D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 43 Citations 6,547 91 World Ranking 4210 National Ranking 1881

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Neuron
  • Alzheimer's disease

His primary areas of investigation include Neuroscience, Alzheimer's disease, Cognitive decline, Cholinergic and Neurotrophin. Scott E. Counts studies Basal forebrain, a branch of Neuroscience. His Alzheimer's disease research is multidisciplinary, incorporating elements of Neuropathology and Dementia, Prodromal Stage.

His Cognitive decline research is multidisciplinary, incorporating perspectives in Progressive disease, Temporal cortex, Autopsy and Degenerative disease. The Cholinergic portion of his research involves studies in Endocrinology and Internal medicine. In his study, Gene knockdown, Neuroprotection, Neurotrophic factors and Brain-derived neurotrophic factor is strongly linked to Cell biology, which falls under the umbrella field of Neurotrophin.

His most cited work include:

  • Cholinergic system during the progression of Alzheimer's disease: therapeutic implications. (362 citations)
  • Down regulation of trk but not p75NTR gene expression in single cholinergic basal forebrain neurons mark the progression of Alzheimer's disease (193 citations)
  • Microarray Analysis of Hippocampal CA1 Neurons Implicates Early Endosomal Dysfunction During Alzheimer's Disease Progression (187 citations)

What are the main themes of his work throughout his whole career to date?

Scott E. Counts mainly focuses on Neuroscience, Alzheimer's disease, Cholinergic, Internal medicine and Basal forebrain. His research integrates issues of Gene expression, Downregulation and upregulation, Neurodegeneration, Disease and Synaptophysin in his study of Neuroscience. The various areas that he examines in his Alzheimer's disease study include Temporal cortex, Receptor, Cognitive decline, Hippocampus and Degenerative disease.

In his research, Tropomyosin receptor kinase B and Cell biology is intimately related to Neurotrophin, which falls under the overarching field of Cholinergic. While the research belongs to areas of Internal medicine, he spends his time largely on the problem of Endocrinology, intersecting his research to questions surrounding Nerve growth factor and Frontal lobe. His Basal forebrain research includes themes of Galanin and Neuron.

He most often published in these fields:

  • Neuroscience (55.56%)
  • Alzheimer's disease (51.85%)
  • Cholinergic (46.30%)

What were the highlights of his more recent work (between 2011-2016)?

  • Alzheimer's disease (51.85%)
  • Neuroscience (55.56%)
  • Amyloid (9.26%)

In recent papers he was focusing on the following fields of study:

Scott E. Counts focuses on Alzheimer's disease, Neuroscience, Amyloid, Hippocampus and Hippocampal formation. His Alzheimer's disease research incorporates elements of Nucleus basalis and Cognitive decline. Neuroplasticity and Gene expression is closely connected to Disease in his research, which is encompassed under the umbrella topic of Neuroscience.

His study on Amyloid also encompasses disciplines like

  • Immunology, which have a strong connection to Cholinergic,
  • Pharmacology that intertwine with fields like Toxicity, Genetically modified mouse and In vivo. His study in Hippocampus is interdisciplinary in nature, drawing from both Postsynaptic potential and Synaptophysin. To a larger extent, he studies Endocrinology with the aim of understanding Hippocampal formation.

Between 2011 and 2016, his most popular works were:

  • Mild cognitive impairment: pathology and mechanisms (139 citations)
  • Synaptic gene dysregulation within hippocampal CA1 pyramidal neurons in mild cognitive impairment (73 citations)
  • Hippocampal Drebrin Loss in Mild Cognitive Impairment (65 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Neuron
  • Internal medicine

Alzheimer's disease, Postsynaptic potential, Cognitive decline, Hippocampus and Neuroscience are his primary areas of study. His studies in Alzheimer's disease integrate themes in fields like Progressive disease and Regulation of gene expression. His Progressive disease research incorporates themes from Neuropathology, Autopsy and Dementia, Prodromal Stage.

His Prodromal Stage study improves the overall literature in Pathology. His Synaptophysin research extends to Postsynaptic potential, which is thematically connected. He integrates many fields in his works, including Memory impairment, Dendritic spine and Hippocampal formation.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Cholinergic system during the progression of Alzheimer's disease: therapeutic implications.

Elliott J Mufson;Scott E Counts;Sylvia E Perez;Stephen D Ginsberg.
Expert Review of Neurotherapeutics (2008)

550 Citations

Enhancing mitochondrial proteostasis reduces amyloid-β proteotoxicity

Vincenzo Sorrentino;Mario Romani;Laurent Mouchiroud;John S. Beck.
Nature (2017)

361 Citations

The role of nerve growth factor receptors in cholinergic basal forebrain degeneration in prodromal Alzheimer disease.

Scott E. Counts;Elliott J. Mufson.
Journal of Neuropathology and Experimental Neurology (2005)

265 Citations

Down regulation of trk but not p75NTR gene expression in single cholinergic basal forebrain neurons mark the progression of Alzheimer's disease

Stephen D. Ginsberg;Shaoli Che;Joanne Wuu;Scott E. Counts.
Journal of Neurochemistry (2006)

253 Citations

Microarray Analysis of Hippocampal CA1 Neurons Implicates Early Endosomal Dysfunction During Alzheimer's Disease Progression

Stephen D. Ginsberg;Stephen D. Ginsberg;Melissa J. Alldred;Melissa J. Alldred;Scott E. Counts;Anne M. Cataldo.
Biological Psychiatry (2010)

248 Citations

Mild cognitive impairment: pathology and mechanisms

Elliott J. Mufson;Lester Binder;Scott E. Counts;Steven T. DeKosky.
Acta Neuropathologica (2012)

241 Citations

Light and electron microscopic localization of presenilin-1 in primate brain.

James J. Lah;Craig J. Heilman;Norman R. Nash;Howard D. Rees.
The Journal of Neuroscience (1997)

221 Citations

Differential expression of synaptic proteins in the frontal and temporal cortex of elderly subjects with mild cognitive impairment.

Scott E. Counts;Muhammad Nadeem;Shivanand P. Lad;Joanne Wuu.
Journal of Neuropathology and Experimental Neurology (2006)

220 Citations

Reduction of cortical TrkA but not p75(NTR) protein in early-stage Alzheimer's disease.

Scott E. Counts;Muhammad Nadeem;Joanne Wuu;Stephen D. Ginsberg.
Annals of Neurology (2004)

209 Citations

Locus coeruleus cellular and molecular pathology during the progression of Alzheimer’s disease

Sarah C. Kelly;Bin He;Sylvia E. Perez;Stephen D. Ginsberg;Stephen D. Ginsberg.
Acta neuropathologica communications (2017)

182 Citations

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