D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 64 Citations 14,243 200 World Ranking 1634 National Ranking 28

Research.com Recognitions

Awards & Achievements

Member of the European Molecular Biology Organization (EMBO)

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Enzyme
  • DNA

Her primary areas of study are Molecular biology, Ubiquitin, Virus, Proteasome and Virology. Her Molecular biology research is multidisciplinary, incorporating elements of DNA repair complex, DNA damage, Antigen and Phosphorylation. As part of one scientific family, Maria G. Masucci deals mainly with the area of Antigen, narrowing it down to issues related to the Cytotoxic T cell, and often Immunology and Interleukin 2.

Her Cell biology research extends to Ubiquitin, which is thematically connected. Her studies deal with areas such as Cell culture, NEDD8, Gene and Immune system as well as Virus. Her Virology research focuses on Epstein–Barr virus in particular.

Her most cited work include:

  • Inhibition of antigen processing by the internal repeat region of the Epstein-Barr virus nuclear antigen-1 (736 citations)
  • Small molecule RITA binds to p53, blocks p53-HDM-2 interaction and activates p53 function in tumors. (611 citations)
  • Short-lived green fluorescent proteins for quantifying ubiquitin/proteasome-dependent proteolysis in living cells (495 citations)

What are the main themes of her work throughout her whole career to date?

Maria G. Masucci mainly investigates Molecular biology, Virology, Cell biology, Epstein–Barr virus and Virus. Her Molecular biology research is multidisciplinary, relying on both Cell culture, Transfection, Interferon gamma, Cytotoxic T cell and Major histocompatibility complex. She interconnects Burkitt's lymphoma, Immune system and Antigen in the investigation of issues within Virology.

Her biological study spans a wide range of topics, including T cell and Antigen presentation. Her study looks at the relationship between Cell biology and topics such as Ubiquitin, which overlap with Proteasome and Proteolysis. Her Virus study combines topics in areas such as In vitro and Lymphoma.

She most often published in these fields:

  • Molecular biology (39.41%)
  • Virology (30.54%)
  • Cell biology (26.11%)

What were the highlights of her more recent work (between 2007-2021)?

  • Cell biology (26.11%)
  • Molecular biology (39.41%)
  • Ubiquitin (19.70%)

In recent papers she was focusing on the following fields of study:

The scientist’s investigation covers issues in Cell biology, Molecular biology, Ubiquitin, Virus and Deubiquitinating enzyme. Her study in Molecular biology is interdisciplinary in nature, drawing from both Transfection, Antigen, Chromatin and Cytotoxic T cell, CTL*. Her work deals with themes such as Innate immune system and Proteasome, which intersect with Ubiquitin.

Maria G. Masucci has researched Virus in several fields, including Apoptosis and DNA damage, Genome instability. The research on Virology and Immunology is part of her Epstein–Barr virus project. Maria G. Masucci interconnects Epitope and Human tumor in the investigation of issues within Virology.

Between 2007 and 2021, her most popular works were:

  • The Epstein–Barr virus nuclear antigen-1 promotes genomic instability via induction of reactive oxygen species (177 citations)
  • Three Epstein-Barr virus latency proteins independently promote genomic instability by inducing DNA damage, inhibiting DNA repair and inactivating cell cycle checkpoints (113 citations)
  • The ER‐resident ubiquitin‐specific protease 19 participates in the UPR and rescues ERAD substrates (107 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • DNA

Maria G. Masucci mainly investigates DNA damage, Molecular biology, Cell biology, Genome instability and Deubiquitinating enzyme. Her DNA damage research incorporates elements of Malignant transformation, Cancer research, DNA repair and Microbiology. Her Molecular biology research is multidisciplinary, incorporating perspectives in Chromatin, Chromatin remodeling, Nucleosome, HMGA and Cell nucleus.

Her Cell biology study combines topics in areas such as Transmembrane domain, Protease, TRIM25 and Cystic fibrosis transmembrane conductance regulator. As a part of the same scientific family, Maria G. Masucci mostly works in the field of Genome instability, focusing on Carcinogenesis and, on occasion, Cell cycle checkpoint and Virus. Maria G. Masucci studies Ubiquitin ligase which is a part of Ubiquitin.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Inhibition of antigen processing by the internal repeat region of the Epstein-Barr virus nuclear antigen-1

Jelena Levitskaya;Michael Coram;Victor Levitsky;Stefan Imreh.
Nature (1995)

1033 Citations

Small molecule RITA binds to p53, blocks p53-HDM-2 interaction and activates p53 function in tumors.

Natalia Issaeva;Przemyslaw Bozko;Martin Enge;Marina Protopopova.
Nature Medicine (2004)

911 Citations

Inhibition of ubiquitin/proteasome-dependent protein degradation by the Gly-Ala repeat domain of the Epstein–Barr virus nuclear antigen 1

Jelena Levitskaya;Anatoly Sharipo;Ainars Leonchiks;Aaron Ciechanover.
Proceedings of the National Academy of Sciences of the United States of America (1997)

702 Citations

Short-lived green fluorescent proteins for quantifying ubiquitin/proteasome-dependent proteolysis in living cells

Nico P. Dantuma;Kristina Lindsten;Rickard Glas;Marianne Jellne.
Nature Biotechnology (2000)

655 Citations

Epstein-Barr virus (EBV) load in bone marrow transplant recipients at risk to develop posttransplant lymphoproliferative disease: prophylactic infusion of EBV-specific cytotoxic T cells.

Åsa Gustafsson;Victor Levitsky;Jie Zhi Zou;Teresa Frisan.
Blood (2000)

465 Citations

Interleukin 10 pretreatment protects target cells from tumor- and allo-specific cytotoxic T cells and downregulates HLA class I expression.

M Matsuda;F Salazar;M Petersson;G Masucci.
Journal of Experimental Medicine (1994)

412 Citations

Aggregate formation inhibits proteasomal degradation of polyglutamine proteins

Lisette G.G.C. Verhoef;Kristina Lindsten;Maria G. Masucci;Nico P. Dantuma.
Human Molecular Genetics (2002)

347 Citations

The Epstein–Barr virus nuclear antigen-1 promotes genomic instability via induction of reactive oxygen species

Bettina Gruhne;Ramakrishna Sompallae;Diego Marescotti;Siamak Akbari Kamranvar.
Proceedings of the National Academy of Sciences of the United States of America (2009)

263 Citations

A transgenic mouse model of the ubiquitin/proteasome system.

Kristina Lindsten;Victoria Menéndez-Benito;Maria G Masucci;Nico P Dantuma.
Nature Biotechnology (2003)

254 Citations

Mutant ubiquitin found in neurodegenerative disorders is a ubiquitin fusion degradation substrate that blocks proteasomal degradation

Kristina Lindsten;Femke M.S. de Vrij;Lisette G.G.C. Verhoef;David F. Fischer.
Journal of Cell Biology (2002)

247 Citations

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