D-Index & Metrics Best Publications
Patrik Verstreken

Patrik Verstreken

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 59 Citations 21,300 156 World Ranking 8171 National Ranking 144

Research.com Recognitions

Awards & Achievements

Member of the European Molecular Biology Organization (EMBO)

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Genetics

His primary areas of investigation include Cell biology, Synaptic vesicle, Mitochondrion, Endocytosis and Neuromuscular junction. His studies in Cell biology integrate themes in fields like Mutation, Synaptic vesicle endocytosis, Biochemistry and Mutant. His Synaptic vesicle study introduces a deeper knowledge of Vesicle.

Patrik Verstreken interconnects PINK1, Synapse, Neuroscience and Drosophila Protein in the investigation of issues within Mitochondrion. Patrik Verstreken works mostly in the field of Synapse, limiting it down to topics relating to Neuroprotection and, in certain cases, Neurodegeneration, as a part of the same area of interest. His study focuses on the intersection of Endocytosis and fields such as Cytoskeleton with connections in the field of Myosin.

His most cited work include:

  • Synaptic Mitochondria Are Critical for Mobilization of Reserve Pool Vesicles at Drosophila Neuromuscular Junctions (627 citations)
  • Synaptic Mitochondria Are Critical for Mobilization of Reserve Pool Vesicles at Drosophila Neuromuscular Junctions (627 citations)
  • Drosophila parkin mutants have decreased mass and cell size and increased sensitivity to oxygen radical stress. (360 citations)

What are the main themes of his work throughout his whole career to date?

Patrik Verstreken spends much of his time researching Cell biology, Neuroscience, Synaptic vesicle, Neurodegeneration and Mitochondrion. His Cell biology study incorporates themes from Parkin, Mutant, Endocytic cycle, Endocytosis and Synapse. As part of one scientific family, he deals mainly with the area of Neuroscience, narrowing it down to issues related to the Disease, and often Induced pluripotent stem cell.

Patrik Verstreken has researched Synaptic vesicle in several fields, including Exocytosis and Neuromuscular junction. His Neurodegeneration research includes themes of Mutation, Endocrinology and Protein degradation. His Mitochondrion study also includes

  • PINK1 which connect with Biochemistry and Electron transport chain,
  • Drosophila Protein which intersects with area such as Cytoskeleton.

He most often published in these fields:

  • Cell biology (66.48%)
  • Neuroscience (26.26%)
  • Synaptic vesicle (26.82%)

What were the highlights of his more recent work (between 2018-2021)?

  • Cell biology (66.48%)
  • Biophysics (11.73%)
  • Neurodegeneration (27.37%)

In recent papers he was focusing on the following fields of study:

Patrik Verstreken mostly deals with Cell biology, Biophysics, Neurodegeneration, Dystonia and Lipid metabolism. His work in the fields of Cell biology, such as Plasma protein binding, intersects with other areas such as Mechanism. In his work, Exocytosis is strongly intertwined with Vesicle, which is a subfield of Biophysics.

His studies in Neurodegeneration integrate themes in fields like Neuroinflammation, Downregulation and upregulation, Mutant and Allele. His Dystonia research is multidisciplinary, incorporating elements of Drosophila melanogaster and Missense mutation. His Lipid metabolism study integrates concerns from other disciplines, such as Mutation, Lipid droplet and Nuclear pore.

Between 2018 and 2021, his most popular works were:

  • Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition) (38 citations)
  • TBC1D24-TLDc-related epilepsy exercise-induced dystonia: rescue by antioxidants in a disease model (17 citations)
  • TBC1D24-TLDc-related epilepsy exercise-induced dystonia: rescue by antioxidants in a disease model (17 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Genetics

His main research concerns Dystonia, Lipid metabolism, Drosophila melanogaster, Endocrinology and Internal medicine. His Dystonia research includes elements of Compound heterozygosity and Epilepsy. The various areas that Patrik Verstreken examines in his Lipid metabolism study include Endoplasmic reticulum, Cell biology and Nuclear pore.

Drosophila melanogaster is the subject of his research, which falls under Genetics. He interconnects Mutation, Neurodegeneration, Disease and Induced pluripotent stem cell in the investigation of issues within Endocrinology. Many of his studies on Internal medicine involve topics that are commonly interrelated, such as Phosphatase.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)

8964 Citations

Synaptic mitochondria are critical for mobilization of reserve pool vesicles at Drosophila neuromuscular junctions.

Patrik Verstreken;Patrik Verstreken;Cindy V. Ly;Koen J.T. Venken;Tong Wey Koh.
Neuron (2005)

818 Citations

Drosophila parkin mutants have decreased mass and cell size and increased sensitivity to oxygen radical stress.

Yakov Pesah;Tuan Pham;Heather Burgess;Brooke Middlebrooks.
Development (2004)

521 Citations

A Single-Cell Transcriptome Atlas of the Aging Drosophila Brain

Kristofer Davie;Jasper Janssens;Duygu Koldere;Maxime De Waegeneer.
Cell (2018)

423 Citations

Synaptojanin Is Recruited by Endophilin to Promote Synaptic Vesicle Uncoating

Patrik Verstreken;Tong Wey Koh;Karen L. Schulze;R. Grace Zhai.
Neuron (2003)

399 Citations

Parkinson's disease mutations in PINK1 result in decreased Complex I activity and deficient synaptic function

Vanessa Morais;Patrik Verstreken;Anne Roethig;Joel Smet.
Embo Molecular Medicine (2009)

390 Citations

Endophilin Mutations Block Clathrin-Mediated Endocytosis but Not Neurotransmitter Release

Patrik Verstreken;Ole Kjaerulff;Thomas E. Lloyd;Richard Atkinson.
Cell (2002)

361 Citations

Shar-pei mediates cell proliferation arrest during imaginal disc growth in Drosophila.

Madhuri Kango-Singh;Riitta Nolo;Chunyao Tao;Patrik Verstreken.
Development (2002)

361 Citations

LRRK2 Controls an EndoA Phosphorylation Cycle in Synaptic Endocytosis

Samer Matta;Kristof Van Kolen;Raquel da Cunha;Geert van den Bogaart;Geert van den Bogaart.
Neuron (2012)

358 Citations

The v-ATPase V0 Subunit a1 Is Required for a Late Step in Synaptic Vesicle Exocytosis in Drosophila

P. Robin Hiesinger;Amir Fayyazuddin;Sunil Q. Mehta;Tanja Rosenmund.
Cell (2005)

326 Citations

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