Kazuhiro Aoki mainly investigates Cell biology, Osteoclast, Bone resorption, MAPK/ERK pathway and Kinase. The Cell biology study combines topics in areas such as Endocrinology, Neurite, Transcription factor, Ectopic expression and Internal medicine. Kazuhiro Aoki combines subjects such as Haematopoiesis and RANKL with his study of Osteoclast.
His biological study spans a wide range of topics, including Multinucleate, Cancer research, Signal transduction, Proto-oncogene tyrosine-protein kinase Src and Resorption. His MAPK/ERK pathway research is multidisciplinary, relying on both Cell, Cell signaling and Cell growth. Kazuhiro Aoki studied Kinase and Extracellular that intersect with Optogenetics, Contraction, Wound healing and Myosin light-chain kinase.
His primary scientific interests are in Cell biology, Internal medicine, Endocrinology, Bone resorption and Osteoclast. Kazuhiro Aoki has researched Cell biology in several fields, including Cell and Förster resonance energy transfer. His research in Endocrinology intersects with topics in Cortical bone and Dental alveolus.
His Bone resorption study combines topics from a wide range of disciplines, such as Bone density, Bone remodeling and Peptide. In Osteoclast, Kazuhiro Aoki works on issues like RANKL, which are connected to Osteoblast, Cancer research and NF-κB. His MAPK/ERK pathway research also works with subjects such as
His primary areas of study are Cell biology, Biophysics, Intracellular, Kinase and Cell. He interconnects Optogenetics, Cell growth and Förster resonance energy transfer in the investigation of issues within Cell biology. The concepts of his Intracellular study are interwoven with issues in Extracellular and MAPK/ERK pathway.
His biological study deals with issues like Contraction, which deal with fields such as Mechanotransduction and Epidermal growth factor receptor. The Kinase study combines topics in areas such as Crosstalk, Programmed cell death and Chromosomal translocation. His Osteopetrosis research incorporates elements of Osteoclast, Bone resorption and RANKL.
Kazuhiro Aoki spends much of his time researching Cell biology, Kinase, Cell, Extracellular and Signal transduction. The various areas that Kazuhiro Aoki examines in his Cell biology study include RUNX2, Osteoblast and Activator. His study in Kinase is interdisciplinary in nature, drawing from both Live cell imaging, Intracellular signal transduction, Blot and Förster resonance energy transfer.
Kazuhiro Aoki combines subjects such as Cell growth and MAPK/ERK pathway with his study of Extracellular. His Signal transduction research is multidisciplinary, incorporating perspectives in Protein subcellular localization prediction and Cytoplasm. His Resorption research includes elements of Osteoclast, Bone resorption and RANKL.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The afterglow of GRB 050709 and the nature of the short-hard γ-ray bursts
Derek B Fox;Derek B Fox;Dale A Frail;Paul Antony Price;Shrinivas R Kulkarni.
Nature (2005)
Development of an optimized backbone of FRET biosensors for kinases and GTPases
Naoki Komatsu;Kazuhiro Aoki;Masashi Yamada;Hiroko Yukinaga.
Molecular Biology of the Cell (2011)
Selective inhibition of NF-|[kappa]|B blocks osteoclastogenesis and prevents inflammatory bone destruction in vivo
Eijiro Jimi;Eijiro Jimi;Kazuhiro Aoki;Hiroaki Saito;Fulvio D'Acquisto.
Nature Medicine (2004)
Cathepsin K-Dependent Toll-Like Receptor 9 Signaling Revealed in Experimental Arthritis
Masataka Asagiri;Toshitake Hirai;Toshihiro Kunigami;Shunya Kamano;Shunya Kamano.
Science (2008)
Dynamics of the Ras/ERK MAPK Cascade as Monitored by Fluorescent Probes
Aki Fujioka;Kenta Terai;Reina E. Itoh;Kazuhiro Aoki.
Journal of Biological Chemistry (2006)
Overexpression of DeltaFosB transcription factor(s) increases bone formation and inhibits adipogenesis.
G Sabatakos;N A Sims;J Chen;K Aoki.
Nature Medicine (2000)
Dual roles of Sema6D in cardiac morphogenesis through region-specific association of its receptor, Plexin-A1, with off-track and vascular endothelial growth factor receptor type 2
Toshihiko Toyofuku;Hong Zhang;Atsushi Kumanogoh;Noriko Takegahara.
Genes & Development (2004)
Regulation of osteoclast differentiation and function by the CaMK-CREB pathway.
Kojiro Sato;Ayako Suematsu;Tomoki Nakashima;Sayaka Takemoto-Kimura.
Nature Medicine (2006)
I[kgr]B[zgr] regulates TH17 development by cooperating with ROR nuclear receptors
Kazuo Okamoto;Yoshiko Iwai;Masatsugu Oh-hora;Masahiro Yamamoto.
Nature (2010)
Coupling of bone resorption and formation by RANKL reverse signalling
Yuki Ikebuchi;Shigeki Aoki;Masashi Honma;Madoka Hayashi.
Nature (2018)
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