His primary areas of study are Receptor, Cell biology, Molecular biology, Lymphokine-activated killer cell and Natural killer cell. His studies deal with areas such as Natural Cytotoxicity Triggering Receptor 1 and CD94/NKG2 as well as Receptor. His research integrates issues of Interleukin 12 and Cytotoxicity in his study of Cell biology.
He combines subjects such as Complementary DNA, Antibody, Signal transduction and Phosphorylation with his study of Molecular biology. His Lymphokine-activated killer cell research includes elements of NK-92 and Natural killer T cell. His study in Natural killer cell is interdisciplinary in nature, drawing from both Human leukocyte antigen, Histocompatibility Antigens Class I, Cytolysis, Bone marrow transplantation and Major histocompatibility complex.
His primary areas of investigation include Receptor, Molecular biology, Immunology, Cell biology and Human leukocyte antigen. His Receptor research is multidisciplinary, incorporating elements of Natural killer cell, Cytolysis, Cytotoxicity and Major histocompatibility complex. His research in Molecular biology intersects with topics in Cell culture, CD3, CD8, Immunoglobulin superfamily and Complementary DNA.
His study on Immunology is mostly dedicated to connecting different topics, such as NKG2D. His study in Cell biology is interdisciplinary in nature, drawing from both Interleukin 12, Natural Cytotoxicity Triggering Receptor 1, Lymphokine-activated killer cell and MHC class I. His work investigates the relationship between Human leukocyte antigen and topics such as Virology that intersect with problems in Antibody.
His main research concerns Immunology, Receptor, Virology, Human leukocyte antigen and Cell biology. The various areas that Roberto Biassoni examines in his Immunology study include Nephrotic syndrome and Calcineurin. His research integrates issues of Gene expression, Macaque, Natural killer T cell, CD8 and NKG2D in his study of Receptor.
His HLA-B study in the realm of Human leukocyte antigen interacts with subjects such as Subtyping. His Cell biology research integrates issues from Cytotoxic T cell, Cytolysis, Innate immune system and Major histocompatibility complex. Roberto Biassoni focuses mostly in the field of Genetics, narrowing it down to matters related to Molecular biology and, in some cases, Cell membrane.
His primary scientific interests are in Receptor, Immunology, Cell biology, CD8 and Interleukin 21. His work on Cell activation as part of general Receptor study is frequently connected to Homo sapiens, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them. His work on Humanized antibody, Ofatumumab and Rituximab as part of his general Immunology study is frequently connected to Lung injury, thereby bridging the divide between different branches of science.
Roberto Biassoni combines subjects such as Cell type, Major histocompatibility complex, Cytolysis and Ligand with his study of Cell biology. His CD8 study combines topics in areas such as Cell, In vitro and Cytokine. Roberto Biassoni usually deals with Interleukin 21 and limits it to topics linked to NKG2D and Cancer research.
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Activating Receptors and Coreceptors Involved in Human Natural Killer Cell-Mediated Cytolysis
Alessandro Moretta;Cristina Bottino;Massimo Vitale;Daniela Pende.
Annual Review of Immunology (2001)
RECEPTORS FOR HLA CLASS-I MOLECULES IN HUMAN NATURAL KILLER CELLS
Alessandro Moretta;Cristina Bottino;Massimo Vitale;Daniela Pende.
Annual Review of Immunology (1996)
Identification and Molecular Characterization of Nkp30, a Novel Triggering Receptor Involved in Natural Cytotoxicity Mediated by Human Natural Killer Cells
Daniela Pende;Silvia Parolini;Anna Pessino;Simona Sivori.
Journal of Experimental Medicine (1999)
Major Histocompatibility Complex Class I-related Chain A and UL16-Binding Protein Expression on Tumor Cell Lines of Different Histotypes Analysis of Tumor Susceptibility to NKG2D-dependent Natural Killer Cell Cytotoxicity
Daniela Pende;Paola Rivera;Stefania Marcenaro;Chien Chung Chang.
Cancer Research (2002)
Molecular clones of the p58 NK cell receptor reveal immunoglobulin-related molecules with diversity in both the extra- and intracellular domains
Nicolai Wagtmann;Roberto Biassoni;Claudia Cantoni;Simonetta Verdiani.
Immunity (1995)
X-Linked Lymphoproliferative Disease 2b4 Molecules Displaying Inhibitory Rather than Activating Function Are Responsible for the Inability of Natural Killer Cells to Kill Epstein-Barr Virus–Infected Cells
Silvia Parolini;Cristina Bottino;Michela Falco;Raffaella Augugliaro.
Journal of Experimental Medicine (2000)
Molecular cloning of NKp46: a novel member of the immunoglobulin superfamily involved in triggering of natural cytotoxicity.
Anna Pessino;Simona Sivori;Cristina Bottino;Angela Malaspina.
Journal of Experimental Medicine (1998)
Transforming growth factor β1 inhibits expression of NKp30 and NKG2D receptors: Consequences for the NK-mediated killing of dendritic cells
Roberta Castriconi;Claudia Cantoni;Mariella Della Chiesa;Massimo Vitale.
Proceedings of the National Academy of Sciences of the United States of America (2003)
The natural killer cell receptor specific for HLA-A allotypes: a novel member of the p58/p70 family of inhibitory receptors that is characterized by three immunoglobulin-like domains and is expressed as a 140-kD disulphide-linked dimer.
D Pende;R Biassoni;C Cantoni;S Verdiani.
Journal of Experimental Medicine (1996)
NKp46 is the major triggering receptor involved in the natural cytotoxicity of fresh or cultured human NK cells. Correlation between surface density of NKp46 and natural cytotoxicity against autologous, allogeneic or xenogeneic target cells
Simona Sivori;Daniela Pende;Cristina Bottino;Emanuela Marcenaro.
European Journal of Immunology (1999)
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