World's Best Scientists 2026 revealed!

D-Index & Metrics

Medicine

D-Index
86
Citations
20507
World Ranking
14316
National Ranking
7240

Overview

Stephen M. Factor is affiliated with the Albert Einstein College of Medicine in the United States. The profile reflects an academic career focused on medical research conducted within this institution.

No specific recent papers, coauthors, publication venues, book publications, fields of study, subfields, main topics of work, or awards have been provided for this researcher. Therefore, there is no detailed information available regarding their scientific publications, collaborative network, or areas of specialized investigation.

As a medical researcher, Stephen M. Factor's professional activities likely involve contributions to the broader biomedical field, although specific details about their research focus or academic output are not present in the data.

Best Publications

  • TBX1 Is Responsible for Cardiovascular Defects in Velo-Cardio-Facial/DiGeorge Syndrome

    Sandra Merscher;Birgit Funke;Jonathan A. Epstein;Joerg Heyer

  • A mechanistic role for cardiac myocyte apoptosis in heart failure

    Detlef Wencker;Madhulika Chandra;Khanh Nguyen;Wenfeng Miao

  • GLUT4 heterozygous knockout mice develop muscle insulin resistance and diabetes

    Antine E. Stenbit;Tsu Shuen Tsao;Jing Li;Rémy Burcelin

  • A comparison of the pathological spectrum of hypertensive, diabetic, and hypertensive-diabetic heart disease.

    K H van Hoeven;S M Factor

  • Cardiac defects and renal failure in mice with targeted mutations in Pkd2.

    Guanqing Wu;G. S. Markowitz;Li Li;V. D. D'agati

  • Microvascular spasm in the cardiomyopathic Syrian hamster: a preventable cause of focal myocardial necrosis.

    S M Factor;T Minase;S Cho;R Dominitz

  • Capillary microaneurysms in the human diabetic heart.

    Stephen M. Factor;Ellen M. Okun;Takashi Minase

  • Hippocampal sclerosis : a common pathological feature of dementia in very old (≥80 years of age) humans

    D. W. Dickson;P. Davies;C. Bevona;K. H. Van Hoeven

  • Caveolin-3 knock-out mice develop a progressive cardiomyopathy and show hyperactivation of the p42/44 MAPK cascade

    Scott Eric Woodman;David S. Park;Alex W. Cohen;Michelle W C Cheung

  • Clinical and morphological features of human hypertensive-diabetic cardiomyopathy

    Stephen M. Factor;Takashi Minase;Edmund H. Sonnenblick

  • The heart as a suction pump.

    Thomas F. Robinson;Stephen M. Factor;Edmund H. Sonnenblick

  • Profound structural alterations of the extracellular collagen matrix in postischemic dysfunctional (stunned) but viable myocardium

    Mengjia Zhao;Hong Zhang;Thomas F. Robinson;Stephen M. Factor

  • Caveolin-1 null mice develop cardiac hypertrophy with hyperactivation of p42/44 MAP kinase in cardiac fibroblasts

    Alex W. Cohen;David S. Park;Scott E. Woodman;Terrence M. Williams

  • Results of endomyocardial biopsy in patients with spontaneous ventricular tachycardia but without apparent structural heart disease.

    J E Strain;R M Grose;S M Factor;J D Fisher

  • Fas pathway is a critical mediator of cardiac myocyte death and MI during ischemia-reperfusion in vivo.

    Peiyee Lee;Masataka Sata;David J. Lefer;Stephen M. Factor

  • Current understanding of immunity to Trypanosoma cruzi infection and pathogenesis of Chagas disease.

    Fabiana S. Machado;Walderez O. Dutra;Walderez O. Dutra;Lisia Esper;Kenneth J. Gollob;Kenneth J. Gollob

  • Cardiac troponin T mutations result in allele-specific phenotypes in a mouse model for hypertrophic cardiomyopathy

    Jil C. Tardiff;Timothy E. Hewett;Bradley M. Palmer;Charlotte Olsson

  • Caveolin-1/3 double-knockout mice are viable, but lack both muscle and non-muscle caveolae, and develop a severe cardiomyopathic phenotype.

    David S. Park;Scott E. Woodman;William Schubert;Alex W. Cohen

  • A truncated cardiac troponin T molecule in transgenic mice suggests multiple cellular mechanisms for familial hypertrophic cardiomyopathy.

    Jil C. Tardiff;Stephen M. Factor;Brian D. Tompkins;Timothy E. Hewett

  • Mice Expressing Mutant Myosin Heavy Chains Are a Model for Familial Hypertrophic Cardiomyopathy

    Karen L. Vikstrom;Stephen M. Factor;Leslie A. Leinwand

Frequent Co-Authors

Herbert B. Tanowitz
Herbert B. Tanowitz Albert Einstein College of Medicine
Louis M. Weiss
Louis M. Weiss Albert Einstein College of Medicine
Murray Wittner
Murray Wittner Yeshiva University
Edmund H. Sonnenblick
Edmund H. Sonnenblick Albert Einstein College of Medicine
Michael P. Lisanti
Michael P. Lisanti University of Salford
Leslie A. Leinwand
Leslie A. Leinwand University of Colorado Boulder
Richard N. Kitsis
Richard N. Kitsis Albert Einstein College of Medicine
Betty Diamond
Betty Diamond Feinstein Institute for Medical Research
Philipp E. Scherer
Philipp E. Scherer The University of Texas Southwestern Medical Center
Richard G. Pestell
Richard G. Pestell The Wistar Institute

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