His scientific interests lie mostly in Systemic lupus erythematosus, Lupus nephritis, Immunology, Autoantibody and Lupus erythematosus. His Systemic lupus erythematosus study incorporates themes from Epitope and T cell. Syamal K. Datta interconnects Kidney, T-cell receptor, Fibrosis, Antibody and Nephritis in the investigation of issues within Lupus nephritis.
His research on Immunology often connects related areas such as Hamster. His Autoantibody research incorporates themes from Molecular biology and CD40. His work in Molecular biology tackles topics such as Antigen which are related to areas like Mesangium.
Syamal K. Datta mainly investigates Immunology, Systemic lupus erythematosus, Autoantibody, Lupus erythematosus and Molecular biology. His research on Immunology frequently connects to adjacent areas such as CD40. His Systemic lupus erythematosus research incorporates elements of Cytotoxic T cell, B cell, Epitope, Lupus nephritis and Histone.
In his study, which falls under the umbrella issue of Lupus nephritis, Glomerulonephritis is strongly linked to Nephritis. Syamal K. Datta interconnects T cell, T-cell receptor, T lymphocyte and Immunoglobulin G in the investigation of issues within Autoantibody. His research integrates issues of Immune complex and Immunopathology in his study of Lupus erythematosus.
The scientist’s investigation covers issues in Immunology, Systemic lupus erythematosus, Autoimmunity, IL-2 receptor and Cell biology. His study in Epitope, Autoantibody, Lupus erythematosus, Immune system and Antigen-presenting cell falls under the purview of Immunology. His research in Lupus erythematosus intersects with topics in Hematopoietic stem cell transplantation, Autoimmune disease and Rituximab.
His work carried out in the field of Systemic lupus erythematosus brings together such families of science as Cytotoxic T cell, Low dose, Antigen, Lupus nephritis and Histone. His Cell biology study integrates concerns from other disciplines, such as T cell, MHC class II, T-cell receptor, Apoptosis and Transformation. Syamal K. Datta works mostly in the field of ZAP70, limiting it down to concerns involving Programmed cell death and, occasionally, Molecular biology.
His primary areas of investigation include Systemic lupus erythematosus, Immunology, Immune system, Lupus erythematosus and CD8. His biological study spans a wide range of topics, including Cytotoxic T cell, Autoimmunity, ZAP70 and Programmed cell death. His Cytotoxic T cell research incorporates themes from Fas ligand, Autoantibody, Apoptosis and Molecular biology.
His research investigates the connection between Immune system and topics such as Lupus nephritis that intersect with issues in Antigen presentation, Antigen, Antigen-presenting cell and Cancer research. His Lupus erythematosus study frequently involves adjacent topics like Autoimmune disease. His CD8 research integrates issues from Epitope, Regulatory T cell, IL-2 receptor and Adoptive cell transfer.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Hyperexpression of CD40 ligand by B and T cells in human lupus and its role in pathogenic autoantibody production.
Ami Desai-Mehta;Liangjun Lu;Rosalind Ramsey-Goldman;Syamal K. Datta.
Journal of Clinical Investigation (1996)
Nucleosome: A major immunogen for pathogenic autoantibody-inducing T cells of lupus
Chandra Mohan;Sharlene Adams;Valerie Stanik;Syamal K. Datta.
Journal of Experimental Medicine (1993)
T cell receptor alpha/beta expressing double-negative (CD4-/CD8-) and CD4+ T helper cells in humans augment the production of pathogenic anti-DNA autoantibodies associated with lupus nephritis.
S Shivakumar;G C Tsokos;S K Datta.
Journal of Immunology (1989)
INTERACTION BETWEEN CD40 AND ITS LIGAND GP39 IN THE DEVELOPMENT OF MURINE LUPUS NEPHRITIS
C. Mohan;Y. Shi;J. D. Laman;Syamal Kumar Datta.
Journal of Immunology (1995)
Induction of a cationic shift in IgG anti-DNA autoantibodies. Role of T helper cells with classical and novel phenotypes in three murine models of lupus nephritis.
S K Datta;H Patel;D Berry.
Journal of Experimental Medicine (1987)
Anti-CD40 Ligand Antibody Treatment of SNF1 Mice with Established Nephritis: Preservation of Kidney Function
Susan L. Kalled;Anne H. Cutler;Syamal K. Datta;David W. Thomas.
Journal of Immunology (1998)
Low-dose peptide tolerance therapy of lupus generates plasmacytoid dendritic cells that cause expansion of autoantigen-specific regulatory T cells and contraction of inflammatory Th17 cells.
Hee Kap Kang;Michael Liu;Syamal K. Datta.
Journal of Immunology (2007)
Very Low-Dose Tolerance with Nucleosomal Peptides Controls Lupus and Induces Potent Regulatory T Cell Subsets
Hee Kap Kang;Marissa A. Michaels;Beate R. Berner;Syamal K. Datta.
Journal of Immunology (2005)
Anti-DNA antibodies form immune deposits at distinct glomerular and vascular sites.
Demetrios V. Vlahakos;Mary H. Foster;Sharlene Adams;Michael Katz.
Kidney International (1992)
Major peptide autoepitopes for nucleosome-specific T cells of human lupus
Liangjun Lu;Arunan Kaliyaperumal;Dimitrios T. Boumpas;Syamal K. Datta.
Journal of Clinical Investigation (1999)
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