Lyanne C. Schlichter spends much of her time researching Microglia, Cell biology, Immunology, Inflammation and Pathology. The various areas that she examines in her Microglia study include Proinflammatory cytokine, Neuroscience and Neuroglia. Her Cell biology research incorporates themes from Potassium channel blocker, Potassium channel, Biochemistry, Voltage-gated ion channel and Patch clamp.
In general Immunology study, her work on Lymphoma often relates to the realm of Haematopoiesis, thereby connecting several areas of interest. Her Inflammation research is multidisciplinary, relying on both Intracerebral hemorrhage and Blood–brain barrier. Lyanne C. Schlichter has researched Pathology in several fields, including Evoked potential and Central nervous system disease.
Her main research concerns Microglia, Cell biology, Immunology, Inflammation and Pathology. The concepts of her Microglia study are interwoven with issues in Tumor necrosis factor alpha, Neuroinflammation, Stimulation, Neuroscience and Intracerebral hemorrhage. Her research integrates issues of Receptor, Calmodulin, Membrane potential and Potassium channel in her study of Cell biology.
Her work in Immunology addresses issues such as In vitro, which are connected to fields such as Lipopolysaccharide. Her Inflammation research includes elements of Myelin and Integrin alpha M. Her studies in Pathology integrate themes in fields like Axon, Blood–brain barrier and Ischemia.
Lyanne C. Schlichter mainly focuses on Microglia, Cell biology, Immunology, Stimulation and Neuroinflammation. Her Microglia study results in a more complete grasp of Inflammation. Lyanne C. Schlichter works mostly in the field of Inflammation, limiting it down to concerns involving Integrin alpha M and, occasionally, Interleukin, Nitric oxide, Messenger RNA and Astrogliosis.
Her Cell biology study combines topics in areas such as Anti-inflammatory, Calmodulin and Inward-rectifier potassium ion channel. Her Immunology study incorporates themes from Reactive oxygen species, In vitro, Ca2+/calmodulin-dependent protein kinase and KCNN4. Neuroscience is closely connected to Chemotaxis in her research, which is encompassed under the umbrella topic of Stimulation.
Her primary areas of study are Immunology, Microglia, In vitro, Tumor necrosis factor alpha and Inflammation. Her Immunology research includes themes of Myelin and Stimulation. Many of her studies on In vitro involve topics that are commonly interrelated, such as Integrin alpha M.
Her work carried out in the field of Tumor necrosis factor alpha brings together such families of science as Receptor, Lipopolysaccharide, Interleukin 10 and Interleukin 4. Her Inflammation study integrates concerns from other disciplines, such as Interleukin, Messenger RNA, Nitric oxide and Cell biology.
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Mechanisms of microglia-mediated neurotoxicity in a new model of the stroke penumbra.
Vikas Kaushal;Lyanne C Schlichter.
The Journal of Neuroscience (2008)
hSK4/hIK1, a calmodulin-binding KCa channel in human T lymphocytes. Roles in proliferation and volume regulation.
Rajesh Khanna;Martin C. Chang;William J. Joiner;Leonard K. Kaczmarek.
Journal of Biological Chemistry (1999)
Functional up-regulation of HERG K+ channels in neoplastic hematopoietic cells.
Garth A.M. Smith;Hing-Wo Tsui;Evan W. Newell;Xinpo Jiang.
Journal of Biological Chemistry (2002)
The Ca2+-Activated K+ Channel KCNN4/KCa3.1 Contributes to Microglia Activation and Nitric Oxide-Dependent Neurodegeneration
Vikas Kaushal;Paulo D. Koeberle;Yimin Wang;Lyanne C. Schlichter.
The Journal of Neuroscience (2007)
K+ channels and the microglial respiratory burst.
Rajesh Khanna;Rajesh Khanna;Lipi Roy;Lipi Roy;Xiaoping Zhu;Lyanne C. Schlichter;Lyanne C. Schlichter.
American Journal of Physiology-cell Physiology (2001)
Microglia Kv1.3 channels contribute to their ability to kill neurons.
Christopher B. Fordyce;Ravi Jagasia;Xiaoping Zhu;Lyanne C. Schlichter.
The Journal of Neuroscience (2005)
A Kv1.5 to Kv1.3 Switch in Endogenous Hippocampal Microglia and a Role in Proliferation
Suhas A. Kotecha;Lyanne C. Schlichter.
The Journal of Neuroscience (1999)
High intracellular calcium concentrations in transformed lymphoblasts from subjects with bipolar I disorder.
Emamghoreishi M;Schlichter L;Li Pp;Parikh S.
American Journal of Psychiatry (1997)
Properties of K+ and Cl- channels and their involvement in proliferation of rat microglial cells
L.C. Schlichter;G. Sakellaropoulos;B. Ballyk;P.S. Pennefather.
Neutrophil depletion reduces blood-brain barrier breakdown, axon injury, and inflammation after intracerebral hemorrhage
Iska Moxon-Emre;Lyanne C. Schlichter.
Journal of Neuropathology and Experimental Neurology (2011)
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