements of Magnetic resonance imaging and Pathology. The Hippocampus study combines topics in areas such as Stroke and Functional imaging. His work on Retromer as part of general Cell biology study is frequently connected to SORL1, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
Scott A. Small mainly focuses on Endosome, Cell biology, Neuroscience, Retromer and Disease. His Endosome research focuses on subjects like Amyloid precursor protein, which are linked to Computational biology, Neuronal ceroid lipofuscinosis and Endoplasmic reticulum. His biological study spans a wide range of topics, including Cell membrane and Ectodomain.
Many of his research projects under Neuroscience are closely connected to Oxygen metabolism with Oxygen metabolism, tying the diverse disciplines of science together. The VPS35 and Retromer complex research Scott A. Small does as part of his general Retromer study is frequently linked to other disciplines of science, such as CLN3, therefore creating a link between diverse domains of science. His work carried out in the field of Disease brings together such families of science as Biomarker, Cerebral cortex and Vulnerability.
Scott A. Small mainly investigates Hippocampal formation, Cell biology, Retromer, Endosome and Psychosis. He combines subjects such as Glutamate receptor, Premovement neuronal activity, Pathology, Microbubbles and Neuroimaging with his study of Hippocampal formation. His Neuroimaging study is associated with Neuroscience.
His work in the fields of Neuropil and Hippocampus overlaps with other areas such as Context. The various areas that Scott A. Small examines in his Cell biology study include Neuronal ceroid lipofuscinosis and Amyloid precursor protein. His research integrates issues of Cholera toxin, Endocytic recycling, Exocytosis, Secretion and PDZ domain in his study of Retromer.
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Imaging correlates of brain function in monkeys and rats isolates a hippocampal subregion differentially vulnerable to aging.
Scott A. Small;Monica K. Chawla;Michael Buonocore;Peter R. Rapp.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Plasma Amyloid Beta-Peptide 1-42 and Incipient Alzheimer's Disease
Richard Paul Mayeux;Mingxin Tang;Diane Jacobs;Jennifer J. Manly.
Annals of Neurology (1999)
Endogenous estrogen levels and Alzheimer’s disease among postmenopausal women
J.J. Manly;C.A. Merchant;D.M. Jacobs;S.A. Small.
Low-density lipoprotein cholesterol and the risk of dementia with stroke.
Joan T. Moroney;Ming Xin Tang;Lars Berglund;Scott Small.
Enhancing dentate gyrus function with dietary flavanols improves cognition in older adults
Adam M. Brickman;Usman A. Khan;Frank A. Provenzano;Lok Kin Yeung.
Nature Neuroscience (2014)
Cognitive test performance among nondemented elderly African Americans and whites
Jennifer J. Manly;Diane Jacobs;Mary Sano;Karen L. Bell.
Brain Morphology in Older African Americans, Caribbean Hispanics, and Whites From Northern Manhattan
Adam M. Brickman;Nicole Schupf;Jennifer J. Manly;José A. Luchsinger.
JAMA Neurology (2008)
Retromer deficiency observed in Alzheimer's disease causes hippocampal dysfunction, neurodegeneration, and Aβ accumulation
Alim Muhammad;Ingrid Flores;Hong Zhang;Rui Yu.
Proceedings of the National Academy of Sciences of the United States of America (2008)
Memory performance in healthy elderly without Alzheimer's disease: effects of time and apolipoprotein-E.
Richard Mayeux;Scott A Small;Ming-Xin Tang;Benjamin Tycko.
Neurobiology of Aging (2001)
Retromer in Alzheimer disease, Parkinson disease and other neurological disorders
Scott A. Small;Gregory A. Petsko.
Nature Reviews Neuroscience (2015)
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