Member of the European Molecular Biology Organization (EMBO)
His primary areas of investigation include Cell biology, Synaptic vesicle, Mitochondrion, Endocytosis and Neuromuscular junction. His studies in Cell biology integrate themes in fields like Mutation, Synaptic vesicle endocytosis, Biochemistry and Mutant. His Synaptic vesicle study introduces a deeper knowledge of Vesicle.
Patrik Verstreken interconnects PINK1, Synapse, Neuroscience and Drosophila Protein in the investigation of issues within Mitochondrion. Patrik Verstreken works mostly in the field of Synapse, limiting it down to topics relating to Neuroprotection and, in certain cases, Neurodegeneration, as a part of the same area of interest. His study focuses on the intersection of Endocytosis and fields such as Cytoskeleton with connections in the field of Myosin.
Patrik Verstreken spends much of his time researching Cell biology, Neuroscience, Synaptic vesicle, Neurodegeneration and Mitochondrion. His Cell biology study incorporates themes from Parkin, Mutant, Endocytic cycle, Endocytosis and Synapse. As part of one scientific family, he deals mainly with the area of Neuroscience, narrowing it down to issues related to the Disease, and often Induced pluripotent stem cell.
Patrik Verstreken has researched Synaptic vesicle in several fields, including Exocytosis and Neuromuscular junction. His Neurodegeneration research includes themes of Mutation, Endocrinology and Protein degradation. His Mitochondrion study also includes
Patrik Verstreken mostly deals with Cell biology, Biophysics, Neurodegeneration, Dystonia and Lipid metabolism. His work in the fields of Cell biology, such as Plasma protein binding, intersects with other areas such as Mechanism. In his work, Exocytosis is strongly intertwined with Vesicle, which is a subfield of Biophysics.
His studies in Neurodegeneration integrate themes in fields like Neuroinflammation, Downregulation and upregulation, Mutant and Allele. His Dystonia research is multidisciplinary, incorporating elements of Drosophila melanogaster and Missense mutation. His Lipid metabolism study integrates concerns from other disciplines, such as Mutation, Lipid droplet and Nuclear pore.
His main research concerns Dystonia, Lipid metabolism, Drosophila melanogaster, Endocrinology and Internal medicine. His Dystonia research includes elements of Compound heterozygosity and Epilepsy. The various areas that Patrik Verstreken examines in his Lipid metabolism study include Endoplasmic reticulum, Cell biology and Nuclear pore.
Drosophila melanogaster is the subject of his research, which falls under Genetics. He interconnects Mutation, Neurodegeneration, Disease and Induced pluripotent stem cell in the investigation of issues within Endocrinology. Many of his studies on Internal medicine involve topics that are commonly interrelated, such as Phosphatase.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Synaptic mitochondria are critical for mobilization of reserve pool vesicles at Drosophila neuromuscular junctions.
Patrik Verstreken;Patrik Verstreken;Cindy V. Ly;Koen J.T. Venken;Tong Wey Koh.
Drosophila parkin mutants have decreased mass and cell size and increased sensitivity to oxygen radical stress.
Yakov Pesah;Tuan Pham;Heather Burgess;Brooke Middlebrooks.
A Single-Cell Transcriptome Atlas of the Aging Drosophila Brain
Kristofer Davie;Jasper Janssens;Duygu Koldere;Maxime De Waegeneer.
Synaptojanin Is Recruited by Endophilin to Promote Synaptic Vesicle Uncoating
Patrik Verstreken;Tong Wey Koh;Karen L. Schulze;R. Grace Zhai.
Parkinson's disease mutations in PINK1 result in decreased Complex I activity and deficient synaptic function
Vanessa Morais;Patrik Verstreken;Anne Roethig;Joel Smet.
Embo Molecular Medicine (2009)
Endophilin Mutations Block Clathrin-Mediated Endocytosis but Not Neurotransmitter Release
Patrik Verstreken;Ole Kjaerulff;Thomas E. Lloyd;Richard Atkinson.
Shar-pei mediates cell proliferation arrest during imaginal disc growth in Drosophila.
Madhuri Kango-Singh;Riitta Nolo;Chunyao Tao;Patrik Verstreken.
LRRK2 Controls an EndoA Phosphorylation Cycle in Synaptic Endocytosis
Samer Matta;Kristof Van Kolen;Raquel da Cunha;Geert van den Bogaart;Geert van den Bogaart.
The v-ATPase V0 Subunit a1 Is Required for a Late Step in Synaptic Vesicle Exocytosis in Drosophila
P. Robin Hiesinger;Amir Fayyazuddin;Sunil Q. Mehta;Tanja Rosenmund.
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