What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Immune system
His primary areas of investigation include CD36, Scavenger receptor, Cell biology, Biochemistry and Endocrinology.
His CD36 study introduces a deeper knowledge of Receptor.
Roy L. Silverstein combines subjects such as Inflammation, Signal transduction and Monocyte with his study of Scavenger receptor.
His research integrates issues of Interleukin, Apoptosis and Peroxisome proliferator-activated receptor in his study of Cell biology.
His Biochemistry research includes themes of Angiogenesis and In vivo.
The study incorporates disciplines such as Endothelial stem cell, Anesthesia and Platelet, Internal medicine, Immunology in addition to Endocrinology.
His most cited work include:
- Immature dendritic cells phagocytose apoptotic cells via alphavbeta5 and CD36, and cross-present antigens to cytotoxic T lymphocytes (1150 citations)
- CD36: a class B scavenger receptor involved in angiogenesis, atherosclerosis, inflammation, and lipid metabolism (913 citations)
- Targeted disruption of the class B scavenger receptor CD36 protects against atherosclerotic lesion development in mice (869 citations)
What are the main themes of his work throughout his whole career to date?
Roy L. Silverstein mostly deals with CD36, Cell biology, Scavenger receptor, Internal medicine and Platelet.
His CD36 research incorporates elements of Molecular biology, Signal transduction and Foam cell.
His studies deal with areas such as Endothelial stem cell, Angiogenesis, Thrombospondin 1, Apoptosis and Integrin as well as Cell biology.
His work deals with themes such as Inflammation, Macrophage and Low-density lipoprotein, which intersect with Scavenger receptor.
His Internal medicine study combines topics in areas such as Gastroenterology and Endocrinology.
His Platelet research is multidisciplinary, incorporating elements of Thrombosis, Pharmacology and Fibrin.
He most often published in these fields:
- CD36 (64.24%)
- Cell biology (42.05%)
- Scavenger receptor (28.48%)
What were the highlights of his more recent work (between 2013-2021)?
- CD36 (64.24%)
- Platelet (25.83%)
- Signal transduction (25.50%)
In recent papers he was focusing on the following fields of study:
His primary scientific interests are in CD36, Platelet, Signal transduction, Cell biology and Platelet activation.
He has included themes like Reactive oxygen species, Endocrinology, Immunology and Scavenger receptor in his CD36 study.
His research in Scavenger receptor intersects with topics in Molecular biology and Foam cell.
The study incorporates disciplines such as Cancer research, Fibrin, Pharmacology, Thrombosis and Annexin A5 in addition to Platelet.
Roy L. Silverstein has researched Signal transduction in several fields, including Internal medicine, Angiogenesis, Arteriogenesis, Lysophosphatidic acid and FOXO1.
His research investigates the connection between Cell biology and topics such as Apoptosis that intersect with problems in Lung injury.
Between 2013 and 2021, his most popular works were:
- Gut Microbial Metabolite TMAO Enhances Platelet Hyperreactivity and Thrombosis Risk. (650 citations)
- Cancer stem cell-specific scavenger receptor CD36 drives glioblastoma progression. (97 citations)
- Platelet-derived S100 family member myeloid-related protein-14 regulates thrombosis (76 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Immune system
Roy L. Silverstein spends much of his time researching CD36, Cell biology, Platelet activation, Signal transduction and Platelet.
His study on CD36 is covered under Biochemistry.
His biological study spans a wide range of topics, including Cell migration, Apoptosis, Annexin, Annexin A5 and Tube formation.
As a member of one scientific family, Roy L. Silverstein mostly works in the field of Platelet activation, focusing on Ubiquitin and, on occasion, Molecular biology.
His study in Signal transduction is interdisciplinary in nature, drawing from both Lysophosphatidic acid, Lung injury, FOXO1 and Interleukin 10.
His research integrates issues of Tyrosine kinase, Phosphorylation, Tyrosine phosphorylation, Thrombosis and Pharmacology in his study of Platelet.
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