2010 - Fellow of the American Association for the Advancement of Science (AAAS)
His main research concerns Genetics, Internal medicine, Genetic association, Single-nucleotide polymorphism and SNP. Qing Kenneth Wang combines subjects such as Molecular biology, Case-control study and Nav1.5 with his study of Genetics. His Internal medicine study incorporates themes from Endocrinology and Cardiology.
Qing Kenneth Wang works mostly in the field of Genetic association, limiting it down to topics relating to Allele and, in certain cases, Genome-wide association study, as a part of the same area of interest. His Single-nucleotide polymorphism research is multidisciplinary, incorporating elements of Genotyping and Locus. His Sudden death study combines topics from a wide range of disciplines, such as Long QT syndrome, KvLQT1, KCNE2, Cardiac arrhythmia and HEK 293 cells.
Qing Kenneth Wang focuses on Genetics, Internal medicine, Single-nucleotide polymorphism, Gene and Molecular biology. His study in Genetic linkage, Locus, Genome-wide association study, Genetic association and Mutation is carried out as part of his studies in Genetics. His research investigates the connection between Genetic association and topics such as Allele that intersect with issues in Genotype.
His work deals with themes such as Endocrinology and Cardiology, which intersect with Internal medicine. In his research, Linkage disequilibrium is intimately related to Case-control study, which falls under the overarching field of Single-nucleotide polymorphism. Qing Kenneth Wang interconnects Long QT syndrome, Nav1.5 and Heart disease in the investigation of issues within Sudden death.
His scientific interests lie mostly in Cell biology, Internal medicine, Genetics, Genetic association and Gene knockdown. His research in Cell biology intersects with topics in HEK 293 cells, Immunology, Zebrafish and Nav1.5. His study looks at the intersection of Nav1.5 and topics like Internalization with Mutation.
His Internal medicine research incorporates themes from Endocrinology and Cardiology. His Genetic association research is multidisciplinary, incorporating perspectives in Coronary artery disease, Coagulation, Gene expression and Allele. His studies deal with areas such as Single-nucleotide polymorphism and Genotype as well as Allele.
His primary areas of study are Immunology, Cell biology, Coronary artery disease, Allele and Cancer research. Qing Kenneth Wang has included themes like Endothelial stem cell, Protein kinase B, Gene, Cell adhesion and PI3K/AKT/mTOR pathway in his Immunology study. His work carried out in the field of Cell biology brings together such families of science as Regulator, Gene knockdown, Pathogenesis and Gene regulatory network.
His Coronary artery disease research integrates issues from Polymorphism, Minor allele frequency, Single-nucleotide polymorphism, Genetic association and Interleukin. His Allele study deals with the bigger picture of Genetics. His research in the fields of Angiogenesis overlaps with other disciplines such as Androgen.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
SCN5A mutations associated with an inherited cardiac arrhythmia, long QT syndrome
Qing Kenneth Wang;Qing Kenneth Wang;Jiaxiang Shen;Jiaxiang Shen;Igor Splawski;Donald Atkinson;Donald Atkinson.
Calcium-sensitive potassium channelopathy in human epilepsy and paroxysmal movement disorder
Wei Du;Jocelyn F. Bautista;Jocelyn F. Bautista;Huanghe Yang;Ana Diez-Sampedro.
Nature Genetics (2005)
Mutations in a human ROBO gene disrupt hindbrain axon pathway crossing and morphogenesis
Joanna C. Jen;Wai Man Chan;Thomas M. Bosley;Jijun Wan.
Mutations in SLC20A2 link familial idiopathic basal ganglia calcification with phosphate homeostasis.
Cheng Wang;Yulei Li;Lei Shi;Jie Ren.
Nature Genetics (2012)
Human intracellular ISG15 prevents interferon-α/β over-amplification and auto-inflammation
Xianqin Zhang;Dusan Bogunovic;Béatrice Payelle-Brogard;Véronique Francois-Newton.
Mutation in Nuclear Pore Component NUP155 Leads to Atrial Fibrillation and Early Sudden Cardiac Death
Xianqin Zhang;Xianqin Zhang;Shenghan Chen;Shin Yoo;Susmita Chakrabarti.
Genome-wide association identifies a susceptibility locus for coronary artery disease in the Chinese Han population
Fan Wang;Cheng Qi Xu;Qing He;Jian Ping Cai.
Nature Genetics (2011)
MicroRNA-503 targets FGF2 and VEGFA and inhibits tumor angiogenesis and growth.
Bisheng Zhou;Ruihua Ma;Wenxia Si;Sisi Li.
Cancer Letters (2013)
Four SNPs on Chromosome 9p21 in a South Korean Population Implicate a Genetic Locus That Confers High Cross-Race Risk for Development of Coronary Artery Disease
Gong Qing Shen;Lin Li;Shaoqi Rao;Kalil G. Abdullah.
Arteriosclerosis, Thrombosis, and Vascular Biology (2007)
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