Her primary areas of investigation include Immunology, CD8, T cell, Cytotoxic T cell and Immune system. Lucy Golden-Mason has included themes like Oxidative stress, Receptor and Virology in her Immunology study. Her studies examine the connections between Receptor and genetics, as well as such issues in CLEC7A, with regards to Endocrinology and Internal medicine.
Her study in CD8 is interdisciplinary in nature, drawing from both Epitope and Interleukin-7 receptor. Lucy Golden-Mason studies Cytotoxic T cell, focusing on Interleukin 21 in particular. Her Interleukin 21 research focuses on IL-2 receptor and how it connects with FOXP3, Acquired immune system, Galectin, Kupffer cell and Proinflammatory cytokine.
Her scientific interests lie mostly in Immunology, Immune system, Virology, Hepatitis C virus and T cell. Immunology and Cytotoxic T cell are commonly linked in her work. Her Immune system research is multidisciplinary, relying on both Inflammation and Natural killer cell.
Her work on Interferon, CD81 and Viral load as part of general Virology research is frequently linked to Population, thereby connecting diverse disciplines of science. Her Hepatitis C virus research incorporates themes from Liver transplantation, Hepatitis C, Interferon gamma, Liver disease and CD5. Her research in T cell focuses on subjects like Receptor, which are connected to CLEC7A and Interleukin 10.
Lucy Golden-Mason focuses on Immunology, Hepatitis C virus, Virology, Immune system and Interferon. Her research on Immunology often connects related topics like Natural killer cell. The study incorporates disciplines such as CD8, Viral load and Cell biology in addition to Interferon.
Her CD8 research integrates issues from Interleukin 2, Cytotoxic T cell, Peripheral blood mononuclear cell and Interferon gamma. The Virus study combines topics in areas such as Molecular biology and Cytokine. Her Cytokine research includes elements of Receptor, Degranulation and Lymphocyte.
Immunology, Inflammation, Viral replication, Immune system and Cancer research are her primary areas of study. Her research integrates issues of NADPH oxidase and Cell signaling in her study of Immunology. Her Inflammation study integrates concerns from other disciplines, such as Oxidative stress, Interleukin 21, Granzyme B production and Interferon gamma.
Her studies in Viral replication integrate themes in fields like Molecular biology, Cytokine and Hepatitis C virus. In the subject of general Immune system, her work in CD8 is often linked to Population, thereby combining diverse domains of study. Her Cancer research study combines topics in areas such as Proinflammatory cytokine, Innate immune system and Cholesterol.
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Negative Immune Regulator Tim-3 Is Overexpressed on T Cells in Hepatitis C Virus Infection and Its Blockade Rescues Dysfunctional CD4+ and CD8+ T Cells
Lucy Golden-Mason;Brent E. Palmer;Nasim Kassam;Lisa Townshend-Bulson.
Journal of Virology (2009)
Upregulation of PD-1 Expression on Circulating and Intrahepatic Hepatitis C Virus-Specific CD8+ T Cells Associated with Reversible Immune Dysfunction
Lucy Golden-Mason;Brent Palmer;Jared Klarquist;John A. Mengshol.
Journal of Virology (2007)
Tim-3 expression on PD-1+ HCV-specific human CTLs is associated with viral persistence, and its blockade restores hepatocyte-directed in vitro cytotoxicity
Rachel H. McMahan;Lucy Golden-Mason;Michael I. Nishimura;Brian J. McMahon.
Journal of Clinical Investigation (2010)
Transmigrating Neutrophils Shape the Mucosal Microenvironment through Localized Oxygen Depletion to Influence Resolution of Inflammation
Eric L. Campbell;Walter J. Bruyninckx;Caleb J. Kelly;Louise E. Glover.
A Crucial Role for Kupffer Cell-Derived Galectin-9 in Regulation of T Cell Immunity in Hepatitis C Infection
John A. Mengshol;Lucy Golden-Mason;Tomohiro Arikawa;Maxwell Smith.
PLOS ONE (2010)
Bile acid receptor activation modulates hepatic monocyte activity and improves nonalcoholic fatty liver disease.
Rachel H. McMahan;Xiaoxin X. Wang;Lin Ling Cheng;Tibor Krisko.
Journal of Biological Chemistry (2013)
Altered natural killer cell subset distributions in resolved and persistent hepatitis C virus infection following single source exposure
Lucy Golden-Mason;Laura Madrigal-Estebas;Emma McGrath;Melissa J Conroy.
Class I HDACs regulate angiotensin II-dependent cardiac fibrosis via fibroblasts and circulating fibrocytes
Sarah M. Williams;Lucy Golden-Mason;Bradley S. Ferguson;Katherine B. Schuetze.
Journal of Molecular and Cellular Cardiology (2014)
Cutting edge: programmed death-1 expression is increased on immunocytes in chronic hepatitis C virus and predicts failure of response to antiviral therapy: race-dependent differences.
Lucy Golden-Mason;Jared Klarquist;Abdus S. Wahed;Hugo R. Rosen.
Journal of Immunology (2008)
Increased natural killer cell cytotoxicity and NKp30 expression protects against hepatitis C virus infection in high‐risk individuals and inhibits replication in vitro
Lucy Golden-Mason;Andrea L. Cox;Jessica A. Randall;Linling Cheng.
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