His scientific interests lie mostly in Immunology, Toxoplasma gondii, Virology, Microsporidia and Trypanosoma cruzi. His Immunology study integrates concerns from other disciplines, such as Disease and Myocarditis. His study in Toxoplasma gondii is interdisciplinary in nature, drawing from both Cyst, Toxoplasmosis, Astrocyte, Antigen and Gene.
His research integrates issues of Transmission, Pregnancy, Serology and Encephalitis in his study of Toxoplasmosis. Louis M. Weiss focuses mostly in the field of Microsporidia, narrowing it down to matters related to Phylogenetics and, in some cases, Ribosomal RNA and Phylogenetic tree. His studies in Trypanosoma cruzi integrate themes in fields like Adipose tissue and Cardiomyopathy.
His primary scientific interests are in Immunology, Toxoplasma gondii, Virology, Microsporidia and Trypanosoma cruzi. His work on Immunology is being expanded to include thematically relevant topics such as Disease. His Toxoplasma gondii study also includes
His research on Virology often connects related areas such as Acquired immunodeficiency syndrome. In his study, Phylogenetic tree is strongly linked to Phylogenetics, which falls under the umbrella field of Microsporidia. His study focuses on the intersection of Trypanosoma cruzi and fields such as Chagas disease with connections in the field of Heart disease and Internal medicine.
His main research concerns Toxoplasma gondii, Immunology, Cell biology, Trypanosoma cruzi and Intracellular parasite. His Toxoplasma gondii research is multidisciplinary, relying on both Cyst, Toxoplasmosis, Microbiology, Vacuole and Effector. The various areas that he examines in his Microbiology study include Cell culture, Virology and Virulence.
His Immunology study combines topics in areas such as Asymptomatic and Disease. His biological study spans a wide range of topics, including Proteome, Polar tube, Proteomics and Sporoplasm. He interconnects Cardiomyopathy, Chagas disease and Immune system in the investigation of issues within Trypanosoma cruzi.
His primary areas of investigation include Toxoplasma gondii, Cell biology, Microbiology, Immunology and Intracellular parasite. His Toxoplasma gondii research is multidisciplinary, incorporating perspectives in Cyst, Chronic infection, Toxoplasmosis and Gene. His Cell biology research includes elements of Proteome, Glycosylation, Proteomics, Mutant and Cell division.
His studies deal with areas such as Trypanosoma cruzi, Chagas disease, Virology and Virulence as well as Microbiology. His work carried out in the field of Virology brings together such families of science as Pyrimethamine and Cell culture, Growth inhibition. His study ties his expertise on Microsporidia together with the subject of Immunology.
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Toxoplasma gondii: from animals to humans
Astrid M. Tenter;Anja R. Heckeroth;Louis M. Weiss.
International Journal for Parasitology (2000)
Toxoplasmosis: a history of clinical observations
Louis M. Weiss;Jitender. P. Dubey.
International Journal for Parasitology (2009)
The Microsporidia And Microsporidiosis
Murray Wittner;Louis M. Weiss.
(1999)
Microsporidiosis: current status.
Elizabeth S Didier;Louis M Weiss.
Current Opinion in Infectious Diseases (2006)
Toxoplasma gondii: the model apicomplexan
Kami Kim;Louis M. Weiss.
International Journal for Parasitology (2004)
Microsporidiosis: Not just in AIDS patients
Elizabeth S. Didier;Louis M. Weiss.
Current Opinion in Infectious Diseases (2011)
The development and biology of bradyzoites of Toxoplasma gondii.
Louis M. Weiss;Kami Kim.
Frontiers in Bioscience (2000)
Zoonotic babesiosis: overview of the disease and novel aspects of pathogen identity.
Jeremy Gray;Annetta Zintl;Anke Hildebrandt;Klaus Peter Hunfeld.
Ticks and Tick-borne Diseases (2010)
Caveolin-1 null mice develop cardiac hypertrophy with hyperactivation of p42/44 MAP kinase in cardiac fibroblasts
Alex W. Cohen;David S. Park;Scott E. Woodman;Terrence M. Williams.
American Journal of Physiology-cell Physiology (2003)
Caveolin-1/3 double-knockout mice are viable, but lack both muscle and non-muscle caveolae, and develop a severe cardiomyopathic phenotype.
David S. Park;Scott E. Woodman;William Schubert;Alex W. Cohen.
American Journal of Pathology (2002)
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