Herbert B. Tanowitz mainly focuses on Immunology, Chagas disease, Trypanosoma cruzi, Internal medicine and Pathology. The various areas that he examines in his Immunology study include Cardiomyopathy, Myocarditis and Cancer research. His Chagas disease research incorporates elements of Heart disease, Disease, Immune system and Pathogenesis.
His study explores the link between Trypanosoma cruzi and topics such as Adipose tissue that cross with problems in Lipid droplet, Adipokine and Receptor. His Internal medicine study integrates concerns from other disciplines, such as Endocrinology and Parasitemia. His Pathology research is multidisciplinary, incorporating elements of Acanthamoeba, Microbiology and Polar filament.
His scientific interests lie mostly in Immunology, Trypanosoma cruzi, Chagas disease, Internal medicine and Pathology. Herbert B. Tanowitz interconnects Adipose tissue and Disease in the investigation of issues within Immunology. The concepts of his Trypanosoma cruzi study are interwoven with issues in Myocarditis, Virology, Molecular biology and Myocyte, Cell biology.
His Molecular biology research is multidisciplinary, incorporating perspectives in Biochemistry, Kinase and MAPK/ERK pathway. The Chagas disease study combines topics in areas such as Pathogenesis, Cardiomyopathy, Microbiology, Fibrosis and Heart disease. His Internal medicine research includes elements of Endocrinology and Cardiology.
Herbert B. Tanowitz mostly deals with Immunology, Chagas disease, Trypanosoma cruzi, Disease and Pathogenesis. His study in Immunology concentrates on Immune system, Parasitemia, Inflammation, Plasmodium berghei and Cytokine. Chagas disease is a subfield of Pathology that he studies.
His studies in Pathology integrate themes in fields like Spleen and Intensive care medicine. His studies deal with areas such as Myocarditis, Toxoplasma gondii, Receptor, Cell biology and Adipose tissue as well as Trypanosoma cruzi. His Disease study incorporates themes from Diabetes mellitus, Chronic infection and Organ transplantation.
Herbert B. Tanowitz spends much of his time researching Immunology, Chagas disease, Trypanosoma cruzi, Disease and Pathogenesis. Herbert B. Tanowitz has researched Immunology in several fields, including Endothelin receptor and Endothelin 1. His research in Chagas disease intersects with topics in Benznidazole, Heart disease, Heart failure and Nifurtimox.
His Trypanosoma cruzi research integrates issues from Adipose tissue, Oxidative stress and Immunity. He has included themes like Cerebral Malaria and Cerebral damage in his Disease study. His Inflammation study deals with Cytokine intersecting with Tumor necrosis factor alpha.
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Antibiotics that target mitochondria effectively eradicate cancer stem cells, across multiple tumor types : treating cancer like an infectious disease
Rebecca Lamb;Bela Ozsvari;Camilla L. Lisanti;Herbert B. Tanowitz.
Caveolin-3 knock-out mice develop a progressive cardiomyopathy and show hyperactivation of the p42/44 MAPK cascade
Scott Eric Woodman;David S. Park;Alex W. Cohen;Michelle W C Cheung.
Journal of Biological Chemistry (2002)
Caveolin-1 null mice develop cardiac hypertrophy with hyperactivation of p42/44 MAP kinase in cardiac fibroblasts
Alex W. Cohen;David S. Park;Scott E. Woodman;Terrence M. Williams.
American Journal of Physiology-cell Physiology (2003)
Transfusion-Associated Acute Chagas Disease Acquired in the United States
Irene H. Grant;Jonathan W.M. Gold;Murray Wittner;Herbert B. Tanowitz.
Annals of Internal Medicine (1989)
Stromal-epithelial metabolic coupling in cancer: integrating autophagy and metabolism in the tumor microenvironment.
Ubaldo E. Martinez-Outschoorn;Stephanos Pavlides;Anthony Howell;Richard G. Pestell.
The International Journal of Biochemistry & Cell Biology (2011)
Caveolin-1/3 double-knockout mice are viable, but lack both muscle and non-muscle caveolae, and develop a severe cardiomyopathic phenotype.
David S. Park;Scott E. Woodman;William Schubert;Alex W. Cohen.
American Journal of Pathology (2002)
Current understanding of immunity to Trypanosoma cruzi infection and pathogenesis of Chagas disease.
Fabiana S. Machado;Walderez O. Dutra;Walderez O. Dutra;Lisia Esper;Kenneth J. Gollob;Kenneth J. Gollob.
Seminars in Immunopathology (2012)
HIF1-alpha functions as a tumor promoter in cancer associated fibroblasts, and as a tumor suppressor in breast cancer cells: Autophagy drives compartment-specific oncogenesis.
Barbara Chiavarina;Diana Whitaker-Menezes;Gemma Migneco;Ubaldo E. Martinez-Outschoorn.
Cell Cycle (2010)
Trypanosoma cruzi: role of the immune response in the natural resistance of inbred strains of mice.
Thomas Trischmann;Herbert Tanowitz;Murray Wittner;Barry Bloom.
Experimental Parasitology (1978)
A Cell Culture System for Study of the Development of Toxoplasma gondii Bradyzoites
Louis M. Weiss;Denise Laplace;Peter M. Takvorian;Herbert B. Tanowitz.
Journal of Eukaryotic Microbiology (1995)
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