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D-Index & Metrics

Biology and Biochemistry

D-Index
71
Citations
18336
World Ranking
6637
National Ranking
3086

Research.com Recognitions

  • 1997 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

David P. Hajjar is affiliated with Cornell University in the United States. Their research spans several subfields of study, including Spectroscopy, Molecular Biology, Pharmacology, and Physiology.

Their research focuses on topics such as Molecular Sensors and Ion Detection, Advanced Biosensing and Bioanalysis Techniques, Pharmacogenetics and Drug Metabolism, and Nitric Oxide and Endothelin Effects.

Among recent publications, the following papers are notable:

  • A Molecular Redox Switch on p21 ras: STRUCTURAL BASIS FOR THE NITRIC OXIDE-p21ras INTERACTION (2021) published by UNC Libraries
  • Inducible Nitric Oxide Synthase Mediates Prostaglandin H2 Synthase Nitration and Suppresses Eicosanoid Production (2020) published by UNC Libraries

Frequent coauthors who have collaborated with David P. Hajjar include:

  • Harry M. Lander
  • Barbara L. Hempstead
  • Urooj A. Mirza
  • Brian T. Chait
  • Sharon L. Campbell

The UNC Libraries is a recurrent venue for their work, hosting at least two publications.

David P. Hajjar was recognized as a Fellow of the American Association for the Advancement of Science (AAAS) in 1997.

Best Publications

  • CD36: a class B scavenger receptor involved in angiogenesis, atherosclerosis, inflammation, and lipid metabolism

    Maria Febbraio;David P. Hajjar;Roy L. Silverstein

  • Targeted disruption of the class B scavenger receptor CD36 protects against atherosclerotic lesion development in mice

    Maria Febbraio;Eugene A. Podrez;Jonathan D. Smith;David P. Hajjar

  • A null mutation in murine CD36 reveals an important role in fatty acid and lipoprotein metabolism.

    Maria Febbraio;Nada A. Abumrad;David P. Hajjar;Kavita Sharma

  • A Molecular Redox Switch on p21ras STRUCTURAL BASIS FOR THE NITRIC OXIDE-p21ras INTERACTION

    Harry M. Lander;David P. Hajjar;Barbara L. Hempstead;Urooj A. Mirza

  • Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species

    Eugene A. Podrez;Maria Febbraio;Nader Sheibani;David Schmitt

  • A novel family of atherogenic oxidized phospholipids promotes macrophage foam cell formation via the scavenger receptor CD36 and is enriched in atherosclerotic lesions.

    Eugene A. Podrez;Eugenia Poliakov;Zhongzhou Shen;Renliang Zhang

  • Inflammation in atherosclerosis and implications for therapy

    Rodolfo Paoletti;Antonio M. Gotto;David P. Hajjar

  • Redox regulation of cell signalling.

    Harry M. Lander;Aaron J. Milbank;James M. Tauras;David P. Hajjar

  • Lipoprotein (a) regulates plasminogen activator inhibitor-1 expression in endothelial cells. A potential mechanism in thrombogenesis.

    O R Etingin;D P Hajjar;K A Hajjar;P C Harpel

  • Neurotrophin and Neurotrophin Receptors in Vascular Smooth Muscle Cells: Regulation of Expression in Response to Injury

    M. J. Donovan;R. C. Miranda;R. Kraemer;T. A. McCaffrey

  • Native and Modified Low Density Lipoproteins Increase the Functional Expression of the Macrophage Class B Scavenger Receptor, CD36 *

    Jihong Han;David P. Hajjar;Maria Febbraio;Andrew C. Nicholson

  • The Role of Phosphatidylinositol 3-Kinase in Vascular Endothelial Growth Factor Signaling

    Geeta D. Thakker;David P. Hajjar;William A. Muller;Todd K. Rosengart

  • Induction of CD36 expression by oxidized LDL and IL-4 by a common signaling pathway dependent on protein kinase C and PPAR-γ

    Jianwei Feng;Jihong Han;S. Freida A. Pearce;Roy L. Silverstein

  • Triglyceride, High-Density Lipoprotein, and Coronary Heart Disease

    E. Rapaport;D. W. Bilheimer;A. V. Chobanian;D. P. Hajjar

  • Infection and atherosclerosis: potential roles of pathogen burden and molecular mimicry.

    Stephen E. Epstein;Jianhui Zhu;Mary Susan Burnett;Yi Fu Zhou

  • Lipoprotein Trafficking in Vascular Cells: MOLECULAR TROJAN HORSES AND CELLULAR SABOTEURS *

    David P. Hajjar;Margaret E. Haberland

  • Tumor necrosis factor-mediated release of platelet-derived growth factor from cultured endothelial cells.

    K A Hajjar;D P Hajjar;R L Silverstein;R L Nachman

  • Transforming Growth Factor-β1 (TGF-β1) and TGF-β2 Decrease Expression of CD36, the Type B Scavenger Receptor, through Mitogen-activated Protein Kinase Phosphorylation of Peroxisome Proliferator-activated Receptor-γ

    Jihong Han;David P. Hajjar;James M. Tauras;Jianwei Feng

  • Prostacyclin modulates cholesteryl ester hydrolytic activity by its effect on cyclic adenosine monophosphate in rabbit aortic smooth muscle cells.

    D P Hajjar;B B Weksler;D J Falcone;J M Hefton

  • Recovery of prostacyclin production by de-endothelialized rabbit aorta. Critical role of neointimal smooth muscle cells.

    A Eldor;D J Falcone;D P Hajjar;C R Minick

Frequent Co-Authors

Antonio M. Gotto
Antonio M. Gotto Cornell University
Roy L. Silverstein
Roy L. Silverstein Medical College of Wisconsin
Maria Febbraio
Maria Febbraio Cleveland Clinic
Steven S. Gross
Steven S. Gross Cornell University
Katherine A. Hajjar
Katherine A. Hajjar Cornell University
Babette B. Weksler
Babette B. Weksler Cornell University
Stanley L. Hazen
Stanley L. Hazen Cleveland Clinic Lerner College of Medicine
Hao Shen
Hao Shen University of Pennsylvania
Nobuyo Maeda
Nobuyo Maeda University of North Carolina at Chapel Hill
Brian T. Chait
Brian T. Chait Rockefeller University

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