What is he best known for?
The fields of study he is best known for:
- Immune system
- Gene
- Antibody
His primary areas of study are Immunology, T cell, Molecular biology, Cytotoxic T cell and T-cell receptor.
His Immunology study often links to related topics such as Cancer research.
His study looks at the relationship between T cell and topics such as Cell biology, which overlap with Nkg2d receptor, Cytotoxicity, NK Cell Lectin-Like Receptor Subfamily K, Ligand and Tumor immunosurveillance.
His work carried out in the field of Molecular biology brings together such families of science as Interleukin 2, Dendritic cell, Mononuclear phagocyte system and Cell growth.
Robert E. Tigelaar interconnects CD8 and Major histocompatibility complex in the investigation of issues within Cytotoxic T cell.
His Antigen-presenting cell research incorporates themes from Natural killer T cell and Antigen.
His most cited work include:
- Regulation of Cutaneous Malignancy by γδ T Cells (799 citations)
- Limited diversity of γδ antigen receptor genes of thy-1+ dendritic epidermal cells (446 citations)
- Immunoregulation in the tissues by |[gamma]||[delta]| T cells (388 citations)
What are the main themes of his work throughout his whole career to date?
Robert E. Tigelaar mostly deals with Immunology, Molecular biology, Antigen, T cell and T-cell receptor.
His work in Immunology covers topics such as Transplantation which are related to areas like C57BL/6.
His biological study spans a wide range of topics, including Langerhans cell, Dendritic cell, Epidermis and Ratón.
His Antigen research incorporates themes from Immune system, Immunity, Cell biology, Antibody and Antigen presentation.
His T cell research is multidisciplinary, incorporating perspectives in Cytotoxic T cell, Cancer research, CD8 and Lymphocyte.
His research investigates the connection between Antigen-presenting cell and topics such as Natural killer T cell that intersect with problems in Interleukin 12.
He most often published in these fields:
- Immunology (49.65%)
- Molecular biology (36.17%)
- Antigen (29.08%)
What were the highlights of his more recent work (between 2008-2020)?
- Immunology (49.65%)
- Antigen (29.08%)
- Cell biology (12.77%)
In recent papers he was focusing on the following fields of study:
Robert E. Tigelaar mainly investigates Immunology, Antigen, Cell biology, Cancer research and Monocyte.
In most of his Immunology studies, his work intersects topics such as Disease.
His study in Antigen is interdisciplinary in nature, drawing from both T cell, Immune system and Sensitization.
In his work, Lymphoma, Cutaneous malignancy and T-cell receptor is strongly intertwined with Receptor, which is a subfield of T cell.
His Function study in the realm of Cell biology interacts with subjects such as Repertoire.
His studies deal with areas such as Carcinogenesis and DMBA as well as Cancer research.
Between 2008 and 2020, his most popular works were:
- Skint-1 is a highly specific, unique selecting component for epidermal T cells (110 citations)
- Langerhans Cells Facilitate Epithelial DNA Damage and Squamous Cell Carcinoma (97 citations)
- Rapid generation of maturationally synchronized human dendritic cells: contribution to the clinical efficacy of extracorporeal photochemotherapy. (68 citations)
In his most recent research, the most cited papers focused on:
- Immune system
- Gene
- Antibody
His primary areas of investigation include Immunology, Antigen-presenting cell, Dendritic cell, Cell biology and Antigen presentation.
His research in Photopheresis, MART-1 Antigen and Immune system are components of Immunology.
Robert E. Tigelaar usually deals with Antigen-presenting cell and limits it to topics linked to Cellular differentiation and CD80.
His research in Dendritic cell focuses on subjects like Monocyte, which are connected to Cutaneous T-cell lymphoma, Immunophenotyping and Transcriptome.
His Cell biology research includes elements of Major histocompatibility complex and Compartment.
Robert E. Tigelaar interconnects Priming and Antigen in the investigation of issues within Antigen presentation.
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