D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 83 Citations 18,981 216 World Ranking 10649 National Ranking 5599

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Gene
  • Pathology

Stephen M. Factor spends much of his time researching Internal medicine, Endocrinology, Pathology, Pathogenesis and Cardiomyopathy. The various areas that Stephen M. Factor examines in his Internal medicine study include Diabetes mellitus and Cardiology. His Endocrinology study combines topics in areas such as Myosin, Cell biology and Genetically modified mouse.

His studies in Pathology integrate themes in fields like Inflammation and Extracellular matrix. His work carried out in the field of Pathogenesis brings together such families of science as Fibrosis, Trypanosoma cruzi, Microcirculation and Chagas disease. His work deals with themes such as Verapamil, Radiology, Electrocardiography, Pulmonary wedge pressure and Hamster, which intersect with Cardiomyopathy.

His most cited work include:

  • TBX1 Is Responsible for Cardiovascular Defects in Velo-Cardio-Facial/DiGeorge Syndrome (797 citations)
  • A mechanistic role for cardiac myocyte apoptosis in heart failure (624 citations)
  • Cardiac defects and renal failure in mice with targeted mutations in Pkd2. (308 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Internal medicine, Cardiology, Pathology, Endocrinology and Cardiomyopathy. His work in Internal medicine is not limited to one particular discipline; it also encompasses Diabetes mellitus. His biological study spans a wide range of topics, including Surgery and Complication.

The study incorporates disciplines such as Inflammation and Anatomy in addition to Pathology. His Endocrinology research is multidisciplinary, relying on both Cell biology, Myosin and Genetically modified mouse. His Cardiomyopathy study integrates concerns from other disciplines, such as Myocyte, Verapamil, Necrosis, Microcirculation and Hamster.

He most often published in these fields:

  • Internal medicine (48.08%)
  • Cardiology (28.37%)
  • Pathology (27.40%)

What were the highlights of his more recent work (between 2001-2014)?

  • Internal medicine (48.08%)
  • Trypanosoma cruzi (12.02%)
  • Endocrinology (22.60%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Internal medicine, Trypanosoma cruzi, Endocrinology, Chagas disease and Immunology. Internal medicine and Cardiology are commonly linked in his work. His study in Endocrinology is interdisciplinary in nature, drawing from both Signal transduction and Cell biology.

The Chagas disease study combines topics in areas such as Fibrosis, Phosphoramidon, Nitric oxide synthase and Pathogenesis. Stephen M. Factor has researched Immunology in several fields, including Reperfusion injury and Myocarditis. His Pathology research integrates issues from Inflammation and Caveolae.

Between 2001 and 2014, his most popular works were:

  • A mechanistic role for cardiac myocyte apoptosis in heart failure (624 citations)
  • Caveolin-3 knock-out mice develop a progressive cardiomyopathy and show hyperactivation of the p42/44 MAPK cascade (248 citations)
  • Caveolin-1 null mice develop cardiac hypertrophy with hyperactivation of p42/44 MAP kinase in cardiac fibroblasts (210 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Gene
  • Pathology

Stephen M. Factor spends much of his time researching Immunology, Pathogenesis, Myocyte, Internal medicine and Endocrinology. His research integrates issues of Trypanosoma cruzi, Heart disease and Myosin in his study of Pathogenesis. His Myocyte research includes themes of Caveolin 1, Muscle hypertrophy and Caveolae.

His Internal medicine research is mostly focused on the topic Adipose tissue. His work in Cardiac myocyte tackles topics such as Cancer research which are related to areas like Dilated cardiomyopathy and Heart failure. His biological study deals with issues like Inflammation, which deal with fields such as Pathology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

TBX1 Is Responsible for Cardiovascular Defects in Velo-Cardio-Facial/DiGeorge Syndrome

Sandra Merscher;Birgit Funke;Jonathan A. Epstein;Joerg Heyer.
Cell (2001)

1001 Citations

A mechanistic role for cardiac myocyte apoptosis in heart failure

Detlef Wencker;Madhulika Chandra;Khanh Nguyen;Wenfeng Miao.
Journal of Clinical Investigation (2003)

901 Citations

A comparison of the pathological spectrum of hypertensive, diabetic, and hypertensive-diabetic heart disease.

K H van Hoeven;S M Factor.
Circulation (1990)

438 Citations

GLUT4 heterozygous knockout mice develop muscle insulin resistance and diabetes

Antine E. Stenbit;Tsu Shuen Tsao;Jing Li;Rémy Burcelin.
Nature Medicine (1997)

436 Citations

Cardiac defects and renal failure in mice with targeted mutations in Pkd2.

Guanqing Wu;G. S. Markowitz;Li Li;V. D. D'agati.
Nature Genetics (2000)

400 Citations

Microvascular spasm in the cardiomyopathic Syrian hamster: a preventable cause of focal myocardial necrosis.

S M Factor;T Minase;S Cho;R Dominitz.
Circulation (1982)

397 Citations

Capillary microaneurysms in the human diabetic heart.

Stephen M. Factor;Ellen M. Okun;Takashi Minase.
The New England Journal of Medicine (1980)

387 Citations

Hippocampal sclerosis : a common pathological feature of dementia in very old (≥80 years of age) humans

D. W. Dickson;P. Davies;C. Bevona;K. H. Van Hoeven.
Acta Neuropathologica (1994)

338 Citations

Caveolin-3 knock-out mice develop a progressive cardiomyopathy and show hyperactivation of the p42/44 MAPK cascade

Scott Eric Woodman;David S. Park;Alex W. Cohen;Michelle W C Cheung.
Journal of Biological Chemistry (2002)

325 Citations

Profound structural alterations of the extracellular collagen matrix in postischemic dysfunctional (stunned) but viable myocardium

Mengjia Zhao;Hong Zhang;Thomas F. Robinson;Stephen M. Factor.
Journal of the American College of Cardiology (1987)

321 Citations

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