H-Index & Metrics Best Publications

H-Index & Metrics

Discipline name H-index Citations Publications World Ranking National Ranking
Medicine D-index 75 Citations 19,051 187 World Ranking 12002 National Ranking 240

Research.com Recognitions

Awards & Achievements

2005 - Polish Academy of Science

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Internal medicine

Tadeusz Wieloch focuses on Ischemia, Internal medicine, Endocrinology, Brain damage and Hippocampus. His work deals with themes such as Necrosis, AMPA receptor, Anesthesia and Pharmacology, which intersect with Ischemia. In his research on the topic of Internal medicine, Metabolism is strongly related with Extracellular.

The concepts of his Endocrinology study are interwoven with issues in Cell, Mitogen-activated protein kinase, Neuronal damage and Protein kinase A. His Brain damage research includes themes of Dentate gyrus, Functional recovery, Regenerative process, Cell growth and Gene expression profiling. His Hippocampus research integrates issues from Constriction, Glutamate receptor, Status epilepticus, Blood pressure and Hippocampal formation.

His most cited work include:

  • Models for studying long-term recovery following forebrain ischemia in the rat. 2. A 2-vessel occlusion model (686 citations)
  • Hypoglycemia-induced neuronal damage prevented by an N-methyl-D-aspartate antagonist. (637 citations)
  • Uncoupling protein-2 prevents neuronal death and diminishes brain dysfunction after stroke and brain trauma. (448 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Internal medicine, Endocrinology, Ischemia, Neuroscience and Anesthesia. His Internal medicine course of study focuses on Protein kinase A and Protein kinase C. Tadeusz Wieloch has researched Endocrinology in several fields, including Glutamate receptor and Neocortex.

Tadeusz Wieloch combines subjects such as Hippocampal formation, Hippocampus and Neuroprotection, Pharmacology with his study of Ischemia. His studies deal with areas such as Programmed cell death and Cell biology as well as Neuroprotection. Functional recovery, Neurology and Inflammation is closely connected to Stroke in his research, which is encompassed under the umbrella topic of Neuroscience.

He most often published in these fields:

  • Internal medicine (39.83%)
  • Endocrinology (39.00%)
  • Ischemia (38.17%)

What were the highlights of his more recent work (between 2006-2021)?

  • Neuroscience (23.24%)
  • Stroke (11.20%)
  • Internal medicine (39.83%)

In recent papers he was focusing on the following fields of study:

His primary areas of investigation include Neuroscience, Stroke, Internal medicine, Endocrinology and Cell biology. His work in the fields of Internal medicine, such as Tyrosine phosphorylation, intersects with other areas such as Apolipoprotein E. His studies in Endocrinology integrate themes in fields like Inflammation, Microglia, Immunology and Receptor.

His Cell biology study combines topics from a wide range of disciplines, such as Excitotoxicity, Calpain and Cytoskeleton. His Neuroprotection research includes elements of Hippocampal formation, Anesthesia, Erythropoietin and Ischemia. The various areas that Tadeusz Wieloch examines in his Hippocampal formation study include Hippocampus, Programmed cell death, Rho-associated protein kinase and Xeroderma pigmentosum.

Between 2006 and 2021, his most popular works were:

  • Improving outcome after stroke: overcoming the translational roadblock. (207 citations)
  • Tyrosine phosphorylation and activation of mitogen-activated protein kinase in the rat brain following transient cerebral ischemia. (141 citations)
  • Pharmacological stimulation of sigma-1 receptors has neurorestorative effects in experimental parkinsonism (117 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Internal medicine

His main research concerns Neuroscience, Internal medicine, Neuroprotection, Receptor and Endocrinology. His research integrates issues of Ischemia and Cell biology in his study of Neuroscience. His Ischemia study integrates concerns from other disciplines, such as Apolipoprotein D, Apolipoprotein B and Lipid metabolism.

His Internal medicine research is multidisciplinary, incorporating elements of Levodopa and Functional recovery. In his study, which falls under the umbrella issue of Neuroprotection, Thalamus, Programmed cell death, Microglia, Phenylephrine and Propidium iodide is strongly linked to Hippocampal formation. His work investigates the relationship between Endocrinology and topics such as Inflammation that intersect with problems in Brain ischemia and Receptor antagonist.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Models for studying long-term recovery following forebrain ischemia in the rat. 2. A 2-vessel occlusion model

Maj‐Lis ‐L Smith;George Bendek;Nils Dahlgren;Ingmar Rosén.
Acta Neurologica Scandinavica (1984)

978 Citations

Hypoglycemia-induced neuronal damage prevented by an N-methyl-D-aspartate antagonist.

Tadeusz Wieloch.
Science (1985)

850 Citations

Uncoupling protein-2 prevents neuronal death and diminishes brain dysfunction after stroke and brain trauma.

Gustav Mattiasson;Mehrdad Shamloo;Gunilla Gido;Kavitha Mathi.
Nature Medicine (2003)

576 Citations

Calcium Accumulation and Neuronal Damage in the Rat Hippocampus following Cerebral Ischemia

J. K. Deshpande;B. K. Siesjö;T. Wieloch.
Journal of Cerebral Blood Flow and Metabolism (1987)

464 Citations

Cyclosporin A, But Not FK 506, Protects Mitochondria and Neurons against Hypoglycemic Damage and Implicates the Mitochondrial Permeability Transition in Cell Death

Hans Friberg;Michel Ferrand-Drake;Finn Bengtsson;Andrew P. Halestrap.
The Journal of Neuroscience (1998)

452 Citations

The distribution of hypoglycemic brain damage.

R. N. Auer;T. Wieloch;Y. Olsson;B. K. Siesjö.
Acta Neuropathologica (1984)

442 Citations

Mechanisms of neural plasticity following brain injury

Tadeusz Wieloch;Karoly Nikolich.
Current Opinion in Neurobiology (2006)

409 Citations

Plasma fibronectin supports neuronal survival and reduces brain injury following transient focal cerebral ischemia but is not essential for skin-wound healing and hemostasis.

Takao Sakai;Kamin J. Johnson;Kamin J. Johnson;Kamin J. Johnson;Michihiro Murozono;Michihiro Murozono;Keiko Sakai.
Nature Medicine (2001)

394 Citations

Postischemic blockade of AMPA but not NMDA receptors mitigates neuronal damage in the rat brain following transient severe cerebral ischemia

B. Nellgård;T. Wieloch.
Journal of Cerebral Blood Flow and Metabolism (1992)

369 Citations

Novel pharmacologic strategies in the treatment of experimental traumatic brain injury : 1998

Tracy K. McINTOSH;Marianne Juhler;Tadeusz Wieloch.
Journal of Neurotrauma (1998)

361 Citations

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