Stephen M. Factor spends much of his time researching Internal medicine, Endocrinology, Pathology, Pathogenesis and Cardiomyopathy. The various areas that Stephen M. Factor examines in his Internal medicine study include Diabetes mellitus and Cardiology. His Endocrinology study combines topics in areas such as Myosin, Cell biology and Genetically modified mouse.
His studies in Pathology integrate themes in fields like Inflammation and Extracellular matrix. His work carried out in the field of Pathogenesis brings together such families of science as Fibrosis, Trypanosoma cruzi, Microcirculation and Chagas disease. His work deals with themes such as Verapamil, Radiology, Electrocardiography, Pulmonary wedge pressure and Hamster, which intersect with Cardiomyopathy.
His scientific interests lie mostly in Internal medicine, Cardiology, Pathology, Endocrinology and Cardiomyopathy. His work in Internal medicine is not limited to one particular discipline; it also encompasses Diabetes mellitus. His biological study spans a wide range of topics, including Surgery and Complication.
The study incorporates disciplines such as Inflammation and Anatomy in addition to Pathology. His Endocrinology research is multidisciplinary, relying on both Cell biology, Myosin and Genetically modified mouse. His Cardiomyopathy study integrates concerns from other disciplines, such as Myocyte, Verapamil, Necrosis, Microcirculation and Hamster.
His primary areas of study are Internal medicine, Trypanosoma cruzi, Endocrinology, Chagas disease and Immunology. Internal medicine and Cardiology are commonly linked in his work. His study in Endocrinology is interdisciplinary in nature, drawing from both Signal transduction and Cell biology.
The Chagas disease study combines topics in areas such as Fibrosis, Phosphoramidon, Nitric oxide synthase and Pathogenesis. Stephen M. Factor has researched Immunology in several fields, including Reperfusion injury and Myocarditis. His Pathology research integrates issues from Inflammation and Caveolae.
Stephen M. Factor spends much of his time researching Immunology, Pathogenesis, Myocyte, Internal medicine and Endocrinology. His research integrates issues of Trypanosoma cruzi, Heart disease and Myosin in his study of Pathogenesis. His Myocyte research includes themes of Caveolin 1, Muscle hypertrophy and Caveolae.
His Internal medicine research is mostly focused on the topic Adipose tissue. His work in Cardiac myocyte tackles topics such as Cancer research which are related to areas like Dilated cardiomyopathy and Heart failure. His biological study deals with issues like Inflammation, which deal with fields such as Pathology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
TBX1 Is Responsible for Cardiovascular Defects in Velo-Cardio-Facial/DiGeorge Syndrome
Sandra Merscher;Birgit Funke;Jonathan A. Epstein;Joerg Heyer.
Cell (2001)
A mechanistic role for cardiac myocyte apoptosis in heart failure
Detlef Wencker;Madhulika Chandra;Khanh Nguyen;Wenfeng Miao.
Journal of Clinical Investigation (2003)
A comparison of the pathological spectrum of hypertensive, diabetic, and hypertensive-diabetic heart disease.
K H van Hoeven;S M Factor.
Circulation (1990)
GLUT4 heterozygous knockout mice develop muscle insulin resistance and diabetes
Antine E. Stenbit;Tsu Shuen Tsao;Jing Li;Rémy Burcelin.
Nature Medicine (1997)
Cardiac defects and renal failure in mice with targeted mutations in Pkd2.
Guanqing Wu;G. S. Markowitz;Li Li;V. D. D'agati.
Nature Genetics (2000)
Microvascular spasm in the cardiomyopathic Syrian hamster: a preventable cause of focal myocardial necrosis.
S M Factor;T Minase;S Cho;R Dominitz.
Circulation (1982)
Capillary microaneurysms in the human diabetic heart.
Stephen M. Factor;Ellen M. Okun;Takashi Minase.
The New England Journal of Medicine (1980)
Hippocampal sclerosis : a common pathological feature of dementia in very old (≥80 years of age) humans
D. W. Dickson;P. Davies;C. Bevona;K. H. Van Hoeven.
Acta Neuropathologica (1994)
Caveolin-3 knock-out mice develop a progressive cardiomyopathy and show hyperactivation of the p42/44 MAPK cascade
Scott Eric Woodman;David S. Park;Alex W. Cohen;Michelle W C Cheung.
Journal of Biological Chemistry (2002)
Profound structural alterations of the extracellular collagen matrix in postischemic dysfunctional (stunned) but viable myocardium
Mengjia Zhao;Hong Zhang;Thomas F. Robinson;Stephen M. Factor.
Journal of the American College of Cardiology (1987)
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