D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 66 Citations 11,272 170 World Ranking 1776 National Ranking 859

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Neuron
  • DNA

His primary scientific interests are in Alzheimer's disease, Neuroscience, Cell biology, Endocrinology and Internal medicine. His Alzheimer's disease research integrates issues from Neuropathology, Hippocampus and Neurodegeneration. His research investigates the connection between Neuroscience and topics such as Neurotrophin that intersect with problems in Tropomyosin receptor kinase B.

His research integrates issues of Messenger RNA, Cathepsin D, Biochemistry and Transcription factor in his study of Cell biology. Stephen D. Ginsberg usually deals with Endocrinology and limits it to topics linked to Downregulation and upregulation and Amyloid precursor protein secretase, Genetically modified mouse and Streptozotocin. His Basal forebrain study integrates concerns from other disciplines, such as Human brain and Cholinergic neuron.

His most cited work include:

  • Cholinergic system during the progression of Alzheimer's disease: therapeutic implications. (362 citations)
  • Expression profile of transcripts in Alzheimer's disease tangle-bearing CA1 neurons. (325 citations)
  • Human cholinergic basal forebrain: chemoanatomy and neurologic dysfunction (234 citations)

What are the main themes of his work throughout his whole career to date?

Neuroscience, Internal medicine, Endocrinology, Alzheimer's disease and Basal forebrain are his primary areas of study. His biological study spans a wide range of topics, including Neurotrophin, Neurodegeneration and Gene expression profiling. His Neurodegeneration study combines topics from a wide range of disciplines, such as Neuropathology and Endosome.

His work carried out in the field of Alzheimer's disease brings together such families of science as Downregulation and upregulation and Immunology. His research in Downregulation and upregulation tackles topics such as Genetically modified mouse which are related to areas like Tauopathy. His study looks at the relationship between Basal forebrain and fields such as Cholinergic neuron, as well as how they intersect with chemical problems.

He most often published in these fields:

  • Neuroscience (60.10%)
  • Internal medicine (36.54%)
  • Endocrinology (36.54%)

What were the highlights of his more recent work (between 2015-2021)?

  • Neuroscience (60.10%)
  • Cell biology (18.27%)
  • Basal forebrain (29.81%)

In recent papers he was focusing on the following fields of study:

Stephen D. Ginsberg focuses on Neuroscience, Cell biology, Basal forebrain, Disease and Downregulation and upregulation. His Nucleus basalis study in the realm of Neuroscience interacts with subjects such as Population. His research in Cell biology intersects with topics in Microvesicles, Transcription factor, Regulation of gene expression, Genetically modified mouse and Tauopathy.

As a part of the same scientific study, Stephen D. Ginsberg usually deals with the Basal forebrain, concentrating on Cholinergic neuron and frequently concerns with Down syndrome, Choline acetyltransferase, CAMK2A, DYRK1A and Forebrain. His studies examine the connections between Neurodegeneration and genetics, as well as such issues in Cholinergic, with regards to Amyloid beta, Cerebral cortex and Gene expression profiling. The study incorporates disciplines such as Internal medicine and Endocrinology in addition to Neurodevelopmental disorder.

Between 2015 and 2021, his most popular works were:

  • Autophagy flux in CA1 neurons of Alzheimer hippocampus: Increased induction overburdens failing lysosomes to propel neuritic dystrophy. (132 citations)
  • Locus coeruleus cellular and molecular pathology during the progression of Alzheimer’s disease (98 citations)
  • Locus coeruleus cellular and molecular pathology during the progression of Alzheimer’s disease (98 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Neuron
  • DNA

His primary areas of investigation include Neuroscience, Neurodegeneration, Cell biology, Downregulation and upregulation and Alzheimer's disease. Neuroscience is frequently linked to Amyloid beta in his study. The various areas that Stephen D. Ginsberg examines in his Neurodegeneration study include Mutation, Genetics and PPT1.

He has researched Cell biology in several fields, including Exosome, Messenger RNA, Brain-derived neurotrophic factor, Genetically modified mouse and Tauopathy. His study in Downregulation and upregulation is interdisciplinary in nature, drawing from both Cathepsin D, Neurotrophic factors, TFEB, Neurotrophin and Knockout mouse. His work on Senile plaques as part of general Alzheimer's disease research is frequently linked to Population, thereby connecting diverse disciplines of science.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Cholinergic system during the progression of Alzheimer's disease: therapeutic implications.

Elliott J Mufson;Scott E Counts;Sylvia E Perez;Stephen D Ginsberg.
Expert Review of Neurotherapeutics (2008)

550 Citations

Expression profile of transcripts in Alzheimer's disease tangle-bearing CA1 neurons.

Stephen D. Ginsberg;Scott E. Hemby;Virginia M.‐Y. Lee;James H. Eberwine.
Annals of Neurology (2000)

429 Citations

Human cholinergic basal forebrain: chemoanatomy and neurologic dysfunction

Elliott J. Mufson;Stephen D. Ginsberg;Milos D. Ikonomovic;Steven T. DeKosky.
Journal of Chemical Neuroanatomy (2003)

333 Citations

Alzheimer’s-related endosome dysfunction in Down syndrome is Aβ-independent but requires APP and is reversed by BACE-1 inhibition

Ying Jiang;Kerry A. Mullaney;Corrinne M. Peterhoff;Shaoli Che.
Proceedings of the National Academy of Sciences of the United States of America (2010)

277 Citations

Down regulation of trk but not p75NTR gene expression in single cholinergic basal forebrain neurons mark the progression of Alzheimer's disease

Stephen D. Ginsberg;Shaoli Che;Joanne Wuu;Scott E. Counts.
Journal of Neurochemistry (2006)

253 Citations

Microarray Analysis of Hippocampal CA1 Neurons Implicates Early Endosomal Dysfunction During Alzheimer's Disease Progression

Stephen D. Ginsberg;Stephen D. Ginsberg;Melissa J. Alldred;Melissa J. Alldred;Scott E. Counts;Anne M. Cataldo.
Biological Psychiatry (2010)

248 Citations

Gene Expression Profile for Schizophrenia: Discrete Neuron Transcription Patterns in the Entorhinal Cortex

Scott E. Hemby;Stephen D. Ginsberg;Brian Brunk;Steven E. Arnold.
Archives of General Psychiatry (2002)

246 Citations

Mild cognitive impairment: pathology and mechanisms

Elliott J. Mufson;Lester Binder;Scott E. Counts;Steven T. DeKosky.
Acta Neuropathologica (2012)

241 Citations

Decreased brain-derived neurotrophic factor depends on amyloid aggregation state in transgenic mouse models of Alzheimer's disease

Shiyong Peng;Diego J. Garzon;Diego J. Garzon;Monica Marchese;William Klein.
The Journal of Neuroscience (2009)

224 Citations

Autophagy flux in CA1 neurons of Alzheimer hippocampus: Increased induction overburdens failing lysosomes to propel neuritic dystrophy.

Matteo Bordi;Martin J Berg;Panaiyur S Mohan;Corrinne M Peterhoff.
Autophagy (2016)

212 Citations

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